Search Results - "Lause, P."

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  1. 1

    The Type 1 Insulin-Like Growth Factor Receptor (IGF-IR) Pathway Is Mandatory for the Follistatin-Induced Skeletal Muscle Hypertrophy by Kalista, S, Schakman, O, Gilson, H, Lause, P, Demeulder, B, Bertrand, L, Pende, M, Thissen, J. P

    Published in Endocrinology (Philadelphia) (01-01-2012)
    “…Myostatin inhibition by follistatin (FS) offers a new approach for muscle mass enhancement. The aim of the present study was to characterize the mediators…”
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  2. 2

    Myostatin Gene Deletion Prevents Glucocorticoid-Induced Muscle Atrophy by Gilson, H, Schakman, O, Combaret, L, Lause, P, Grobet, L, Attaix, D, Ketelslegers, J. M, Thissen, J. P

    Published in Endocrinology (Philadelphia) (01-01-2007)
    “…Glucocorticoids mediate muscle atrophy in many catabolic states. Myostatin expression, a negative regulator of muscle growth, is increased by glucocorticoids…”
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  3. 3

    Role of IGF-I and the TNFα/NF-κB pathway in the induction of muscle atrogenes by acute inflammation by Schakman, O, Dehoux, M, Bouchuari, S, Delaere, S, Lause, P, Decroly, N, Shoelson, S E, Thissen, J-P

    “…Several catabolic states (sepsis, cancer, etc.) associated with acute inflammation are characterized by a loss of skeletal muscle due to accelerated…”
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  4. 4

    Role of Akt/GSK-3β/β-Catenin Transduction Pathway in the Muscle Anti-Atrophy Action of Insulin-Like Growth Factor-I in Glucocorticoid-Treated Rats by Schakman, O, Kalista, S, Bertrand, L, Lause, P, Verniers, J, Ketelslegers, J. M, Thissen, J. P

    Published in Endocrinology (Philadelphia) (01-08-2008)
    “…Decrease of muscle IGF-I plays a critical role in muscle atrophy caused by glucocorticoids (GCs) because IGF-I gene electrotransfer prevents muscle atrophy…”
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  5. 5

    Insulin-Like Growth Factor-I Gene Transfer by Electroporation Prevents Skeletal Muscle Atrophy in Glucocorticoid-Treated Rats by Schakman, O, Gilson, H, de Coninck, V, Lause, P, Verniers, J, Havaux, X, Ketelslegers, J. M, Thissen, J. P

    Published in Endocrinology (Philadelphia) (01-04-2005)
    “…Catabolic states caused by injury are characterized by a loss of skeletal muscle. The anabolic action of IGF-I on muscle and the reduction of its muscle…”
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  6. 6

    Urotensin II induction of adult cardiomyocytes hypertrophy involves the Akt/GSK-3β signaling pathway by Gruson, D., Ginion, A., Decroly, N., Lause, P., Vanoverschelde, J.L., Ketelslegers, J.M., Bertrand, L., Thissen, J.P.

    Published in Peptides (New York, N.Y. : 1980) (01-07-2010)
    “…Urotensin II (UII) a potent vasoactive peptide is upregulated in the failing heart and promotes cardiomyocytes hypertrophy, in particular through…”
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    In Vitro Effects of Oestradiol on Galanin Gene Expression in Rat Anterior Pituitary Cells by Ponchon, M., Lause, P., Maiter, D.

    Published in Journal of neuroendocrinology (01-06-2000)
    “…While the pituitary galanin gene is highly responsive to oestrogen stimulation in vivo, in vitro effects of oestrogens on pituitary galanin gene expression…”
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  10. 10

    Failure of exogenous IGF-I to restore normal growth in rats submitted to dietary zinc deprivation by Ninh, NX, Maiter, D, Verniers, J, Lause, P, Ketelslegers, JM, Thissen, JP

    Published in Journal of endocrinology (01-11-1998)
    “…Dietary zinc deficiency in rats causes growth retardation associated with decreased circulating IGF-I concentrations. To investigate the potential role of low…”
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    Monoclonal antibodies to growth hormone (GH) prolong liver GH binding and GH-induced IGF-I/IGFBP-3 synthesis by Beauloye, V, Muaku, S M, Lause, P, Portetelle, D, Renaville, R, Robert, A R, Ketelslegers, J M, Maiter, D

    Published in The American journal of physiology (01-08-1999)
    “…This time-course study further explored the mechanisms whereby monoclonal antibodies (MAbs) may enhance growth hormone (GH) effects. Hypophysectomized rats…”
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  15. 15

    Continuous administration of growth hormone does not prevent the decrease of IGF-I gene expression in zinc-deprived rats despite normalization of liver GH binding by Ninh, N.X., Maiter, D., Lause, P., Chrzanowska, B., Underwood, L.E., Ketelslegers, J.M., Thissen, J.P.

    Published in Growth hormone & IGF research (01-12-1998)
    “…To determine the role of reduced liver GH binding (GHR) in the decreased IGF-I observed in zinc-deficient (ZD) animals, we investigated the effects of GHR…”
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  16. 16

    Role of the Insulin-Like Growth Factor I Decline in the Induction of Atrogin-1/MAFbx during Fasting and Diabetes by Dehoux, Mischael, Van Beneden, Ronald, Pasko, Nevi, Lause, Pascale, Verniers, Josiane, Underwood, Louis, Ketelslegers, Jean-Marie, Thissen, Jean-Paul

    Published in Endocrinology (Philadelphia) (01-11-2004)
    “…In catabolic conditions, atrogin-1/MAFbx, a muscle-specific ubiquitin-ligase required for muscle atrophy, is increased, and concentrations of IGF-I, a growth…”
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    Circulating Activin A is predictive of survival of cancer patients by Loumaye, A., de Barsy, M., Nachit, M., Lause, P., Frateur, L., Van Maanen, A., Trefois, P., Gruson, D., Thissen, J.-P.

    Published in Nutrition clinique et métabolisme (01-03-2016)
    “…We demonstrated that human cancer cachexia is associated with increased circulating concentrations of Activin A. Given the cachectic and anorectic effect of…”
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    Role of Akt/GSK-3beta/beta-catenin transduction pathway in the muscle anti-atrophy action of insulin-like growth factor-I in glucocorticoid-treated rats by Schakman, O, Kalista, S, Bertrand, L, Lause, P, Verniers, J, Ketelslegers, J M, Thissen, J P

    Published in Endocrinology (Philadelphia) (01-08-2008)
    “…Decrease of muscle IGF-I plays a critical role in muscle atrophy caused by glucocorticoids (GCs) because IGF-I gene electrotransfer prevents muscle atrophy…”
    Get full text
    Journal Article
  20. 20

    Urotensin II induction of adult cardiomyocytes hypertrophy involves the Akt/GSK-3b signaling pathway by Gruson, D, Ginion, A, Decroly, N, Lause, P, Vanoverschelde, J L, Ketelslegers, J M, Bertrand, L, Thissen, J P

    Published in Peptides (New York, N.Y. : 1980) (01-07-2010)
    “…Urotensin II (UII) a potent vasoactive peptide is upregulated in the failing heart and promotes cardiomyocytes hypertrophy, in particular through…”
    Get full text
    Journal Article