Search Results - "Laine Herborg, Laura"

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  1. 1

    Mapping the CLEC12A expression on myeloid progenitors in normal bone marrow; implications for understanding CLEC12A‐related cancer stem cell biology by Bill, Marie, B. van Kooten Niekerk, Peter, S. Woll, Petter, Laine Herborg, Laura, Stidsholt Roug, Anne, Hokland, Peter, Nederby, Line

    Published in Journal of cellular and molecular medicine (01-04-2018)
    “…The C‐type lectin domain family 12, member A (CLEC12A) receptor has emerged as a leukaemia‐associated and cancer stem cell marker in myeloid malignancies…”
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    Journal Article
  2. 2

    Mapping the CLEC 12A expression on myeloid progenitors in normal bone marrow; implications for understanding CLEC 12A‐related cancer stem cell biology by Bill, Marie, B. van Kooten Niekerk, Peter, S. Woll, Petter, Laine Herborg, Laura, Stidsholt Roug, Anne, Hokland, Peter, Nederby, Line

    Published in Journal of cellular and molecular medicine (01-04-2018)
    “…The C‐type lectin domain family 12, member A ( CLEC 12A) receptor has emerged as a leukaemia‐associated and cancer stem cell marker in myeloid malignancies…”
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    Journal Article
  3. 3

    RNA-Sequencing of Cytogenetically Normal Acute Myeloid Leukemia to Extend Routine Molecular Diagnostics by Herborg, Laura Laine, Hansen, Marcus Celik, Hansen, Maria, Roug, Anne Stidsholt, Hokland, Peter

    Published in Blood (03-12-2015)
    “…Introduction Despite the discovery of new genetic alterations the cytogenetically normal acute myeloid leukemia (CN-AML) subset is still insufficiently…”
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    Journal Article
  4. 4

    Kinetics of del(7q) driven leukemogenesis in a patient with JAK2 V617F and TET2 mutated chronic myeloproliferative neoplasm by Herborg, Laura Laine, Nederby, Line, Kjeldsen, Eigil, Grønbæk, Kirsten, Hokland, Peter, Hansen, Maria, Celik Hansen, Marcus, Roug, Anne Stidsholt

    Published in Leukemia research reports (2013)
    “…Abstract Chronic myeloid neoplasms have susceptibility to transform into acute myeloid leukemia due to attainment of additional molecular lesions. We here…”
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    Journal Article