Search Results - "Kuriya, G"

Double deficiency in IL-17 and IFN-γ signalling significantly suppresses the development of diabetes in the NOD mouse by Kuriya, G., Uchida, T., Akazawa, S., Kobayashi, M., Nakamura, K., Satoh, T., Horie, I., Kawasaki, E., Yamasaki, H., Yu, L., Iwakura, Y., Sasaki, H., Nagayama, Y., Kawakami, A., Abiru, N.

“…Aims/hypothesis T helper type (Th) 17 cells have been shown to play important roles in mouse models of several autoimmune diseases that have been classified as…”
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Association between anti-ZnT8 autoantibody specificities and SLC30A8 Arg325Trp variant in Japanese patients with type 1 diabetes by Kawasaki, E, Uga, M, Nakamura, K, Kuriya, G, Satoh, T, Fujishima, K, Ozaki, M, Abiru, N, Yamasaki, H, Wenzlau, J. M, Davidson, H. W, Hutton, J. C, Eguchi, K

“…Aims/hypothesis We analysed the association between humoral autoreactivity to zinc transporter-8 (ZnT8) and the SLC30A8 rs13266634 polymorphism (Arg325Trp),…”
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Genetic deletion of granzyme B does not confer resistance to the development of spontaneous diabetes in non‐obese diabetic mice by Kobayashi, M., Kaneko‐Koike, C., Abiru, N., Uchida, T., Akazawa, S., Nakamura, K., Kuriya, G., Satoh, T., Ida, H., Kawasaki, E., Yamasaki, H., Nagayama, Y., Sasaki, H., Kawakami, A.

“…Summary Granzyme B (GzmB) and perforin are proteins, secreted mainly by natural killer cells and cytotoxic T lymphocytes that are largely responsible for the…”
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CHOP deletion does not impact the development of diabetes but suppresses the early production of insulin autoantibody in the NOD mouse by Satoh, T., Abiru, N., Kobayashi, M., Zhou, H., Nakamura, K., Kuriya, G., Nakamura, H., Nagayama, Y., Kawasaki, E., Yamasaki, H., Yu, L., Eisenbarth, G. S., Araki, E., Mori, M., Oyadomari, S., Eguchi, K.

“…C/EBP homologous protein (CHOP) has been proposed as a key transcription factor for endoplasmic reticulum (ER) stress-mediated β-cell death induced by…”
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Double deficiency in IL-17 and IFN-[gamma] signalling significantly suppresses the development of diabetes in the NOD mouse by Kuriya, G, Uchida, T, Akazawa, S, Kobayashi, M, Nakamura, K, Satoh, T, Horie, I, Kawasaki, E, Yamasaki, H, Yu, L, Iwakura, Y, Sasaki, H, Nagayama, Y, Kawakami, A, Abiru, N

“…T helper type (Th) 17 cells have been shown to play important roles in mouse models of several autoimmune diseases that have been classified as Th1 diseases…”
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