Search Results - "Kudriavtsev, Igor V"

Regulation of Apolipoprotein A-I Gene Expression in Human Macrophages by Oxidized Low-Density Lipoprotein by Nekrasova, Ekaterina V., Larionova, Ekaterina E., Danko, Katerina, Kuzmina, Darya O., Shavva, Vladimir S., Kudriavtsev, Igor V., Orlov, Sergey V.

“…Apolipoprotein A-I (ApoA-I) is a key component of reverse cholesterol transport in humans. In the previous studies, we demonstrated expression of the apoA-I…”
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Tumor necrosis factor α stimulates endogenous apolipoprotein A-I expression and secretion by human monocytes and macrophages: role of MAP-kinases, NF-κB, and nuclear receptors PPARα and LXRs by Shavva, Vladimir S., Mogilenko, Denis A., Nekrasova, Ekaterina V., Trulioff, Andrey S., Kudriavtsev, Igor V., Larionova, Ekaterina E., Babina, Anna V., Dizhe, Ella B., Missyul, Boris V., Orlov, Sergey V.

“…Apolipoprotein A-I (ApoA-I) is the main structural and functional protein component of high-density lipoprotein. ApoA-I has been shown to regulate lipid…”
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Differentiation of human macrophages with anaphylatoxin C3a impairs alternative M2 polarization and decreases lipopolysaccharide‐induced cytokine secretion by Mogilenko, Denis A, Danko, Katerina, Larionova, Ekaterina E, Shavva, Vladimir S, Kudriavtsev, Igor V, Nekrasova, Ekaterina V, Burnusuz, Alexandra V, Gorbunov, Nikolay P, Trofimov, Alexander V, Zhakhov, Alexander V, Ivanov, Igor A, Orlov, Sergey V

“…Anaphylatoxin C3a is a small signaling polypeptide that is generated during complement activation. C3a is involved in the regulation of various innate and…”
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Peroxisome Proliferator-activated Receptor α Positively Regulates Complement C3 Expression but Inhibits Tumor Necrosis Factor α-mediated Activation of C3 Gene in Mammalian Hepatic-derived Cells by Mogilenko, Denis A., Kudriavtsev, Igor V., Shavva, Vladimir S., Dizhe, Ella B., Vilenskaya, Ekaterina G., Efremov, Alexander M., Perevozchikov, Andrej P., Orlov, Sergey V.

“…Complement C3 is a pivotal component of three cascades of complement activation. The liver is the main source of C3 in circulation and expression and secretion…”
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Modified Low Density Lipoprotein Stimulates Complement C3 Expression and Secretion via Liver X Receptor and Toll-like Receptor 4 Activation in Human Macrophages by Mogilenko, Denis A., Kudriavtsev, Igor V., Trulioff, Andrey S., Shavva, Vladimir S., Dizhe, Ella B., Missyul, Boris V., Zhakhov, Alexander V., Ischenko, Alexander M., Perevozchikov, Andrej P., Orlov, Sergey V.

“…Complement C3 is a pivotal component of three cascades of complement activation. C3 is expressed in human atherosclerotic lesions and is involved in…”
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Endogenous apolipoprotein A‐I stabilizes ATP‐binding cassette transporter A1 and modulates Toll‐like receptor 4 signaling in human macrophages by Mogilenko, Denis A., Orlov, Sergey V., Trulioff, Andrey S., Ivanov, Andrey V., Nagumanov, Vadim K., Kudriavtsev, Igor V., Shavva, Vladimir S., Tanyanskiy, Dmitry A., Perevozchikov, Andrej P.

“…Apolipoprotein A‐I (ApoA‐I) is the main functional protein component of human high‐density lipoproteins. ApoA‐I shows various anti‐inflammatory and…”
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Tumor necrosis factor [alpha] stimulates endogenous apolipoprotein A-I expression and secretion by human monocytes and macrophages: role of MAP-kinases, NF-[kappa]B, and nuclear receptors PPAR[alpha] and LXRs by Shavva, Vladimir S, Mogilenko, Denis A, Nekrasova, Ekaterina V, Trulioff, Andrey S, Kudriavtsev, Igor V, Larionova, Ekaterina E, Babina, Anna V

“…Apolipoprotein A-I (ApoA-I) is the main structural and functional protein component of high-density lipoprotein. ApoA-I has been shown to regulate lipid…”
Get full text