Search Results - "Krebs, Jutta"

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    Local injection of stem cell factor (SCF) improves myocardial homing of systemically delivered c-kit + bone marrow-derived stem cells by Lutz, Matthias, Rosenberg, Mark, Kiessling, Fabian, Eckstein, Volker, Heger, Thomas, Krebs, Jutta, Ho, Anthony D., Katus, Hugo A., Frey, Norbert

    Published in Cardiovascular research (01-01-2008)
    “…Aims Recent studies have shown that stem cell therapy may alleviate the detrimental effects of myocardial infarction. Yet, most of these reports observed only…”
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    Differential expression of cardiac neurotrophic factors and sympathetic nerve ending abnormalities within the failing heart by Kreusser, Michael M, Buss, Sebastian J, Krebs, Jutta, Kinscherf, Ralf, Metz, Jürgen, Katus, Hugo A, Haass, Markus, Backs, Johannes

    “…Abstract In congestive heart failure (CHF), cardiac sympathetic nerve endings transdifferentiate from a balanced norepinephrine (NE) storage/release/uptake…”
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    Coculture with hematopoietic stem cells protects cardiomyocytes against apoptosis via paracrine activation of AKT by Rosenberg, Mark, Lutz, Matthias, Kühl, Constantin, Will, Rainer, Eckstein, Volker, Krebs, Jutta, Katus, Hugo A, Frey, Norbert

    Published in Journal of translational medicine (06-06-2012)
    “…Previous experimental studies concluded that stem cells (SC) may exert their beneficial effects on the ischemic heart by paracrine activation of antiapoptotic…”
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    Cardiac remodeling is not modulated by overexpression of muscle LIM protein (MLP) by Kuhn, Christian, Frank, Derk, Dierck, Franziska, Oehl, Ulrike, Krebs, Jutta, Will, Rainer, Lehmann, Lorenz H., Backs, Johannes, Katus, Hugo A., Frey, Norbert

    Published in Basic research in cardiology (01-05-2012)
    “…Muscle LIM protein (MLP) has been proposed to be a central player in the pathogenesis of heart muscle disease. In line with this notion, the homozygous loss of…”
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    Cyclic AMP represses pathological MEF2 activation by myocyte-specific hypo-phosphorylation of HDAC5 by He, Tao, Huang, Jiale, Chen, Lan, Han, Gang, Stanmore, David, Krebs-Haupenthal, Jutta, Avkiran, Metin, Hagenmüller, Marco, Backs, Johannes

    “…Class IIa histone deacetylases (HDACs) critically regulate cardiac function through the repression of the activity of myocyte enhancer factor 2…”
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