Search Results - "Kowaltowski, Alicia Juliana"
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Early Postnatal Cardiomyocyte Proliferation Requires High Oxidative Energy Metabolism
Published in Scientific reports (13-11-2017)“…Cardiac energy metabolism must cope with early postnatal changes in tissue oxygen tensions, hemodynamics, and cell proliferation to sustain development. Here,…”
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Intermittent Fasting Results in Tissue-Specific Changes in Redox State
Published in Free radical biology & medicine (01-11-2014)Get full text
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233 - Intermittent Fasting Results in Tissue-Specific Changes in Redox State
Published in Free radical biology & medicine (01-11-2014)Get full text
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Very Long Chain Acyl-CoA Dehydrogenase Is a Diet-Sensitive Source of Reactive Oxygen Species in Liver Mitochondria
Published in Free radical biology & medicine (01-11-2012)Get full text
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Calorie restriction changes lipidomic profiles and maintains mitochondrial function and redox balance during isoproterenol-induced cardiac hypertrophy
Published in Journal of physiology and biochemistry (01-02-2022)“…Typically, healthy cardiac tissue utilizes more fat than any other organ. Cardiac hypertrophy induces a metabolic shift leading to a preferential consumption…”
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6
Entrevista
Published in Revista de medicina (São Paulo, Brazil) (18-06-2012)Get full text
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Mitochondrial ATP-sensitive Potassium Channel Opening Inhibits Isoproterenol-induced Cardiac Hypertrophy by Preventing Oxidative Damage
Published in Journal of cardiovascular pharmacology (01-04-2015)“…Cardiac hypertrophy is a chronic complex disease that occurs in response to hemodynamic load and is accompanied by oxidative stress and mitochondrial…”
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Effect of Calorie Restriction and Aging on Yeast Proteasome Activity
Published in Free radical biology & medicine (2010)Get full text
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9
cis-4-decenoic acid provokes mitochondrial bioenergetic dysfunction in rat brain
Published in Life sciences (1973) (31-07-2010)“…In the present work we investigated the in vitro effect of cis-4-decenoic acid, the pathognomonic metabolite of medium-chain acyl-CoA dehydrogenase deficiency,…”
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REDOX PROPERTIES OF ATP‐SENSITIVE K+ CHANNELS IN BRAIN MITOCHONDRIA
Published in The FASEB journal (01-03-2008)“…Abstract only Brain mitochondrial ATP‐sensitive K + channel (mitoK ATP ) opening by diazoxide protects against ischemic damage and excitotoxic cell death. We…”
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