Search Results - "Kordich, Lucía"

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  1. 1

    Impact of homocysteine-thiolactone on plasma fibrin networks by Genoud, Valeria, Lauricella, Ana María, Kordich, Lucía C., Quintana, Irene

    Published in Journal of thrombosis and thrombolysis (01-11-2014)
    “…Epidemiologic studies have shown that hyperhomocysteinemia is an independent risk factor for vascular disease. Homocysteine (Hcy) circulates as different…”
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    Journal Article
  2. 2

    Alterations of fibrin network structure mediated by dermatan sulfate by Lauricella, Ana María, Castañon, María Mercedes, Kordich, Lucía C., Quintana, Irene L.

    Published in Journal of thrombosis and thrombolysis (01-02-2013)
    “…Dermatan sulfate (DS) is well-known for its anticoagulant activity through binding to heparin cofactor II (HCII) to enhance thrombin inhibition. It has also…”
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  3. 3

    Insight into the profibrinolytic activity of dermatan sulfate: Effects on the activation of plasminogen mediated by tissue and urinary plasminogen activators by Castañon, María Mercedes, Gamba, Cecilia, Kordich, Lucía C

    Published in Thrombosis research (2007)
    “…Abstract Introduction Dermatan sulfate (DS) is well-known for its anticoagulant activity through binding to heparin cofactor II to enhance antithrombin action…”
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  4. 4

    Plasma homocysteine cutoff values for venous thrombosis by Castañon, María Mercedes, Lauricella, Ana María, Kordich, Lucía, Quintana, Irene

    Published in Clinical chemistry and laboratory medicine (01-01-2007)
    “…Hyperhomocysteinemia is considered an independent risk factor for vascular occlusive diseases. To date, there is no general agreement on hyperhomocysteinemia…”
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  5. 5

    Impaired fibrin gel permeability by high homocysteine levels by Quintana, Irene L, Oberholzer, María V, Kordich, Lucía, Lauricella, Ana M

    Published in Thrombosis research (01-01-2011)
    “…Abstract Mechanisms involved in the relationship between hyperhomocysteinemia and thrombosis are still unclear. In previous reports we have shown that high…”
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  6. 6

    Homocysteine modifies fibrin clot deformability: another possible explanation of harm by Rojas, Ana María, Kordich, Lucía, Lauricella, Ana María

    Published in Biorheology (Oxford) (2009)
    “…Several studies have associated elevated plasma homocysteine (Hcy) levels with higher risk of thrombosis. In order to evaluate possible changes in fibrin…”
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  7. 7

    Effects of homocysteine thiol group on fibrin networks: another possible mechanism of harm by Lauricella, Ana M, Quintana, Irene L, Kordich, Lucı́a C

    Published in Thrombosis research (15-07-2002)
    “…On the basis of growing clinical evidence, it is well known that elevated plasma homocysteine (Hcy) levels are associated with higher risk of venous and…”
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  8. 8

    Influence of homocysteine on fibrin network lysis by Lauricella, Ana María, Quintana, Irene, Castañon, Mercedes, Sassetti, Beatriz, Kordich, Lucía

    Published in Blood coagulation & fibrinolysis (01-04-2006)
    “…To elucidate some of the links between homocysteine and vascular disease, we have evaluated the effect of the amino acid on the formation (by kinetics…”
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    Increased lipid peroxidation correlates with platelet activation but not with markers of endothelial cell and blood coagulation activation in patients with antiphospholipid antibodies by Martinuzzo, Marta E., Forastiero, Ricardo R., Kordich, Lucía, Carreras, Luis O.

    Published in British journal of haematology (01-09-2001)
    “…Recent studies have shown that patients with antiphospholipid antibodies (aPL) have increased lipid peroxidation. We evaluated the urinary excretion of…”
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  11. 11

    Plasmatic homocysteine response to vitamin supplementation in elderly people by Murúa, Alicia L, Quintana, Irene, Janson, Jorge, Batista, Mariana, Camera, Mario I, Kordich, Lucı́a C

    Published in Thrombosis research (15-12-2000)
    “…Homocysteine (Hcy) increase is now widely accepted as a risk factor for vascular disease. The effects of folic acid (FA) and vitamins B 12 and B 6 in lowering…”
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    Markers of platelet, endothelial cell and blood coagulation activation in leprosy patients with antiphospholipid antibodies by MARTINUZZO, M. E, DE LARRANAGA, G. F, FORASTIERO, R. R, PELEGRI, Y, FARINA, M. H, ALONSO, B. S, KORDICH, L. C, CARRERAS, L. O

    Published in Clinical and experimental rheumatology (01-07-2002)
    “…To evaluate plasma levels of markers of platelet, endothelial cell and blood coagulation activation in leprosy patients with or without antiphospholipid…”
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  17. 17

    Hemolytic uremic syndrome in children : Platelet aggregation and membrane glycoproteins by SASSETTI, B, VIZCARGÜENAGA, M. I, ZANARO, N. L, SILVA, M. V, KORDICH, L, FLORENTINI, L, DIAZ, M, VITACCO, M, SANCHEZ AVALOS, J. C

    Published in Journal of pediatric hematology/oncology (01-03-1999)
    “…Several mechanisms have been proposed to explain the fibrin-platelet thrombosis at the microcirculation level in the different clinical conditions of hemolytic…”
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  18. 18

    IMPAIRED CLOT LYSIS BY rt-PA CATALYZED MINI-PLASMINOGEN ACTIVATION by Duboscq, Cristina, Genoud, Valeria, Parborell, M.Fernanda, Kordich, Lucía C.

    Published in Thrombosis research (15-06-1997)
    “…The fibrinolytic system contains a proenzyme plasminogen (Plg) which is converted to plasmin (Plm) by the action of Plg activators. Physiological Plg…”
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    Plasminogen: an important hemostatic parameter in septic patients by Duboscq, C, Quintana, I, Bassilotta, E, Bergonzelli, G E, Porterie, P, Sassetti, B, Haedo, A S, Wainsztein, N, Kruithof, E K, Kordich, L

    Published in Thrombosis and haemostasis (01-06-1997)
    “…Previously we observed in some but not all septic patients a low plasma concentration of plasminogen. To investigate prospectively whether plasma levels of…”
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    LIPID PROFILE IN A GROUP OF SHOOLCHILDREN AT SOCIAL RISK by Perris, Paula, Fernandez, Inés, Feliu, Maria Susana, Marina, Gurevich, Boschi, Elena, Kordich, Lucia, Bordoni, Noemi, Slobodianik, Nora

    Published in The FASEB journal (01-03-2006)
    “…Clinical and epidemiologic studies established a relationship between an abnormal plasma lipoprotein status and the risk of cardiovascular disease. The…”
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