Search Results - "Kloss, Christian U. A."

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  1. 1

    Microvascular Basal Lamina Injury After Experimental Focal Cerebral Ischemia and Reperfusion in the Rat by Hamann, Gerhard F., Liebetrau, Martin, Martens, Helge, Burggraf, Dorothe, Kloss, Christian U. A., Bültemeier, Gundula, Wunderlich, Natalie, Jäger, Gabriele, Pfefferkorn, Thomas

    “…To define the location and extent of microvascular damage of the basal lamina after cerebral ischemia and reperfusion in rats, the authors subjected animals (n…”
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  2. 2

    Immune Surveillance in the Injured Nervous System: T-Lymphocytes Invade the Axotomized Mouse Facial Motor Nucleus and Aggregate around Sites of Neuronal Degeneration by Raivich, Gennadij, Jones, Leonard L, Kloss, Christian UA, Werner, Alexander, Neumann, Harald, Kreutzberg, Georg W

    Published in The Journal of neuroscience (01-08-1998)
    “…Although the CNS is an established immune-privileged site, it is under surveillance by the immune system, particularly under pathological conditions. In the…”
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  3. 3

    Loss of microglial ramification in microglia-astrocyte cocultures: Involvement of adenylate cyclase, calcium, phosphatase, and Gi-protein systems by Kalla, Roger, Bohatschek, Marion, Kloss, Christian U.A., Krol, Janna, Von Maltzan, Xenia, Raivich, Gennadij

    Published in Glia (01-01-2003)
    “…Reduction in microglial branching is a common feature in brain pathology and culminates in the transformation into small, rounded, microglia‐derived phagocytes…”
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  4. 4

    Prolonged cerebral transit time in CADASIL: A transcranial ultrasound study by LIEBETRAU, Martin, HERZOG, Jürgen, KLOSS, Christian U. A, HAMANN, Gerhard F, DICHGANS, Martin

    Published in Stroke (1970) (01-02-2002)
    “…Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) is a hereditary angiopathy caused by mutations in Notch3…”
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  5. 5

    Effect of Lipopolysaccharide on the Morphology and Integrin Immunoreactivity of Ramified Microglia in the Mouse Brain and in Cell Culture by Kloss, Christian U.A., Bohatschek, Marion, Kreutzberg, Georg W., Raivich, Gennadij

    Published in Experimental neurology (01-03-2001)
    “…Microglial cells form the first line of defense in brain infection. They are related to monocytes and macrophages and can be readily activated by cell wall…”
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  6. 6

    Lymphocyte infiltration in the injured brain: Role of proinflammatory cytokines by Raivich, Gennadij, Bohatschek, Marion, Werner, Alexander, Jones, Leonard L., Galiano, Matthias, Kloss, Christian U.A., Zhu, Xing-Zu, Pfeffer, Klaus, Liu, Zhi Qiang

    Published in Journal of neuroscience research (15-06-2003)
    “…Studies using mouse axotomised facial motoneuron model show a strong and highly selective entry of CD3+ lymphocytes into the affected nucleus, with a maximum…”
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  7. 7

    Tissue-saving infarct volumetry using histochemistry validated by MRI in rat focal ischemia by Kloss, Christian U.A., Thomassen, Nora, Fesl, Gunther, Helge Martens, K., Yousri, Tarek A., Hamann, Gerhard F.

    Published in Neurological research (New York) (01-10-2002)
    “…Lesion size is an important outcome parameter in experimental stroke research. However, most methods of measuring the infarct volume in rodents either require…”
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  8. 8
  9. 9

    Plasminogen activation in experimental permanent focal cerebral ischemia by Pfefferkorn, Thomas, Wiessner, Christoph, Allegrini, Peter R., Staufer, Brigitte, Vosko, Milan R., Liebetrau, Martin, Bueltemeier, Gundula, Kloss, Christian U.A., Hamann, Gerhard F.

    Published in Brain research (03-11-2000)
    “…Background: Previous experimental work using in situ zymography has shown very early increased plasminogen activation in ischemic regions after 3 h of ischemia…”
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  10. 10

    MRI predicts area of increased plasminogen activation in permanent focal cerebral ischemia by Pfefferkorn, Thomas, Wiessner, Christoph, Allegrini, Peter R., Kloss, Christian U.A., Hamann, Gerhard F.

    Published in Neurological research (New York) (01-12-2002)
    “…To determine if MRI can predict intracerebral plasminogen activation after focal cerebral ischemia (FCI), ischemic regions detected by MRI after 48 h of…”
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