Search Results - "Kitabayashi, Chika"

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  1. 1

    Interleukin-17 Promotes Autoimmunity by Triggering a Positive-Feedback Loop via Interleukin-6 Induction by Ogura, Hideki, Murakami, Masaaki, Okuyama, Yuko, Tsuruoka, Mineko, Kitabayashi, Chika, Kanamoto, Minoru, Nishihara, Mika, Iwakura, Yoichiro, Hirano, Toshio

    Published in Immunity (Cambridge, Mass.) (17-10-2008)
    “…Dysregulated cytokine expression and signaling are major contributors to a number of autoimmune diseases. Interleukin-17A (IL-17A) and IL-6 are important in…”
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    Journal Article
  2. 2

    Zinc suppresses Th17 development via inhibition of STAT3 activation by Kitabayashi, Chika, Fukada, Toshiyuki, Kanamoto, Minoru, Ohashi, Wakana, Hojyo, Shintaro, Atsumi, Toru, Ueda, Naoko, Azuma, Ichiro, Hirota, Hiroshi, Murakami, Masaaki, Hirano, Toshio

    Published in International immunology (01-05-2010)
    “…Zinc (Zn) is an essential trace metal required by many enzymes and transcription factors for their activity or the maintenance of their structure. Zn has a…”
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    Journal Article
  3. 3

    IL-6–gp130–STAT3 in T cells directs the development of IL-17+ Th with a minimum effect on that of Treg in the steady state by Nishihara, Mika, Ogura, Hideki, Ueda, Naoko, Tsuruoka, Mineko, Kitabayashi, Chika, Tsuji, Fumio, Aono, Hiroyuki, Ishihara, Katsuhiko, Huseby, Eric, Betz, Ulrich A. K., Murakami, Masaaki, Hirano, Toshio

    Published in International immunology (01-06-2007)
    “…IL-17-producing Th (Th17) comprise a distinct lineage of pro-inflammatory Th that are major contributors to autoimmune diseases. Treatment with IL-6 and…”
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    Journal Article
  4. 4

    IL-6-gp130-STAT3 in T cells directs the development of IL-17+ T[sub]h with a minimum effect on that of Treg in the steady state by Nishihara, Mika, Ogura, Hideki, Ueda, Naoko, Tsuruoka, Mineko, Kitabayashi, Chika, Tsuji, Fumio, Aono, Hiroyuki, Ishihara, Katsuhiko, Huseby, Eric, Betz, Ulrich A K, Murakami, Masaaki, Hirano, Toshio

    Published in International immunology (01-06-2007)
    “…IL-17-producing Th (Th 17) comprise a distinct lineage of pro-inflammatory Th that are major contributors to autoimmune diseases. Treatment with IL-6 and…”
    Get full text
    Journal Article
  5. 5

    Hepatic Interleukin-7 Expression Regulates T Cell Responses by Sawa, Yukihisa, Arima, Yasunobu, Ogura, Hideki, Kitabayashi, Chika, Jiang, Jing-Jing, Fukushima, Toru, Kamimura, Daisuke, Hirano, Toshio, Murakami, Masaaki

    Published in Immunity (Cambridge, Mass.) (20-03-2009)
    “…Systemic cytokine activity in response to Toll-like receptor (TLR) signaling induces the expression of various proteins in the liver after infections. Here we…”
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    Journal Article
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    Homeostatically proliferating CD4^sup +^ T cells are involved in the pathogenesis of an Omenn syndrome murine model by Khiong, Khie, Murakami, Masaaki, Kitabayashi, Chika, Ueda, Naoko

    Published in The Journal of clinical investigation (01-05-2007)
    “…Patients with Omenn syndrome (OS) have hypomorphic RAG mutations and develop varying manifestations of severe combined immunodeficiency. It is not known which…”
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    Journal Article
  11. 11

    Interleukin-17 Promotes Autoimmunity by Triggering a Positive-Feedback Loop via Interleukin-6 Induction by Ogura, Hideki, Murakami, Masaaki, Okuyama, Yuko, Tsuruoka, Mineko, Kitabayashi, Chika, Kanamoto, Minoru, Nishihara, Mika, Iwakura, Yoichiro, Hirano, Toshio

    Published in Immunity (Cambridge, Mass.) (01-10-2008)
    “…Dysregulated cytokine expression and signaling are major contributors to a number of autoimmune diseases. Interleukin-17A (IL-17A) and IL-6 are important in…”
    Get full text
    Journal Article
  12. 12

    IL-6-gp130-STAT3 in T cells directs the development of IL-17+ T sub(h) with a minimum effect on that of Treg in the steady state by Nishihara, Mika, Ogura, Hideki, Ueda, Naoko, Tsuruoka, Mineko, Kitabayashi, Chika, Tsuji, Fumio, Aono, Hiroyuki, Ishihara, Katsuhiko, Huseby, Eric, Betz, Ulrich AK, Murakami, Masaaki, Hirano, Toshio

    Published in International immunology (01-06-2007)
    “…IL-17-producing T sub(h) (T sub(h)17) comprise a distinct lineage of pro-inflammatory T sub(h) that are major contributors to autoimmune diseases. Treatment…”
    Get full text
    Journal Article
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