Search Results - "Keri, R.A"
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1
FOXA1 represses the molecular phenotype of basal breast cancer cells
Published in Oncogene (31-01-2013)“…Breast cancer is a heterogeneous disease that comprises multiple subtypes. Luminal subtype tumors confer a more favorable patient prognosis, which is, in part,…”
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2
LMO4 is an essential mediator of ErbB2 HER2 Neu-induced breast cancer cell cycle progression
Published in Oncogene (15-10-2009)“…ErbB2/HER2/Neu-overexpressing breast cancers are characterized by poor survival due to high proliferation and metastasis rates and identifying downstream…”
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3
Ovarian hyperstimulation induces centrosome amplification and aneuploid mammary tumors independently of alterations in p53 in a transgenic mouse model of breast cancer
Published in Oncogene (13-03-2008)“…Aneuploidy and genomic instability are common features of human cancers, including breast cancer; however, mechanisms by which such abnormalities develop are…”
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4
Transcriptional repression of the glycoprotein hormone alpha subunit gene by androgen may involve direct binding of androgen receptor to the proximal promoter
Published in The Journal of biological chemistry (25-06-1993)“…Testicular androgens suppress the synthesis and secretion of the pituitary gonadotropins, in particular, luteinizing hormone. This suppressive effect includes…”
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5
Implementing transgenic and embryonic stem cell technology to study gene expression, cell-cell interactions and gene function
Published in Biology of reproduction (01-02-1995)“…This review highlights the use of transgenic mice and gene targeting in the study of reproduction, pituitary gene expression, and cell lineage. Since 1980…”
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6
Different combinations of regulatory elements may explain why placenta-specific expression of the glycoprotein hormone alpha-subunit gene occurs only in primates and horses
Published in Biology of reproduction (01-02-1991)“…Expression of the glycoprotein hormone alpha-subunit gene occurs in the pituitary of all mammals but in placenta of only primates and horses. In humans, two…”
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