Search Results - "Ke, Z. J."

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  1. 1

    Thiamine deficiency induces anorexia by inhibiting hypothalamic AMPK by Liu, M, Alimov, A.P, Wang, H, Frank, J.A, Katz, W, Xu, M, Ke, Z.-J, Luo, J

    Published in Neuroscience (16-05-2014)
    “…Highlights • Thiamine deficiency decreases food intake and increases basal energy expenditure. • Thiamine deficiency induces a severe weight loss. • Thiamine…”
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  2. 2

    The Resveratrol Attenuates Ethanol-Induced Hepatocyte Apoptosis Via Inhibiting ER-Related Caspase-12 Activation and PDE Activity In Vitro by Liu, LQ, Fan, ZQ, Tang, YF, Ke, ZJ

    “…Background Endoplasmic reticulum (ER) stress plays a key role in cell apoptosis pathways. Caspase‐12, a proapoptotic gene induced by ER stress, is also the key…”
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  3. 3

    Thiamine deficiency induces endoplasmic reticulum stress in neurons by Wang, X, Wang, B, Fan, Z, Shi, X, Ke, Z.-J, Luo, J

    Published in Neuroscience (09-02-2007)
    “…Abstract Thiamine (vitamin B1) deficiency (TD) causes region selective neuronal loss in the brain; it has been used to model neurodegeneration that accompanies…”
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  4. 4

    Improvement of cognitive deficits in SAMP8 mice by 3-n-butylphthalide by Wang, F., Chen, H., Sun, X-J., Ke, Z-J.

    Published in Neurological research (New York) (01-03-2014)
    “…The herbal extract 3-n-butylphthalide (NBP) is used in clinical practice for ischemic patients in China. It has been shown to have various neuroprotective…”
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  5. 5

    Estrogen modulation of the cyclic AMP response element-binding protein pathway. Effects of long-term and acute treatments by Carlstrom, L, Ke, Z J, Unnerstall, J R, Cohen, R S, Pandey, S C

    Published in Neuroendocrinology (01-10-2001)
    “…Actions of estrogen include mechanisms leading to alterations in gene transcription that may be independent of nuclear estrogen receptors, as well as those…”
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  6. 6

    Single crystal growth and electrical transport properties of η-Mo4O11 by Ke, J.Z., Yang, M., Zuo, H.K., Zhu, H.P., Liu, C.B., Chen, R., Dong, C., Liu, W.X., Shi, M.Y., Wang, J.F.

    Published in Journal of alloys and compounds (15-09-2020)
    “…Single crystals of quasi-two-dimensional charge-density-wave (CDW) material η-Mo4O11 were successfully grown by chemical vapor transport (CVT) method with…”
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  7. 7

    Apolipoprotein E is a genetic risk factor for fetal iodine deficiency disorder in China by WANG, H. Y, ZHANG, F. C, KE, Z. J, GUO, G. R, FENG, G. Y, BREEN, G, ST CLAIR, D, HE, L, GAO, J. J, FAN, J. B, LIU, P, ZHENG, Z. J, XI, H, SUN, Y, GAO, X. C, HUANG, T. Z

    Published in Molecular psychiatry (01-07-2000)
    “…Fetal iodine deficiency disorder (FIDD) is the principal form of endemic cretinism, and the most common cause of preventable mental deficiency in the world…”
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  8. 8

    Selective response of various brain cell types during neurodegeneration induced by mild impairment of oxidative metabolism by Ke, Zun-Ji, Gibson, Gary E

    Published in Neurochemistry international (01-07-2004)
    “…Age-related neurodegenerative diseases are characterized by selective neuron loss, glial activation, inflammation and abnormalities in oxidative metabolism…”
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  9. 9

    Reversal of Thiamine Deficiency-Induced Neurodegeneration by KE, ZUN-JI, DEGIORGIO, LORRAINE A, VOLPE, BRUCE T, GIBSON, GARY E

    “…Neurodegenerative diseases are characterized by abnormalities in oxidative processes, region-selective neuron loss, and diminished thiamine-dependent enzymes…”
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  10. 10

    CD40–CD40L interactions promote neuronal death in a model of neurodegeneration due to mild impairment of oxidative metabolism by Ke, Zun-Ji, Calingasan, Noel Y., DeGiorgio, Lorraine A., Volpe, Bruce T., Gibson, Gary E.

    Published in Neurochemistry international (01-08-2005)
    “…Abnormalities in oxidative processes, region-selective neuron loss, inflammation and diminished activity of thiamine-dependent enzymes characterize age-related…”
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  11. 11

    CD40L deletion delays neuronal death in a model of neurodegeneration due to mild impairment of oxidative metabolism by Ke, Zun-Ji, Calingasan, Noel Y., Karuppagounder, Saravanan S., DeGiorgio, Lorraine A., Volpe, Bruce T., Gibson, Gary E.

    Published in Journal of neuroimmunology (01-07-2005)
    “…Inflammatory/immune processes are important in the pathogenesis of neurodegenerative diseases. Thiamine deficiency (TD) models the region selective neuronal…”
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  12. 12

    Synthesis and physical properties of the theoretically predicted spin-triplet superconductor Li0.9Mo6O17 by Ke, J.Z., Dong, C., Zhu, H.P., Liu, W.X., Shi, M.Y., Du, Y.Q., Wang, J.F., Yang, M.

    Published in Ceramics international (15-09-2021)
    “…We report the single crystal growth and characterization of the quasi-one-dimensional superconductor Li0.9Mo6O17 via temperature-gradient flux method. The…”
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  13. 13

    The neuroethological basis of learning and memory decline in aged rats by Ke, Z J

    Published in Sheng li kʻo hsüeh chin chan (01-10-1997)
    “…The spatial reference and working memory abilities, GFAP-positive astrocytes and GABAergic interneurons were studied quantitatively in young, aged…”
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    Senescence accelerated mouse strain is sensitive to neurodegeneration induced by mild impairment of oxidative metabolism by Zhang, Qipeng, Ding, Hanqing, Li, Wenxia, Fan, Zhiqin, Sun, Anyang, Luo, Jia, Ke, Zun-Ji

    Published in Brain research (06-04-2009)
    “…Abstract Neuronal loss and impairment of oxidative metabolism are frequently observed in aging associated neurodegenerative diseases. Thiamine deficiency (TD)…”
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  16. 16

    The intracellular domain of CD44 promotes the fusion of macrophages by Cui, Weiguo, Ke, Juan Zhang, Zhang, Qing, Ke, Hua-Zhu, Chalouni, Cécile, Vignery, Agnès

    Published in Blood (15-01-2006)
    “…Macrophages seed all tissues in which they have the ability, in specific and rare instances, to fuse with themselves and to differentiate into osteoclasts in…”
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  17. 17

    Tricarboxylic acid cycle enzymes following thiamine deficiency by Bubber, Parvesh, Ke, Zun-Ji, Gibson, Gary E.

    Published in Neurochemistry international (01-12-2004)
    “…Thiamine (Vitamin B1) deficiency (TD) leads to memory deficits and neurological disease in animals and humans. The thiamine-dependent enzymes of the…”
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