Search Results - "Kattla, Jayesh J."

A systematic approach to protein glycosylation analysis: a path through the maze by Bones, Jonathan, Kattla, Jayesh J, Rudd, Pauline M, Mariño, Karina

“…Protein glycosylation is an important post-translational modification. It is a feature that enhances the functional diversity of proteins and influences their…”
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Genomics meets glycomics-the first GWAS study of human N-Glycome identifies HNF1α as a master regulator of plasma protein fucosylation by Lauc, Gordan, Essafi, Abdelkader, Huffman, Jennifer E, Hayward, Caroline, Knežević, Ana, Kattla, Jayesh J, Polašek, Ozren, Gornik, Olga, Vitart, Veronique, Abrahams, Jodie L, Pučić, Maja, Novokmet, Mislav, Redžić, Irma, Campbell, Susan, Wild, Sarah H, Borovečki, Fran, Wang, Wei, Kolčić, Ivana, Zgaga, Lina, Gyllensten, Ulf, Wilson, James F, Wright, Alan F, Hastie, Nicholas D, Campbell, Harry, Rudd, Pauline M, Rudan, Igor

“…Over half of all proteins are glycosylated, and alterations in glycosylation have been observed in numerous physiological and pathological processes. Attached…”
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The O-Linked Glycome and Blood Group Antigens ABO on Mucin-Type Glycoproteins in Mucinous and Serous Epithelial Ovarian Tumors by Vitiazeva, Varvara, Kattla, Jayesh J, Flowers, Sarah A, Lindén, Sara K, Premaratne, Pushpa, Weijdegård, Birgitta, Sundfeldt, Karin, Karlsson, Niclas G

“…Mucins are heavily O-glycosylated proteins where the glycosylation has been shown to play an important role in cancer. Normal epithelial ovarian cells do not…”
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Allelic Depletion of grem1 Attenuates Diabetic Kidney Disease by Roxburgh, Sarah A., Kattla, Jayesh J., Curran, Simon P., O'Meara, Yvonne M., Pollock, Carol A., Goldschmeding, Roel, Godson, Catherine, Martin, Finian, Brazil, Derek P.

“…Allelic Depletion of grem1 Attenuates Diabetic Kidney Disease Sarah A. Roxburgh 1 , Jayesh J. Kattla 1 , Simon P. Curran 1 , Yvonne M. O'Meara 1 , 2 , Carol A…”
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Mutations in HNF1A Result in Marked Alterations of Plasma Glycan Profile by THANABALASINGHAM, Gaya, HUFFMAN, Jennifer E, HASSANALI, Neelam, PUCIC, Maja, BENNETT, Amanda J, ESSAFI, Abdelkader, POLASEK, Ozren, MUGHAL, Saima A, REDZIC, Irma, PRIMORAC, Dragan, ZGAGA, Lina, KOLCIC, Ivana, KATTLA, Jayesh J, HANSEN, Torben, GASPERIKOVA, Daniela, TJORA, Erling, STRACHAN, Mark W. J, NIELSEN, Trine, STANIK, Juraj, KLIMES, Iwar, PEDERSEN, Oluf B, NJØLSTAD, Pål R, WILD, Sarah H, NOVOKMET, Mislav, GYLLENSTEN, Ulf, GORNIK, Olga, WILSON, James F, HASTIE, Nicholas D, CAMPBELL, Harry, MCCARTHY, Mark I, RUDD, Pauline M, OWEN, Katharine R, LAUC, Gordan, WRIGHT, Alan F, RUDAN, Igor, GLOYN, Anna L, HAYWARD, Caroline, ADAMCZYK, Barbara, REYNOLDS, Rebecca M, MUZINIC, Ana

“…A recent genome-wide association study identified hepatocyte nuclear factor 1-α (HNF1A) as a key regulator of fucosylation. We hypothesized that…”
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Association of Medication with the Human Plasma N-Glycome by Saldova, Radka, Huffman, Jennifer E, Adamczyk, Barbara, Mužinić, Ana, Kattla, Jayesh J, Pučić, Maja, Novokmet, Mislav, Abrahams, Jodie L, Hayward, Caroline, Rudan, Igor, Wild, Sarah H, Wright, Alan F, Polašek, Ozren, Lauc, Gordan, Campbell, Harry, Wilson, James F, Rudd, Pauline M

“…Glycosylation is highly variable depending on many environmental factors. Using our fully quantitative high-throughput normal phase hydrophilic interaction…”
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Protein kinase B/Akt activity is involved in renal TGF-beta1-driven epithelial-mesenchymal transition in vitro and in vivo by Kattla, Jayesh J, Carew, Rosemarie M, Heljic, Mediha, Godson, Catherine, Brazil, Derek P

“…The molecular pathogenesis of diabetic nephropathy (DN), the leading cause of end-stage renal disease worldwide, is complex and not fully understood…”
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Allelic Depletion of grrem1 Attenuates Diabetic Kidney Disease by ROXBURGH, Sarah A, KATTLA, Jayesh J, CURRAN, Simon P, O'MEARA, Yvonne M, POLLOCK, Carol A, GOLDSCHMEDING, Roel, GODSON, Catherine, MARTIN, Finian, BRAZIL, Derek P

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Genomics Meets Glycomics--The First GWAS Study of Human N-Glycome Identifies HNF1[alpha] as a Master Regulator of Plasma Protein Fucosylation: e1001256 by Lauc, Gordan, Essafi, Abdelkader, Huffman, Jennifer E, Hayward, Caroline, Knezevic, Ana, Kattla, Jayesh J, Polasek, Ozren, Gornik, Olga, Vitart, Veronique, Abrahams, Jodie L, Pucic, Maja, Novokmet, Mislav, Redzic, Irma, Campbell, Susan, Wild, Sarah H, Borovecki, Fran, Wang, Wei, Kolcic, Ivana, Zgaga, Lina, Gyllensten, Ulf, Wilson, James F, Wright, Alan F, Hastie, Nicholas D, Campbell, Harry, Rudd, Pauline M, Rudan, Igor

“…Over half of all proteins are glycosylated, and alterations in glycosylation have been observed in numerous physiological and pathological processes. Attached…”
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Genomics meets glycomics--the first GWAS study of human N-glycome identifies HNF1[alpha] as a master regulator of plasma protein fucosylation by Lauc, Gordan, Essafi, Abdelkader, Huffman, Jennifer E, Hayward, Caroline, Knezevic, Ana, Kattla, Jayesh J, Polasek, Ozren, Gornik, Olga, Vitart, Veronique, Abrahams, Jodie L, Pucic, Maja, Novokmet, Mislav, Redzic, Irma, Campbell, Susan, Wild, Sarah H, Borovecki, Fran, Wang, Wei, Kolcic, Ivana, Zgaga, Lina, Gyllensten, Ulf, Wilson, James F, Wright, Alan F, Hastie, Nicholas D, Campbell, Harry, Rudd, Pauline M, Rudan, Igor

“…Over half of all proteins are glycosylated, and alterations in glycosylation have been observed in numerous physiological and pathological processes. Attached…”
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Enhanced sensitivity of protein kinase B/Akt to insulin in hypoxia is independent of HIF1 α and promotes cell viability by Barry, Robert E., Allan, Bernard B., Cummins, Eoin P., Kattla, Jayesh J., Giblin, Aoife, Scally, Niamh, Taylor, Cormac T., Brazil, Derek P.

“…Maintenance of oxygen homeostasis is a key requirement to ensure normal mammalian cell growth and differentiation. Hypoxia arises when oxygen demand exceeds…”
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Enhanced sensitivity of protein kinase B/Akt to insulin in hypoxia is independent of HIF1alpha and promotes cell viability by Barry, Robert E, Allan, Bernard B, Cummins, Eoin P, Kattla, Jayesh J, Giblin, Aoife, Scally, Niamh, Taylor, Cormac T, Brazil, Derek P

“…Maintenance of oxygen homeostasis is a key requirement to ensure normal mammalian cell growth and differentiation. Hypoxia arises when oxygen demand exceeds…”
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