Search Results - "KINNEY, Hannah C"
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The Near-Term (Late Preterm) Human Brain and Risk for Periventricular Leukomalacia: A Review
Published in Seminars in perinatology (01-04-2006)“…Historically the major focus in neonatal neurology has been on brain injury in premature infants born less than 30 gestational weeks. This focus reflects the…”
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The developing oligodendrocyte: key cellular target in brain injury in the premature infant
Published in International journal of developmental neuroscience (01-06-2011)“…► Premyelinating oligodendrocyte the key cellular target in white matter injury in premature infants. ► Pathogenesis of injury is initiated by two initiating…”
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3
The Human Connectome Project and beyond: Initial applications of 300mT/m gradients
Published in NeuroImage (Orlando, Fla.) (15-10-2013)“…The engineering of a 3T human MRI scanner equipped with 300mT/m gradients – the strongest gradients ever built for an in vivo human MRI scanner – was a major…”
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Neuroanatomic Connectivity of the Human Ascending Arousal System Critical to Consciousness and Its Disorders
Published in Journal of neuropathology and experimental neurology (01-06-2012)“…ABSTRACTThe ascending reticular activating system (ARAS) mediates arousal, an essential component of human consciousness. Lesions of the ARAS cause coma, the…”
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Risk Factor Changes for Sudden Infant Death Syndrome After Initiation of Back-to-Sleep Campaign
Published in Pediatrics (Evanston) (01-04-2012)“…To test the hypothesis that the profile of sudden infant death syndrome (SIDS) changed after the Back-to-Sleep (BTS) campaign initiation, document prevalence…”
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Neuron deficit in the white matter and subplate in periventricular leukomalacia
Published in Annals of neurology (01-03-2012)“…Objective: The cellular basis of cognitive abnormalities in preterm infants with periventricular leukomalacia (PVL) is uncertain. One important possibility is…”
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High serum serotonin in sudden infant death syndrome
Published in Proceedings of the National Academy of Sciences - PNAS (18-07-2017)“…Sudden infant death syndrome (SIDS), the leading cause of postneonatal infant mortality, likely comprises heterogeneous disorders with the common phenotype of…”
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The brainstem and serotonin in the sudden infant death syndrome
Published in Annual review of pathology (01-01-2009)“…The sudden infant death syndrome (SIDS) is the sudden death of an infant under one year of age that is typically associated with sleep and that remains…”
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Late Development of the GABAergic System in the Human Cerebral Cortex and White Matter
Published in Journal of neuropathology and experimental neurology (01-10-2011)“…Despite the key role of γ-aminobutyric acid (GABA) neurons in the modulation of cerebral cortical output, little is known about their development in the human…”
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Dysregulation of platelet serotonin, 14–3–3, and GPIX in sudden infant death syndrome
Published in Scientific reports (15-05-2024)“…Sudden infant death syndrome (SIDS) is the leading cause of post-neonatal infant mortality, but the underlying cause(s) are unclear. A subset of SIDS infants…”
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The Cerebral Cortex Overlying Periventricular Leukomalacia: Analysis of Pyramidal Neurons
Published in Brain pathology (Zurich, Switzerland) (01-07-2010)“…The role of the cerebral cortex in the cognitive deficits in preterm survivors is poorly understood. Periventricular leukomalacia (PVL), the key feature of…”
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The Encephalopathy of Prematurity: One Pediatric Neuropathologist's Perspective
Published in Seminars in pediatric neurology (01-12-2009)“…A major challenge in understanding brain injury in the premature brain is the establishment of the precise human neuropathology at the cellular and molecular…”
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Reflections on a Career in Pediatric Neuropathology, with a Note of Gratitude
Published in Annual review of pathology (16-07-2024)“…I am honored to be asked by the journal to write this personal essay about my career in pediatric neuropathology-a life of immense satisfaction, meaning, and…”
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Nitrosative and Oxidative Injury to Premyelinating Oligodendrocytes in Periventricular Leukomalacia
Published in Journal of neuropathology and experimental neurology (01-05-2003)“…Periventricular leukomalacia (PVL), the major substrate of cerebral palsy in survivors of prematurity, is defined as focal periventricular necrosis and diffuse…”
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Reprint of “The developing oligodendrocyte: key cellular target in brain injury in the premature infant”
Published in International journal of developmental neuroscience (01-10-2011)“…► Premyelinating oligodendrocyte the key cellular target in white matter injury in premature infants. ► Pathogenesis of injury is initiated by two initiating…”
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16
Prenatal nicotine-exposure alters fetal autonomic activity and medullary neurotransmitter receptors: implications for sudden infant death syndrome
Published in Journal of applied physiology (1985) (01-11-2009)“…1 Department of Pathology, Children's Hospital Boston and Harvard Medical School, Boston, Massachusetts; ; Departments of 2 Pediatrics and ; 3 Psychiatry,…”
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Expression of EAAT2 in neurons and protoplasmic astrocytes during human cortical development
Published in Journal of comparative neurology (1911) (01-12-2012)“…The major regulators of synaptic glutamate in the cerebral cortex are the excitatory amino acid transporters 1–3 (EAAT1–3). In this study, we determined the…”
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Concurrent prenatal drinking and smoking increases risk for SIDS: Safe Passage Study report
Published in EClinicalMedicine (01-02-2020)“…Sudden infant death syndrome (SIDS) is the leading cause of postneonatal mortality. Although the rate has plateaued, any unexpected death of an infant is a…”
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Potential asphyxia and brainstem abnormalities in sudden and unexpected death in infants
Published in Pediatrics (Evanston) (01-12-2013)“…Sudden and unexplained death is a leading cause of infant mortality. Certain characteristics of the sleep environment increase the risk for sleep-related…”
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Axonal development in the cerebral white matter of the human fetus and infant
Published in Journal of comparative neurology (1911) (04-04-2005)“…After completion of neuronal migration to form the cerebral cortex, axons undergo rapid elongation to their intra‐ and subcortical targets, from midgestation…”
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