Search Results - "KATZE, M. G"

Functional Genomic and Serological Analysis of the Protective Immune Response Resulting from Vaccination of Macaques with an NS1-Truncated Influenza Virus by Baskin, C. R., Bielefeldt-Ohmann, H., García-Sastre, A., Tumpey, T. M., Van Hoeven, N., Carter, V. S., Thomas, M. J., Proll, S., Solórzano, A., Billharz, R., Fornek, J. L., Thomas, S., Chen, C.-H., Clark, E. A., Murali-Krishna, Kaja, Katze, M. G.

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Integrated Molecular Signature of Disease: Analysis of Influenza Virus-Infected Macaques through Functional Genomics and Proteomics by BAAS, T, BASKIN, C. R, PROLL, S, JACOBS, J. M, CALDWELL, Z. R, GRITSENKO, M. A, HUKKANEN, R. R, CAMP, D. G, SMITH, R. D, KATZE, M. G, DIAMOND, D. L, GARCIA-SASTRE, A, BIELEFELDT-OHMANN, H, TUMPEY, T. M, THOMAS, M. J, CARTER, V. S, TEAL, T. H, VAN HOEVEN, N

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Translational Control of Viral Gene Expression in Eukaryotes by GALE, M. JR, TAN, S.-L, KATZE, M. G

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Extrapulmonary tissue responses in cynomolgus macaques (Macaca fascicularis) infected with highly pathogenic avian influenza A (H5N1) virus by Tolnay, A.-E., Baskin, C. R., Tumpey, T. M., Sabourin, P. J., Sabourin, C. L., Long, J. P., Pyles, J. A., Albrecht, R. A., García-Sastre, A., Katze, M. G., Bielefeldt-Ohmann, H.

“…The mechanisms responsible for virulence of influenza viruses in humans remain poorly understood. A prevailing hypothesis is that the highly pathogenic virus…”
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Tumor Suppressor Function of the Interferon-Induced Double-Stranded RNA-Activated Protein Kinase by Meurs, Eliane F., Galabru, Julien, Barber, Glen N., Katze, Michael G., Hovanessian, Ara G.

“…RNA-dependent protein kinase is a Mr68,000 protein in human cells (p68 kinase) or a Mr65,000 protein in murine cells (p65 kinase). p65/p68 is a…”
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Global Impact of Influenza Virus on Cellular Pathways Is Mediated by both Replication-Dependent and -Independent Events by Geiss, G K, An, M C, Bumgarner, R E, Hammersmark, E, Cunningham, D, Katze, M G

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Mutants of the RNA-dependent protein kinase (PKR) lacking double-stranded RNA binding domain I can act as transdominant inhibitors and induce malignant transformation by Barber, Glen N., Wambach, Marlene, Thompson, Samantha, Jagus, Rosemary, Katze, Michael G.

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The 58,000-dalton cellular inhibitor of the interferon-induced double-stranded RNA-activated protein kinase (PKR) is a member of the tetratricopeptide repeat family of proteins by Lee, T G, Tang, N, Thompson, S, Miller, J, Katze, M G

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Malignant Transformation by a Mutant of the IFN-Inducible dsRNA-Dependent Protein Kinase by Koromilas, Antonis E., Roy, Sophie, Barber, Glen N., Katze, Michael G., Sonenberg, Nahum

“…The double-stranded RNA-dependent protein kinase (dsRNA-PK) is thought to be a key mediator of the antiviral and antiproliferative effects of interferons…”
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Viruses and interferon: a fight for supremacy by Katze, Michael G, He, Yupeng, Gale, Michael

“…The action of interferons (IFNs) on virus-infected cells and surrounding tissues elicits an antiviral state that is characterized by the expression and…”
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Mammalian Eukaryotic Initiation Factor 2α Kinases Functionally Substitute for GCN2 Protein Kinase in the GCN4 Translational Control Mechanism of Yeast by Dever, Thomas E., Chen, Jane-Jane, Barber, Glen N., Cigan, A. Mark, Feng, Lan, Donahue, Thomas F., London, Irving M., Katze, Michael G., Hinnebusch, Alan G.

“…Phosphorylation of the α subunit of eukaryotic initiation factor 2 (eIF-2α) in Saccharomyces cerevisiae by the GCN2 protein kinase stimulates the translation…”
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Functional expression and RNA binding analysis of the interferon-induced, double-stranded RNA-activated, 68,000-Mr protein kinase in a cell-free system by KATZE, M. G, WAMBACH, M, MIN-LIANG WONG, GARFINKEL, M, MEURS, E, CHONG, K, WILLIAMS, B. R. G, HOVANESSIAN, A. G, BARBER, G. N

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The cellular 68,000-Mr protein kinase is highly autophosphorylated and activated yet significantly degraded during poliovirus infection: implications for translational regulation by BLACK, T. L, SAFER, B, HOVANESSIAN, A, KATZE, M. G

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Purification and partial characterization of a cellular inhibitor of the interferon-induced protein kinase of Mr 68,000 from influenza virus-infected cells by TAE GYU LEE, TOMITA, J, HOVANESSIAN, A. G, KATZE, M. G

“…A number of eukaryotic viruses have evolved mechanisms to downregulate activity of the interferon-induced, double-stranded RNA-activated protein kinase…”
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Characterization and regulation of the 58,000-dalton cellular inhibitor of the interferon-induced, dsRNA-activated protein kinase by TAE GYU LEE, TOMITA, J, HOVANESSIAN, A. G, KATZE, M. G

“…The P68 protein kinase is a serine/threonine kinase induced by interferon treatment and activated by double-stranded RNAs (dsRNAs). Once activated, the kinase…”
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Translational Regulation by the Interferon-Induced Double-Stranded-RNA- Activated 68-kDa Protein Kinase by Barber, Glen N., Wambach, Marlene, Wong, Min-Liang, Dever, Thomas E., Hinnebusch, Alan G., Katze, Michael G.

“…Activation of the interferon-inducible 68-kDa protein kinase (referred to as P68) by double-stranded RNA catalyzes phosphorylation of the α subunit of…”
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Evading the interferon response: hepatitis C virus and the interferon-induced protein kinase, PKR by Korth, M J, Katze, M G

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The 58-Kilodalton Inhibitor of the Interferon-Induced Double-Stranded RNA Activated Protein Kinase is a Tetratricopeptide Repeat Protein with Oncogenic Properties by Barber, Glen N., Thompson, Samantha, Lee, Tae Gyu, Strom, Ted, Jagus, Rosemary, Darveau, Andre, Katze, Michael G.

“…The interferon-induced RNA-dependent protein kinase (PKR) is considered to play an important role in the cellular defense against viral infection and, in…”
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The integrity of the stem structure of human immunodeficiency virus type 1 Tat-responsive sequence of RNA is required for interaction with the interferon-induced 68,000-Mr protein kinase by ROY, S, AGY, M, HOVANESSIAN, A. G, SONENBERG, N, KATZE, M. G

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Infection of Macaca nemestrina by Human Immunodeficiency Virus Type-1 by Agy, Michael B., Frumkin, Lyn R., Corey, Lawrence, Coombs, Robert W., Wolinsky, Steven M., Koehler, James, Morton, William R., Katze, Michael G.

“…After observations that Macaca nemestrina were exceptionally susceptible to simian immunodeficiency virus and human immunodeficiency virus type-2 (HIV-2),…”
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