Search Results - "Jeraj, Natasha"

Apolipoprotein genetic variants and hereditary amyloidosis by Jeraj, Natasha, Hegele, Robert A., Berberich, Amanda J.

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Apolipoprotein genetic variants and hereditary amyloidosis by Jeraj, Natasha, Hegele, Robert A., Berberich, Amanda J.

“…Amyloidosis is caused by the deposition of misfolded aggregated proteins called amyloid fibrils that in turn cause organ damage and dysfunction. In this…”
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Treatment of Homozygous Familial Hypercholesterolemia With EvinacumabNovel Teaching Points by Natasha Jeraj, BSc, Shih-Han S. Huang, MD, Brooke A. Kennedy, BSc, Robert A. Hegele, MD

“…Patients with homozygous familial hypercholesterolemia (HoFH) have extremely elevated levels of low-density lipoprotein cholesterol (LDL-C), with premature…”
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Treatment of Homozygous Familial Hypercholesterolemia With Evinacumab by Jeraj, Natasha, Huang, Shih-Han S., Kennedy, Brooke A., Hegele, Robert A.

“…Patients with homozygous familial hypercholesterolemia (HoFH) have extremely elevated levels of low-density lipoprotein cholesterol (LDL-C), with premature…”
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Hepatocyte-derived DPP4 regulates portal GLP-1 bioactivity, modulates glucose production, and when absent influences NAFLD progression by Trzaskalski, Natasha A, Vulesevic, Branka, Nguyen, My-Anh, Jeraj, Natasha, Fadzeyeva, Evgenia, Morrow, Nadya M, Locatelli, Cassandra Aa, Travis, Nicole, Hanson, Antonio A, Nunes, Julia Rc, O'Dwyer, Conor, van der Veen, Jelske N, Lorenzen-Schmidt, Ilka, Seymour, Rick, Pulente, Serena M, Clément, Andrew C, Crawley, Angela M, Jacobs, René L, Doyle, Mary-Anne, Cooper, Curtis L, Kim, Kyoung-Han, Fullerton, Morgan D, Mulvihill, Erin E

“…Elevated circulating dipeptidyl peptidase-4 (DPP4) is a biomarker for liver disease, but its involvement in gluconeogenesis and metabolic associated fatty…”
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Treatment of Homozygous Familial Hypercholesterolemia With Evinacumab by Jeraj, Natasha, Huang, Shih-Han S, Kennedy, Brooke A, Hegele, Robert A

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