Search Results - "Jangde, Nitish"

p32 promotes melanoma progression and metastasis by targeting EMT markers, Akt/PKB pathway, and tumor microenvironment by Sinha, Sunita, Singh, Satyendra Kumar, Jangde, Nitish, Ray, Rashmi, Rai, Vivek

“…Melanoma originates from melanin-producing cells called melanocytes. Melanoma poses a great risk because of its rapid ability to spread and invade new organs…”
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MYH9 suppresses melanoma tumorigenesis, metastasis and regulates tumor microenvironment by Singh, Satyendra Kumar, Sinha, Sunita, Padhan, Jyotirmayee, Jangde, Nitish, Ray, Rashmi, Rai, Vivek

“…Non-muscle myosin IIA heavy chain (MYH9) has been implicated in many physiological and pathological functions including cell adhesion, polarity, motility to…”
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Lysophosphatidic acid-RAGE axis promotes lung and mammary oncogenesis via protein kinase B and regulating tumor microenvironment by Ray, Rashmi, Jangde, Nitish, Singh, Satyendra Kumar, Sinha, Sunita, Rai, Vivek

“…Receptor for advanced glycation end products (RAGE) is a multi-ligand transmembrane receptor of the immunoglobulin superfamily. Lysophosphatidic acid (LPA) is…”
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MDB-37. RAC1 INHIBITION FOR THE TREATMENT OF MEDULLOBLASTOMA by Ayad, Nagi, Jangde, Nitish

“…Despite considerable advances in identifying genetic and epigenetic abnormalities in medulloblastoma, many patients succumb to the disease. In addition,…”
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MDB-41. TARGETING EPIGENETIC REGULATORS TO INHIBIT GROUP 3 MEDULLOBLASTOMA MIGRATION by Jangde, Nitish, Walter, Thomas Jordan, Ayad, Nagi

“…Abstract Medulloblastoma highly malignant type of childhood brain cancer which develops at the cerebellum. Medulloblastoma can be grouped into four molecular…”
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EPCO-36. INVESTIGATING THE BAIAP2-CDC42 INTERACTION IN MEDULLOBLASTOMA by Ruiz, Luz, Jangde, Nitish, Ayad, Nagi

“…Abstract Medulloblastoma is the most common malignant brain tumor found in children. The standard of care is surgical removal, radiation, and chemotherapy…”
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MODL-14. TARGETING GL1/GL2-DNMT1-UHRF1 EPIGENETIC COMPLEX DISRUPTION VIA BET INHIBITION IMPAIRS SHH MB DEVELOPMENT by Jangde, Nitish, Wynn, Daniel, Egan, Isabelle, Robbins, David, Ayad, Nagi

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ANGI-09. UNRAVELING THE INVOLVEMENT OF BAIAP2 AND CDC42 IN MEDULLOBLASTOMA PATHOGENESIS by Ruiz, Luz, Jangde, Nitish, Suter, Robert, Ayad, Nagi

“…Abstract The most common malignant pediatric brain tumor is medulloblastoma. Despite advancements in treatment, medulloblastoma survivors have significant…”
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Abstract 4307: RAGE is the key receptor for LPA in lung and mammary cancer progression and metastasis by Jangde, Nitish, Ray, Rashmi, Rai, Vivek

“…Abstract Lung and breast cancer signaling are very complex; receptor for advanced glycation end products (RAGE) is highly expressed in various cancers and is…”
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MDB-01. ELUCIDATING THE ROLE OF THE BAIAP2-CDC42 INTERACTION MEDULLOBLASTOMA by Ruiz, Luz, Suter, Rober, Jangde, Nitish, Ayad, Nagi

“…Abstract Medulloblastoma is the most common malignant pediatric brain tumor. Despite advancements in treatment, there is still no significant improvement in…”
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EPCO-37. IDENTIFICATION OF KEY EPIGENETIC REGULATORS FOR MEDULLOBLASTOMA INVASION AND METASTASIS by Jangde, Nitish, Walter, T Jordan, Ruiz, Luz, Ayad, Nagi

“…Abstract Medulloblastoma (MB) is most common type of pediatric brain cancer. It starts from the hind brain or cerebellum. It is actually an embryonic malignant…”
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MDB-85. THE MIGRATORY ROLE OF BAIAP2 IN MEDULLOBLASTOMA by Ruiz, Luz C, Jangde, Nitish, Suter, Robert, Jermacowikz, Anna, Ayad, Nagi

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BSCI-19 A LENTIVIRAL CRISPR SCREEN FOR EPIGENETIC MODULATORS OF ANTIGENS TARGETED BY CAR T CELLS IN GLIOBLASTOMA by Jordan Walter, T, Jangde, Nitish, Ayad, Nagi

“…Abstract Glioblastoma (GBM) is the most common adult brain tumor and is very difficult to treat. One promising treatment strategy is CAR T cell therapy, in…”
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Cysteine mediated disulfide bond formation in RAGE V domain facilitates its functionally relevant dimerization by Jangde, Nitish, Ray, Rashmi, Sinha, Sunita, Rana, Khokan, Singh, Satyendra Kumar, Khandagale, Prashant, Acharya, Narottam, Rai, Vivek

“…Receptor for Advanced Glycation End product (RAGE) is a multiligand receptor implicated in diverse pathological conditions such as diabetes, atherosclerosis,…”
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