Search Results - "Huycke, M M"
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1
Defective Intestinal Mucin-Type O-Glycosylation Causes Spontaneous Colitis-Associated Cancer in Mice
Published in Gastroenterology (New York, N.Y. 1943) (01-07-2016)“…Background & Aims Core 1– and core 3–derived mucin-type O-linked oligosaccharides (O-glycans) are major components of the colonic mucus layer. Defective forms…”
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2
Glutathione S-transferase alpha 4 induction by activator protein 1 in colorectal cancer
Published in Oncogene (03-11-2016)“…Glutathione S -transferase alpha 4 (GSTA4) is a phase II detoxifying enzyme that metabolizes electrophiles and carcinogens including 4-hydroxy-2-nonenal…”
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3
It takes a village: microbiota, parainflammation, paligenosis and bystander effects in colorectal cancer initiation
Published in Disease models & mechanisms (01-05-2021)“…Sporadic colorectal cancer (CRC) is a leading cause of worldwide cancer mortality. It arises from a complex milieu of host and environmental factors, including…”
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4
Enterococcus faecalis produces extracellular superoxide and hydrogen peroxide that damages colonic epithelial cell DNA
Published in Carcinogenesis (New York) (01-03-2002)“…Enterococcus faecalis is a commensal microorganism of the human intestinal tract that produces substantial extracellular superoxide (O2−), and derivative…”
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5
Enterococcus faecalis induces aneuploidy and tetraploidy in colonic epithelial cells through a bystander effect
Published in Cancer research (Chicago, Ill.) (01-12-2008)“…Intestinal commensals are potential important contributors to the etiology of sporadic colorectal cancer, but mechanisms by which bacteria can initiate tumors…”
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6
Adenoid cystic carcinoma of the breast in the United States (1977 to 2006): a population-based cohort study
Published in Breast cancer research : BCR (01-01-2010)“…Adenoid cystic carcinoma of the breast (breast-ACC) is a rare and special type of basal-like tumor for which scant population-based descriptive data exist. We…”
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7
Commensal bacteria, redox stress, and colorectal cancer: mechanisms and models
Published in Experimental biology and medicine (Maywood, N.J.) (01-07-2004)“…The potential role for commensal bacteria in colorectal carcinogenesis is explored in this review. Most colorectal cancers (CRCs) occur sporadically and arise…”
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8
Multiple-drug resistant enterococci: the nature of the problem and an agenda for the future
Published in Emerging infectious diseases (01-06-1998)“…Enterococci, leading causes of nosocomial bacteremia, surgical wound infection, and urinary tract infection, are becoming resistant to many and sometimes all…”
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9
Variable phenotypes of enterocolitis in interleukin 10-deficient mice monoassociated with two different commensal bacteria
Published in Gastroenterology (New York, N.Y. 1943) (01-04-2005)“…Background & Aims: To explore the hypothesis that selective immune responses to distinct components of the intestinal microflora induce intestinal…”
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10
Small Intestinal Cancer: a Population-Based Study of Incidence and Survival Patterns in the United States, 1992 to 2006
Published in Cancer epidemiology, biomarkers & prevention (01-08-2010)“…The etiology of cancers of the small intestine is largely unknown. To gain insight into these rare malignancies, we evaluated contemporaneous incidence and…”
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11
Simplified agar plate method for quantifying viable bacteria
Published in BioTechniques (01-10-1997)Get full text
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12
Outbreak of Invasive Aspergillus Infection in Surgical Patients, Associated with a Contaminated Air-Handling System
Published in Clinical infectious diseases (15-09-2003)“…An outbreak of Aspergillus infection at a tertiary care hospital was identified among inpatients who had amputation wounds, peritonitis, allograft nephritis,…”
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13
Extracellular superoxide production by Enterococcus faecalis requires demethylmenaquinone and is attenuated by functional terminal quinol oxidases
Published in Molecular microbiology (01-11-2001)“…The intestinal commensal bacterium, Enterococcus faecalis, is unusual among prokaryotic organisms in its ability to produce substantial extracellular…”
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14
Bacteria-induced intestinal cancer in mice with disrupted Gpx1 and Gpx2 genes
Published in Cancer research (Chicago, Ill.) (01-02-2004)“…Two glutathione peroxidase (GPX) isozymes, GPX-1 and GPX-2 (GPX-GI), are the major enzymes that reduce hydroperoxides in intestinal epithelium. We have…”
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15
Cyclooxygenase-2 Generates the Endogenous Mutagen trans-4-Hydroxy-2-nonenal in Enterococcus faecalis―Infected Macrophages
Published in Cancer prevention research (Philadelphia, Pa.) (01-03-2013)“…Infection of macrophages by the human intestinal commensal Enterococcus faecalis generates DNA damage and chromosomal instability in mammalian cells through…”
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16
Infection-derived Enterococcus faecalis strains are enriched in esp, a gene encoding a novel surface protein
Published in Infection and immunity (1999)“…We report the identification of a new cell wall-associated protein of Enterococcus faecalis. Studies on the distribution of the gene encoding this novel…”
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17
Primary cutaneous adenoid cystic carcinoma in the United States: Incidence, survival, and associated cancers, 1976 to 2005
Published in Journal of the American Academy of Dermatology (01-07-2010)“…Background Primary cutaneous adenoid cystic carcinoma (PCACC) is a rare appendageal tumor of uncertain origin. Details on epidemiologic features of PCACC are…”
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18
Virulence of enterococci
Published in Clinical Microbiology Reviews (01-10-1994)“…Classifications Services CMR Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit…”
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19
Stump appendicitis
Published in Radiology case reports (01-07-2022)“…Stump appendicitis is a rare and late complication following appendectomy and can often be overlooked. Our case details a 42-year-old male who presented to the…”
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20
Microbiome-driven carcinogenesis in colorectal cancer: Models and mechanisms
Published in Free radical biology & medicine (01-04-2017)“…Colorectal cancer (CRC) is a leading cause of cancer death and archetype for cancer as a genetic disease. However, the mechanisms for genetic change and their…”
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