Search Results - "Hoveida, Shirin"

Mutations in TP53 or DNA damage repair genes define poor prognostic subgroups in primary prostate cancer by Nientiedt, Cathleen, Budczies, Jan, Endris, Volker, Kirchner, Martina, Schwab, Constantin, Jurcic, Christina, Behnisch, Rouven, Hoveida, Shirin, Lantwin, Philippa, Kaczorowski, Adam, Geisler, Christine, Dieffenbacher, Svenja, Falkenbach, Fabian, Franke, Desiree, Görtz, Magdalena, Heller, Martina, Himmelsbach, Ruth, Pecqueux, Carine, Rath, Mathias, Reimold, Philipp, Schütz, Viktoria, Simunovic, Iva, Walter, Elena, Hofer, Luisa, Gasch, Claudia, Schönberg, Gita, Pursche, Lars, Hatiboglu, Gencay, Nyarangi-Dix, Joanne, Sültmann, Holger, Zschäbitz, Stefanie, Koerber, Stefan A., Jäger, Dirk, Debus, Jürgen, Duensing, Anette, Schirmacher, Peter, Hohenfellner, Markus, Stenzinger, Albrecht, Duensing, Stefan

“…•We show that a substantial proportion of patients undergoing RPX for localized high-risk or locally advanced prostate cancer harbor mutations in DNA damage…”
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High prevalence of DNA damage repair gene defects and TP53 alterations in men with treatment-naïve metastatic prostate cancer –Results from a prospective pilot study using a 37 gene panel by Nientiedt, Cathleen, Endris, Volker, Jenzer, Maximilian, Mansour, Josef, Sedehi, Nassim Tawanaie Pour, Pecqueux, Carine, Volckmar, Anna-Lena, Leichsenring, Jonas, Neumann, Olaf, Kirchner, Martina, Hoveida, Shirin, Lantwin, Philippa, Kaltenecker, Katrin, Dieffenbacher, Svenja, Gasch, Claudia, Hofer, Luisa, Franke, Desiree, Tosev, Georgi, Görtz, Magdalena, Schütz, Viktoria, Radtke, Jan-Philipp, Nyarangi-Dix, Joanne, Hatiboglu, Gencay, Simpfendörfer, Tobias, Schönberg, Gita, Isaac, Sanjay, Teber, Dogu, Koerber, Stefan A., Christofi, Georgia, Czink, Elena, Kreuter, Rebecca, Apostolidis, Leonidas, Kratochwil, Clemens, Giesel, Frederik, Haberkorn, Uwe, Debus, Jürgen, Sültmann, Holger, Zschäbitz, Stefanie, Jäger, Dirk, Duensing, Anette, Schirmacher, Peter, Grüllich, Carsten, Hohenfellner, Markus, Stenzinger, Albrecht, Duensing, Stefan

“…•Pilot study that demonstrates the feasibility, performance and clinical relevance of somatic targeted NGS using a novel 37 DNA damage repair and checkpoint…”
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