Search Results - "HOHL, DANIEL"

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  1. 1

    The human epidermal differentiation complex: cornified envelope precursors, S100 proteins and the 'fused genes' family by Kypriotou, Magdalini, Huber, Marcel, Hohl, Daniel

    Published in Experimental dermatology (01-09-2012)
    “…:  The skin is essential for survival and protects our body against biological attacks, physical stress, chemical injury, water loss, ultraviolet radiation and…”
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    Journal Article
  2. 2

    Skin Colonization by Staphylococcus aureus Precedes the Clinical Diagnosis of Atopic Dermatitis in Infancy by Meylan, Patrick, Lang, Caroline, Mermoud, Sophie, Johannsen, Alexandre, Norrenberg, Sarah, Hohl, Daniel, Vial, Yvan, Prod’hom, Guy, Greub, Gilbert, Kypriotou, Magdalini, Christen-Zaech, Stéphanie

    Published in Journal of investigative dermatology (01-12-2017)
    “…Atopic dermatitis (AD) has a well-established association with skin colonization or infection by Staphylococcus aureus, which can exacerbate the disease…”
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  3. 3

    HOPX Exhibits Oncogenic Activity during Squamous Skin Carcinogenesis by Pavlova, Olesya, Lefort, Karine, Mariotto, Anita, Huber, Marcel, Hohl, Daniel

    Published in Journal of investigative dermatology (01-10-2021)
    “…Cutaneous squamous cell carcinomas (SCCs) are frequent heterogeneous tumors arising from sun-exposed regions of the skin and characterized by complex…”
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  4. 4

    Birt–Hogg–Dubé syndrome by Daccord, Cécile, Good, Jean-Marc, Morren, Marie-Anne, Bonny, Olivier, Hohl, Daniel, Lazor, Romain

    Published in European respiratory review (30-09-2020)
    “…Birt–Hogg–Dubé syndrome (BHD) is a rare inherited autosomal dominant disorder caused by germline mutations in the tumour suppressor gene FLCN , encoding the…”
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  5. 5

    NRF3 suppresses squamous carcinogenesis, involving the unfolded protein response regulator HSPA5 by Gurri, Selina, Siegenthaler, Beat, Cangkrama, Michael, Restivo, Gaetana, Huber, Marcel, Saliba, James, Dummer, Reinhard, Blank, Volker, Hohl, Daniel, Werner, Sabine

    Published in EMBO molecular medicine (08-11-2023)
    “…Epithelial skin cancers are extremely common, but the mechanisms underlying their malignant progression are still poorly defined. Here, we identify the NRF3…”
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    The Fate of Epidermal Tight Junctions in the stratum corneum: Their Involvement in the Regulation of Desquamation and Phenotypic Expression of Certain Skin Conditions by Haftek, Marek, Oji, Vinzenz, Feldmeyer, Laurence, Hohl, Daniel, Hadj-Rabia, Smaïl, Abdayem, Rawad

    “…We evaluated the presence of tight junction (TJ) remnants in the stratum corneum (SC) of in vitro reconstructed human epidermis and human skin explants…”
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  8. 8

    Challenges in Treating Genodermatoses: New Therapies at the Horizon by Morren, Marie-Anne, Legius, Eric, Giuliano, Fabienne, Hadj-Rabia, Smail, Hohl, Daniel, Bodemer, Christine

    Published in Frontiers in pharmacology (05-01-2022)
    “…Genodermatoses are rare inherited skin diseases that frequently affect other organs. They often have marked effects on wellbeing and may cause early death…”
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  9. 9

    PASS Syndrome: An IL-1-Driven Autoinflammatory Disease by Leuenberger, Mathieu, Berner, Jeanne, Di Lucca, Julie, Fischer, Lara, Kaparos, Nikolaos, Conrad, Curdin, Hohl, Daniel, So, Alexander, Gilliet, Michel

    Published in Dermatology (Basel) (01-01-2016)
    “…PASS syndrome is a rare inflammatory disease characterized by a chronic-relapsing course of pyoderma gangrenosum, acne vulgaris, hidradenitis suppurativa and…”
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    Dual Role of the Antioxidant Enzyme Peroxiredoxin 6 in Skin Carcinogenesis by ROLFS, Frank, HUBER, Marcel, WERNER, Sabine, GRUBER, Florian, BÖHM, Friederike, PFISTER, Herbert J, BOCHKOV, Valery N, TSCHACHLER, Erwin, DUMMER, Reinhard, HOHL, Daniel, SCHÄFER, Matthias

    Published in Cancer research (Chicago, Ill.) (01-06-2013)
    “…The antioxidant enzyme peroxiredoxin 6 (Prdx6) is a key regulator of the cellular redox balance, particularly under stress conditions. We identified Prdx6 as…”
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    Case Report: Pansclerotic Morphea-Clinical Features, Differential Diagnoses and Modern Treatment Concepts by Ventéjou, Sarah, Schwieger-Briel, Agnes, Nicolai, Rebecca, Christen-Zaech, Stephanie, Schnider, Caroline, Hofer, Michael, Bogiatzi, Sofia, Hohl, Daniel, De Benedetti, Fabrizio, Morren, Marie-Anne

    Published in Frontiers in immunology (09-03-2021)
    “…Pansclerotic morphea (PSM) is a rare skin disease characterized by progressive stiffening of the skin with or without the typical superficial skin changes…”
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    Defects in Stratum Corneum Desquamation Are the Predominant Effect of Impaired ABCA12 Function in a Novel Mouse Model of Harlequin Ichthyosis by Zhang, Lei, Ferreyros, Michael, Feng, Weiguo, Hupe, Melanie, Crumrine, Debra A, Chen, Jiang, Elias, Peter M, Holleran, Walter M, Niswander, Lee, Hohl, Daniel, Williams, Trevor, Torchia, Enrique C, Roop, Dennis R

    Published in PloS one (23-08-2016)
    “…Harlequin Ichthyosis is a severe skin disease caused by mutations in the human gene encoding ABCA12. Here, we characterize a novel mutation in intron 29 of the…”
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    Activin promotes skin carcinogenesis by attraction and reprogramming of macrophages by Antsiferova, Maria, Piwko‐Czuchra, Aleksandra, Cangkrama, Michael, Wietecha, Mateusz, Sahin, Dilara, Birkner, Katharina, Amann, Valerie C, Levesque, Mitchell, Hohl, Daniel, Dummer, Reinhard, Werner, Sabine

    Published in EMBO molecular medicine (01-01-2017)
    “…Activin has emerged as an important player in different types of cancer, but the underlying mechanisms are largely unknown. We show here that activin…”
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    UVB-Induced Skin Inflammation and Cutaneous Tissue Injury Is Dependent on the MHC Class I–Like Protein, CD1d by Ryser, Stephan, Schuppli, Marlène, Gauthier, Beatrice, Hernandez, Dianelys R., Roye, Olivier, Hohl, Daniel, German, Bruce, Holzwarth, James A., Moodycliffe, Angus M.

    Published in Journal of investigative dermatology (01-01-2014)
    “…CD1d is a major histocompatibility complex class 1–like molecule that regulates the function and development of natural killer T (NKT) cells. Previously, we…”
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