Search Results - "Gut, Marta Ortova"
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Monoclonal antibodies generated in carbonic anhydrase IX-deficient mice recognize different domains of tumour-associated hypoxia-induced carbonic anhydrase IX
Published in Journal of immunological methods (01-11-2003)“…Transmembrane carbonic anhydrase IX (CA IX) is frequently expressed in human tumours in response to hypoxia and may serve as a tumour marker and therapeutic…”
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Gastric pit cell hyperplasia and glandular atrophy in carbonic anhydrase IX knockout mice : studies on two strains C57/BL6 and BALB/C
Published in Transgenic research (01-10-2005)“…Carbonic anhydrase (CA) isoenzyme IX is a hypoxia-inducible enzyme, which is expressed in the human and rodent gastrointestinal tract and overexpressed in…”
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Carbonic Anhydrase Isozyme-II-Deficient Mice Lack the Duodenal Bicarbonate Secretory Response to Prostaglandin E2
Published in Proceedings of the National Academy of Sciences - PNAS (18-10-2005)“…Duodenal bicarbonate secretion (DBS) is accepted as the primary mucosal defense against acid discharged from the stomach and is impaired in patients with…”
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Gastric hyperplasia in mice with targeted disruption of the carbonic anhydrase gene Car9
Published in Gastroenterology (New York, N.Y. 1943) (01-12-2002)“…Background & Aims: Carbonic anhydrase (CA) IX is a highly active enzyme with adhesion capacity that is functionally implicated in acid-base balance and…”
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Carbonic anhydrase isozyme-II-deficient mice lack the duodenal bicarbonate secretory response to prostaglandin E 2
Published in Proceedings of the National Academy of Sciences - PNAS (18-10-2005)“…Duodenal bicarbonate secretion (DBS) is accepted as the primary mucosal defense against acid discharged from the stomach and is impaired in patients with…”
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Carbonic anhydrase isozyme-II-deficient mice lack the duodenal bicarbonate secretory response to prostaglandin E sub(2)
Published in Proceedings of the National Academy of Sciences - PNAS (01-10-2005)“…Duodenal bicarbonate secretion (DBS) is accepted as the primary mucosal defense against acid discharged from the stomach and is impaired in patients with…”
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