Search Results - "Gujral, Jaspreet S"

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  1. 1

    Reduced oncotic necrosis in fas receptor‐deficient C57BL/6J‐lpr mice after bile duct ligation by Gujral, Jaspreet S., Liu, Jie, Farhood, Anwar, Jaeschke, Hartmut

    Published in Hepatology (Baltimore, Md.) (01-10-2004)
    “…Neutrophils aggravate cholestatic liver injury after bile duct ligation (BDL). Recently, it was suggested that hepatocellular apoptosis might be critical for…”
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    Journal Article
  2. 2

    Apoptosis and necrosis in liver disease by Jaeschke, Hartmut, Gujral, Jaspreet S., Bajt, Mary Lynn

    Published in Liver international (01-04-2004)
    “…: Liver cell injury and cell death is a prominent feature in all liver disease processes. During the last 5–10 years, most research activities focused almost…”
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  3. 3

    NADPH oxidase-derived oxidant stress is critical for neutrophil cytotoxicity during endotoxemia by Gujral, Jaspreet S, Hinson, Jack A, Farhood, Anwar, Jaeschke, Hartmut

    “…Neutrophils can cause liver injury during endotoxemia through generation of reactive oxygen species. However, the enzymatic source of the oxidant stress and…”
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  4. 4

    Functional importance of ICAM-1 in the mechanism of neutrophil-induced liver injury in bile duct-ligated mice by Gujral, Jaspreet S, Liu, Jie, Farhood, Anwar, Hinson, Jack A, Jaeschke, Hartmut

    “…Cholestasis-induced liver injury during bile duct obstruction causes an acute inflammatory response. To further characterize the mechanisms underlying the…”
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    Journal Article
  5. 5

    Generation and functional significance of CXC chemokines for neutrophil-induced liver injury during endotoxemia by Dorman, Robert B, Gujral, Jaspreet S, Bajt, Mary Lynn, Farhood, Anwar, Jaeschke, Hartmut

    “…The hypothesis that the neutrophil chemoattractant CXC chemokines KC and macrophage inflammatory protein-2 (MIP-2) are involved in neutrophil transmigration…”
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    Mechanism of cell death during warm hepatic ischemia-reperfusion in rats: Apoptosis or necrosis? by Gujral, Jaspreet S., Bucci, Thomas J., Farhood, Anwar, Jaeschke, Hartmut

    Published in Hepatology (Baltimore, Md.) (01-02-2001)
    “…Reperfusion injury can cause liver dysfunction after cold storage and warm ischemia. Recently it has been suggested that more than 50% of hepatocytes and…”
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  8. 8

    Mode of Cell Death after Acetaminophen Overdose in Mice: Apoptosis or Oncotic Necrosis? by Gujral, Jaspreet S., Knight, Tamara R., Farhood, Anwar, Bajt, Mary Lynn, Jaeschke, Hartmut

    Published in Toxicological sciences (01-06-2002)
    “…Acetaminophen (AAP) overdose can cause severe liver injury and liver failure in experimental animals and humans. Recently, several authors proposed that…”
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  9. 9

    Neutrophils aggravate acute liver injury during obstructive cholestasis in bile duct-ligated mice by Gujral, Jaspreet S., Farhood, Anwar, Bajt, Mary Lynn, Jaeschke, Hartmut

    Published in Hepatology (Baltimore, Md.) (01-08-2003)
    “…Obstruction of the common bile duct in a variety of clinical settings leads to cholestatic liver injury. An important aspect of this injury is hepatic…”
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  10. 10

    Water Adherence Factors for Human Skin by Gujral, Jaspreet S., Proctor, Deborah M., Su, Steave H., Fedoruk, Joseph M.

    Published in Risk analysis (01-08-2011)
    “…On incidental dermal exposure to chemicals in water, a key exposure factor is the amount of water adhering to skin. Although soil adherence factors have been…”
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  11. 11

    Oncotic necrosis and caspase-dependent apoptosis during galactosamine-induced liver injury in rats by Gujral, Jaspreet S, Farhood, Anwar, Jaeschke, Hartmut

    Published in Toxicology and applied pharmacology (01-07-2003)
    “…The mode of cell death during galactosamine (Gal)-induced liver injury was originally thought to be oncotic necrosis but recently it was suggested to be…”
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  12. 12

    Chlorotyrosine protein adducts are reliable biomarkers of neutrophil-induced cytotoxicity in vivo by Gujral, Jaspreet S, Hinson, Jack A, Jaeschke, Hartmut

    Published in Comparative hepatology (14-01-2004)
    “…INTRODUCTION: A limitation for investigating the pathophysiological role of neutrophils in vivo is the lack of a reliable biomarker for neutrophil cytotoxicity…”
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    Protection against Fas receptor-mediated apoptosis in hepatocytes and nonparenchymal cells by a caspase-8 inhibitor in vivo : Evidence for a postmitochondrial processing of caspase-8 by BAJT, Mary Lynn, LAWSON, Judy A, VONDERFECHT, Steven L, GUJRAL, Jasprect S, JAESCHKE, Hartmut

    Published in Toxicological sciences (01-11-2000)
    “…Lymphocytes can kill target cells including hepatocytes during various inflammatory diseases by Fas receptor-mediated apoptosis. Caspase-8 is activated at the…”
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