Search Results - "Gonatas, J.O."

Disruption of the structure of the Golgi apparatus and the function of the secretory pathway by mutants G93A and G85R of Cu, Zn superoxide dismutase (SOD1) of familial amyotrophic lateral sclerosis by Stieber, Anna, Gonatas, J.O., Moore, J.S., Bantly, Andrew, Yim, H.-S., Yim, M.B., Gonatas, N.K.

“…The Golgi apparatus of motor neurons (GA) is fragmented in sporadic amyotrophic lateral sclerosis (ALS), in familial ALS with SOD1 mutations, and in mice that…”
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Stathmin, a microtubule depolymerizing protein, Hsp25, Hsp27, and the antioxidant peroxiredoxin 6 are differentially expressed in a mouse model of familial amyotrophic lateral sclerosis by Strey, C., Spellman, D., Stieber, A., Gonatas, J.O., Wang, X., Lambris, J.D., Gonatas, N.K.

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Assignment of the GLG1 Gene for MG-160, a Fibroblast Growth Factor and E-Selectin Binding Membrane Sialoglycoprotein of the Golgi Apparatus, to Chromosome 16q22-q23 by Fluorescence in Situ Hybridization by Mourelatos, Zissimos, Gonatas, Jacqueline O., Nycum, Lynn M., Gonatas, Nicholas K., Biegel, Jaclyn A.

“…MG-160, originally isolated from rat brain, is a conserved membrane sialoglycoprotein residing in the medial cisternae of the Golgi apparatus of virtually all…”
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