Search Results - "Glaubitz, Juliane"

Examination of duodenal and colonic microbiome changes in mouse models of acute and chronic pancreatitis by Lange, Rabea, Glaubitz, Juliane, Frost, Fabian, Geisz, Andreas, Aghdassi, Ali A., Weiss, F. Ulrich, Sendler, Matthias

“…The exocrine pancreas is the main source of digestive enzymes which are released from secretory vesicles of acinar cells into the small intestine. Enzymes,…”
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IRAK3-mediated suppression of pro-inflammatory MyD88/IRAK signaling affects disease severity in acute pancreatitis by Thiel, Franziska G., Asgarbeik, Saeedeh, Glaubitz, Juliane, Wilden, Anika, Lerch, Markus M., Weiss, Frank Ulrich, Sendler, Matthias

“…Acute pancreatitis (AP), which is characterized by self-digestion of the pancreas by its own prematurely activated digestive proteases, is a major reason for…”
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In mouse chronic pancreatitis CD25+FOXP3+ regulatory T cells control pancreatic fibrosis by suppression of the type 2 immune response by Glaubitz, Juliane, Wilden, Anika, Golchert, Janine, Homuth, Georg, Völker, Uwe, Bröker, Barbara M., Thiele, Thomas, Lerch, Markus M., Mayerle, Julia, Aghdassi, Ali A., Weiss, Frank U., Sendler, Matthias

“…Chronic pancreatitis (CP) is characterized by chronic inflammation and the progressive fibrotic replacement of exocrine and endocrine pancreatic tissue. We…”
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Immune response mechanisms in acute and chronic pancreatitis: strategies for therapeutic intervention by Glaubitz, Juliane, Asgarbeik, Saeedeh, Lange, Rabea, Mazloum, Hala, Elsheikh, Hager, Weiss, Frank Ulrich, Sendler, Matthias

“…Acute pancreatitis (AP) is one of the most common inflammatory diseases of the gastrointestinal tract and a steady rising diagnosis for inpatient…”
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Systemic Bile Acids Affect the Severity of Acute Pancreatitis in Mice Depending on Their Hydrophobicity and the Disease Pathogenesis by Tran, Quang Trung, Sendler, Matthias, Wiese, Mats L., Doller, Julia, Zierke, Lukas, Gischke, Marcel, Glaubitz, Juliane, Tran, Van Huy, Lalk, Michael, Bornscheuer, Uwe T., Weiss, Frank Ulrich, Lerch, Markus M., Aghdassi, Ali A.

“…Acute pancreatitis (AP) is a major, globally increasing gastrointestinal disease and a biliary origin is the most common cause. However, the effects of bile…”
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Mobilization of CD11b+/Ly6chi monocytes causes multi organ dysfunction syndrome in acute pancreatitis by Wilden, Anika, Glaubitz, Juliane, Otto, Oliver, Biedenweg, Doreen, Nauck, Matthias, Mack, Matthias, Ribback, Silvia, Bröker, Barbara M., Rheinbaben, Sabrina Freiin von, Lerch, Markus M., Aghdassi, Ali Alexander, Weiss, Frank Ulrich, Sendler, Matthias

“…Objective Acute pancreatitis (AP) is an inflammatory disorder, the severe form of which is burdened with multi-organ dysfunction and high mortality. The…”
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NLRP3 Inflammasome Regulates Development of Systemic Inflammatory Response and Compensatory Anti-Inflammatory Response Syndromes in Mice With Acute Pancreatitis by Sendler, Matthias, van den Brandt, Cindy, Glaubitz, Juliane, Wilden, Anika, Golchert, Janine, Weiss, Frank Ulrich, Homuth, Georg, De Freitas Chama, Laura L., Mishra, Neha, Mahajan, Ujjwal Mukund, Bossaller, Lukas, Völker, Uwe, Bröker, Barbara M., Mayerle, Julia, Lerch, Markus M.

“…Pancreatitis starts with primarily sterile local inflammation that induces systemic inflammatory response syndrome, followed by compensatory anti-inflammatory…”
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Pro-inflammatory cytokines of the IL-6 family induce regulatory T-cells and determine the severity of acute pancreatitis by Glaubitz, Juliane, Zimdahl, Anna, Weiss, Frank Ulrich, Sendler, Matthias

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The tissue remodeling of chronic pancreatitis is driven by a local type 2 immune response by Glaubitz, Juliane, Weiß, F., Sendler, M.

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Activated regulatory T-cells promote duodenal bacterial translocation into necrotic areas in severe acute pancreatitis by Glaubitz, Juliane, Wilden, Anika, Frost, Fabian, Ameling, Sabine, Homuth, Georg, Mazloum, Hala, Rühlemann, Malte Christoph, Bang, Corinna, Aghdassi, Ali A, Budde, Christoph, Pickartz, Tilmann, Franke, Andre, Bröker, Barbara M, Voelker, Uwe, Mayerle, Julia, Lerch, Markus M, Weiss, Frank-Ulrich, Sendler, Matthias

“…In acute pancreatitis (AP), bacterial translocation and subsequent infection of pancreatic necrosis are the main risk factors for severe disease and late…”
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Tu1203: BILE ACIDS MODULATE THE SEVERITY OF ACUTE PANCREATITIS DEPENDING ON THEIR HYDROPHOBICITY AND BY INTERACTING WITH CCK1 RECEPTOR by Tran, Quang Trung, Sendler, Matthias, Weiss, Frank Ulrich, Wiese, Mats L., Doller, Julia, Glaubitz, Juliane, Zierke, Lukas, Lalk, Michael, Bornscheuer, Uwe T., Lerch, Markus M., Aghdassi, Ali A.

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Experimental pancreatitis is characterized by rapid T cell activation, Th2 differentiation that parallels disease severity, and improvement after CD4+ T cell depletion by Glaubitz, Juliane, Wilden, Anika, van den Brandt, Cindy, Weiss, Frank U., Bröker, Barbara M., Mayerle, Julia, Lerch, Markus M., Sendler, Matthias

“…Acute pancreatitis is a gastrointestinal disorder of high incidence resulting in life threatening complications in up to 20% of patients. Its severe form is…”
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Role of Bile Acids and Bile Salts in Acute Pancreatitis: From the Experimental to Clinical Studies by Tran, Quang Trung, Tran, Van Huy, Sendler, Matthias, Doller, Julia, Wiese, Mats, Bolsmann, Robert, Wilden, Anika, Glaubitz, Juliane, Modenbach, Jana Marielle, Thiel, Franziska Gisela, de Freitas Chama, Laura L., Weiss, Frank Ulrich, Lerch, Markus M., Aghdassi, Ali A.

“…Acute pancreatitis (AP) is one of the most common gastroenterological disorders leading to hospitalization. It has long been debated whether biliary AP, about…”
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Pancreatitis severity in mice with impaired CFTR function but pancreatic sufficiency is mediated via ductal and inflammatory cells‐Not acinar cells by Trapp, Simon, Aghdassi, Ali A., Glaubitz, Juliane, Sendler, Matthias, Weiss, Frank Ulrich, Kühn, Jens Peter, Kromrey, Marie‐Luise, Mahajan, Ujjwal M., Pallagi, Petra, Rakonczay, Zoltán, Venglovecz, Viktória, Lerch, Markus M., Hegyi, Peter, Mayerle, Julia

“…Mutations in the cystic fibrosis transmembrane conductance regulator gene (CFTR) are an established risk factor for cystic fibrosis (CF) and chronic…”
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The depletion of Foxp3+/CD25+ regulatory T cells drives a Th2 immune response and reduces severity in a mouse model of acute pancreatitis by Glaubitz, Juliane, Wilden, Anika, van den Brandt, Cindy, Weiss, Frank Ulrich, Mayerle, Julia, Lerch, Markus M., Sendler, Matthias

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Interleukin 33 regulates Th2 T-cell differentiation and macrophage polarization during acute pancreatitis by Wilden, Anika, Glaubitz, Juliane, Golchert, Janine, Homuth, Georg, Weiss, Frank Ulrich, Lerch, Markus M., Sendler, Matthias

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Non classical macrophage polarization regulates the immune response during acute pancreatitis by Wilden, Anika, Glaubitz, Juliane, Golchert, Janine, Homuth, Georg, Weiss, Frank Ulrich, Mayerle, Julia, Lerch, Markus M., Sendler, Matthias

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Depletion of CD4+ T-helper cells reduces pancreatic damage in two models of acute pancreatitis by Glaubitz, Juliane, Wilden, Anika, van den Brandt, Cindy, Weiss, Frank Ulrich, Mayerle, Julia, Lerch, Markus M., Sendler, Matthias

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Macrophage-derived cytokines of the IL1 family simultaneously regulate pro- and anti-inflammation during severe acute pancreatitis by Sendler, Matthias, van den Brandt, Cindy, Wilden, Anika, Glaubitz, Juliane, Golchert, Janine, Homuth, Georg, Mahajan, Ujjwal Mukund, Weiss, Frank Ulrich, Mayerle, Julia, Lerch, Markus M.

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Adaptive immune response during acute pancreatitis is regulated by NLRP3 inflammasome activation via interleukin 18 by Sendler, Matthias, van den Brandt, Cindy, Glaubitz, Juliane, Wilden, Anika, Weiss, F. Ulrich, Golchert, Janine, Homuth, Georg, Bossaller, Lukas, Mayerle, Julia, Lerch, Markus M.

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