Search Results - "GRIEVE, D. J"

The influence of vestibular stimulation on metabolism and body composition by McKeown, J., McGeoch, P. D., Grieve, D. J.

“…Obesity, diabetes and metabolic disease represent an ongoing and rapidly worsening public health issue in both the developed, and much of the developing world…”
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Chronic treatment with a stable obestatin analog significantly alters plasma triglyceride levels but fails to influence food intake; fluid intake; body weight; or body composition in rats by Agnew, A., Calderwood, D., Chevallier, O.P., Greer, B., Grieve, D.J., Green, B.D.

“…▶ N-terminally PEGylated obestatin is approximately 3 times more stable than native obestatin. ▶ Chronic treatment with N-terminally PEGylated obestatin…”
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Oxidative stress and inflammation in COVID-19: potential application OF GLP-1 receptor agonists by Abudalo, R A, Alqudah, A M, Roarty, C, Athamneh, R Y, Grieve, D J

“…COVID-19 is a global pandemic with devastating economic and public health impacts, which is particularly associated with increased incidence of respiratory and…”
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A national survey of infection control practice by New Zealand anaesthetists by RYAN, A. J, WEBSTER, C. S, MERRY, A. F, GRIEVE, D. J

“…Anaesthetists have an important role in preventing nosocomial infection. Failures in this role have resulted in critical reports in the media. We ascertained…”
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Contrasting Roles of NADPH Oxidase Isoforms in Pressure-Overload Versus Angiotensin II–Induced Cardiac Hypertrophy by Byrne, Jonathan A, Grieve, David J, Bendall, Jennifer K, Li, Jian-Mei, Gove, Christopher, Lambeth, J David, Cave, Alison C, Shah, Ajay M

“…Increased production of reactive oxygen species (ROS) is implicated in the development of left ventricular hypertrophy (LVH). Phagocyte-type NADPH oxidases are…”
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The influence of chylomicron remnants on endothelial cell function in the isolated perfused rat aorta by Grieve, David J, Avella, Michael A, Elliott, Jonathan, Botham, Kathleen M

“…A system for the perfusion of the isolated rat aorta which allowed the study of both the uptake of chylomicron remnants by the artery wall and their effects on…”
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Effects of oral propylthiouracil treatment on nitric oxide production in rat aorta by Grieve, David J, Fletcher, Sara, Pitsillides, Andrew A, Botham, Kathleen M, Elliott, Jonathan

“…The effects of oral propylthiouracil (PTU) treatment on vascular nitric oxide (NO) production were studied in the rat aorta. Rats were fed a standard low fat…”
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Chylomicron remnants potentiate phenylephrine-induced contractions of rat aorta by an endothelium-dependent mechanism by Grieve, David J, Avella, Michael A, Botham, Kathleen M, Elliott, Jonathan

“…The effects of chylomicron remnants on endothelium-dependent contraction of rat aorta were studied in vitro. Chylomicron remnant particles were prepared in…”
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Effects of chylomicrons and chylomicron remnants on endothelium-dependent relaxation of rat aorta by Grieve, David J., Avella, Michael A., Botham, Kathleen M., Elliott, Jonathan

“…The effects of chylomicrons and chylomicron remnants on endothelium-dependent relaxation of rat aorta were studied in vitro. Chylomicrons and chylomicron…”
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Impaired endothelial regulation of ventricular relaxation in cardiac hypertrophy: Role of reactive oxygen species and NADPH oxidase by MACCARTHY, Philip A, GRIEVE, David J, LI, Jian-Mei, DUNSTER, Christina, KELLY, Frank J, SHAH, Ajay M

“…Endothelium-derived nitric oxide (NO) selectively enhances myocardial relaxation. In experimental left ventricular hypertrophy (LVH), this…”
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Oxidative stress and heart failure by Byrne, J A, Grieve, D J, Cave, A C, Shah, A M

“…Despite advances in treatment, chronic congestive heart failure carries a poor prognosis and remains a leading cause of cardiovascular death. Accumulating…”
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Results of posterior cervical foraminotomy for treatment of cervical spondylitic radiculopathy by GRIEVE, J. P, KITCHEN, N. D, MOORE, A. J, MARSH, H. T

“…We evaluated the results of posterior cervical foraminotomy for spondylitic radiculopathy using a questionnaire sent to all 77 patients who had undergone…”
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Divergent Biological Actions of Coronary Endothelial Nitric Oxide During Progression of Cardiac Hypertrophy by Grieve, David J, MacCarthy, Philip A, Gall, Nicholas P, Cave, Alison C, Shah, Ajay M

“…ABSTRACT—Coronary endothelial NO synthase expression and NO bioactivity were investigated at sequential stages during the progression of left ventricular…”
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Investigation of endothelial nitric oxide (NO) signalling in response to the incretin hormone glucose-dependent insulinotropic polypeptide (GIP) by Savage, PJ, Harvey, AP, Robinson, E, Grieve, DJ

“…Doc number: O38…”
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Activation of NADPH Oxidase During Progression of Cardiac Hypertrophy to Failure by Li, Jian-Mei, Gall, Nick P, Grieve, David J, Chen, Mingyou, Shah, Ajay M

“…ABSTRACT—Increased reactive oxygen species (ROS) production is implicated in the pathophysiology of left ventricular (LV) hypertrophy and heart failure…”
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Involvement of the Nicotinamide Adenosine Dinucleotide Phosphate Oxidase Isoform Nox2 in Cardiac Contractile Dysfunction Occurring in Response to Pressure Overload by Grieve, David J., Byrne, Jonathan A., Siva, Anjana, Layland, Joanne, Johar, Sofian, Cave, Alison C., Shah, Ajay M.

“…Involvement of the Nicotinamide Adenosine Dinucleotide Phosphate Oxidase Isoform Nox2 in Cardiac Contractile Dysfunction Occurring in Response to Pressure…”
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Oxidative stress in heart failure. More than just damage by Grieve, David J, Shah, Ajay M

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NADPH oxidase and heart failure by Murdoch, Colin E, Grieve, David J, Cave, Alison C, Looi, Yee Hoo, Shah, Ajay M

“…Reactive oxygen species play important roles in the pathophysiology of chronic heart failure secondary to chronic left ventricular hypertrophy or myocardial…”
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INCREASED EXPRESSION AND ACTIVITY OF AN NADPH OXIDASE DURING PROGRESSIVE CARDIAC HYPERTROPHY by Li, J-M, Grieve, D J, Gall, N P, Shah, A M

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Essential role of troponin I in the positive inotropic response to isoprenaline in mouse hearts contracting auxotonically by Layland, Joanne, Grieve, David J., Cave, Alison C., Sparks, Emma, Solaro, R John, Shah, Ajay M.

“…PKA-dependent phosphorylation of cardiac troponin I (cTnI) contributes significantly to β-adrenergic agonist-induced acceleration of myocardial relaxation…”
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