Search Results - "Feyder, M."
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Mice lacking the AMPA GluR1 receptor exhibit striatal hyperdopaminergia and 'schizophrenia-related' behaviors
Published in Molecular psychiatry (01-06-2008)“…There is growing evidence implicating dysfunctional glutamatergic neurotransmission and abnormal interactions between the glutamate and dopamine (DA) systems…”
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Durable fear memories require PSD-95
Published in Molecular psychiatry (01-07-2015)“…Traumatic fear memories are highly durable but also dynamic, undergoing repeated reactivation and rehearsal over time. Although overly persistent fear memories…”
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3
Impaired Pavlovian fear extinction is a common phenotype across genetic lineages of the 129 inbred mouse strain
Published in Genes, brain and behavior (01-11-2009)“…Fear extinction is impaired in psychiatric disorders such as post‐traumatic stress disorder and schizophrenia, which have a major genetic component. However,…”
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4
Durable fear memories require PSD-95
Published in Molecular psychiatry (01-07-2015)Get full text
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Reversal of neurobehavioral social deficits in dystrophic mice using inhibitors of phosphodiesterases PDE5A and PDE9A
Published in Translational psychiatry (27-09-2016)“…Duchenne muscular dystrophy is caused by mutations in the DYSTROPHIN gene. Although primarily associated with muscle wasting, a significant portion of patients…”
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Induction of functional dopamine neurons from human astrocytes in vitro and mouse astrocytes in a Parkinson's disease model
Published in Nature biotechnology (01-05-2017)“…In vivo reprogramming of astrocytes to dopamine neurons improves motor behavior in a mouse model of Parkinson's disease. Cell replacement therapies for…”
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Pathophysiology of L-dopa-induced motor and non-motor complications in Parkinson's disease
Published in Progress in neurobiology (01-09-2015)“…Involuntary movements, or dyskinesia, represent a debilitating complication of levodopa (L-dopa) therapy for Parkinson's disease (PD). L-dopa-induced…”
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A Role for Mitogen- and Stress-Activated Kinase 1 in L-DOPA-Induced Dyskinesia and Delta FosB Expression
Published in Biological psychiatry (1969) (01-03-2016)“…BACKGROUND: Abnormal regulation of extracellular signal-regulated kinases 1 and 2 has been implicated in 3,4-dihydroxy-L-phenylalanine (L-DOPA)-induced…”
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A Role for Mitogen- and Stress-Activated Kinase 1 in L-DOPA –Induced Dyskinesia and ∆FosB Expression
Published in Biological psychiatry (1969) (01-03-2016)“…Abstract Background Abnormal regulation of extracellular signal - regulated kinases 1 and 2 has been implicated in 3,4-dihydroxy- l -phenylalanine ( L-DOPA…”
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Involvement of Autophagy in Levodopa‐Induced Dyskinesia
Published in Movement disorders (01-05-2021)“…Background Autophagy is intensively studied in cancer, metabolic and neurodegenerative diseases, but little is known about its role in pathological conditions…”
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Plasma reciprocal pool specific activity predicts that of intracellular free leucine for protein synthesis
Published in The American journal of physiology (01-09-1989)“…We previously proposed that, during the infusion of either labeled leucine or its alpha-ketoacid, alpha-ketoisocaproate (KIC), the plasma specific activity…”
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L-DOPA-Induced Dyskinesia and Abnormal Signaling in Striatal Medium Spiny Neurons: Focus on Dopamine D1 Receptor-Mediated Transmission
Published in Frontiers in behavioral neuroscience (2011)“…Dyskinesia is a serious motor complication caused by prolonged administration of l-DOPA to patients affected by Parkinson's disease. Accumulating evidence…”
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Dopamine- and cAMP-regulated Phosphoprotein of 32-kDa (DARPP-32)-dependent Activation of Extracellular Signal-regulated Kinase (ERK) and Mammalian Target of Rapamycin Complex 1 (mTORC1) Signaling in Experimental Parkinsonism
Published in The Journal of biological chemistry (10-08-2012)“…Dyskinesia, a motor complication caused by prolonged administration of the antiparkinsonian drug l-3,4-dihydroxyphenylalanine (l-DOPA), is accompanied by…”
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Dopamine signaling leads to loss of Polycomb repression and aberrant gene activation in experimental parkinsonism
Published in PLoS genetics (01-09-2014)“…Polycomb group (PcG) proteins bind to and repress genes in embryonic stem cells through lineage commitment to the terminal differentiated state. PcG repressed…”
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Dopamine-dependent long-term depression at subthalamo-nigral synapses is lost in experimental parkinsonism
Published in The Journal of neuroscience (04-09-2013)“…Impairments of synaptic plasticity are a hallmark of several neurological disorders, including Parkinson's disease (PD) which results from the progressive loss…”
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Erratum: Durable fear memories require PSD-95
Published in Molecular psychiatry (01-07-2015)“…Correction to: Molecular Psychiatry advance online publication, 16 December 2014; doi:10.1038/mp.2014.161 Following publication of this paper, the authors…”
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Reward-related behavioral paradigms for addiction research in the mouse: performance of common inbred strains
Published in PloS one (10-01-2011)“…The mouse has emerged as a uniquely valuable species for studying the molecular and genetic basis of complex behaviors and modeling neuropsychiatric disease…”
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Fear memory impairing effects of systemic treatment with the NMDA NR2B subunit antagonist, Ro 25-6981, in mice: Attenuation with ageing
Published in Pharmacology, biochemistry and behavior (01-01-2009)“…N-methyl- d-aspartate receptors (NMDARs) are mediators of synaptic plasticity and learning and are implicated in the pathophysiology of neuropsychiatric…”
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The Prevalence and Inaccessibility of Internet References in the Biomedical Literature at the Time of Publication
Published in Journal of the American Medical Informatics Association : JAMIA (01-03-2007)“…To determine the prevalence and inaccessibility of Internet references in the bibliography of biomedical publications when first released in PubMed®. During a…”
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Deciphering the Actions of Antiparkinsonian and Antipsychotic Drugs on cAMP/DARPP-32 Signaling
Published in Frontiers in neuroanatomy (2011)“…The basal ganglia are affected by several neuropsychiatric and neurodegenerative diseases, many of which are treated with drugs acting on the dopamine system…”
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