Search Results - "Ferrari, Merari de Fátima Ramires"

Induced degeneration and regeneration in aged muscle reduce tubular aggregates but not muscle function by de Vasconcelos, Felipe Tadeu Galante Rocha, Ribeiro Júnior, Antonio Fernando, Souza, Brandow Willy, Zogbi, Isabela de Aquino, Carvalho, Laura Machado Lara, Feitosa, Letícia Nogueira, Souza, Lucas Santos, Saldys, Nathália Gagliardi, Ferrari, Merari de Fátima Ramires, Vainzof, Mariz

“…Tubular aggregates (TA) are skeletal muscle structures that arise from the progressive accumulation of sarcoplasmic reticulum proteins. Cytoplasmic aggregates…”
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Neuroprogenitor Cells From Patients With TBCK Encephalopathy Suggest Deregulation of Early Secretory Vesicle Transport by Moreira, Danielle de Paula, Suzuki, Angela May, Silva, André Luiz Teles E, Varella-Branco, Elisa, Meneghetti, Maria Cecília Zorél, Kobayashi, Gerson Shigeru, Fogo, Mariana, Ferrari, Merari de Fátima Ramires, Cardoso, Rafaela Regina, Lourenço, Naila Cristina Vilaça, Griesi-Oliveira, Karina, Zachi, Elaine Cristina, Bertola, Débora Romeo, Weinmann, Karina de Souza, de Lima, Marcelo Andrade, Nader, Helena Bonciani, Sertié, Andrea Laurato, Passos-Bueno, Maria Rita

“…Biallelic pathogenic variants in TBCK cause encephaloneuropathy, infantile hypotonia with psychomotor retardation, and characteristic facies 3 (IHPRF3). The…”
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Simvastatin ameliorates experimental autoimmune encephalomyelitis by inhibiting Th1/Th17 response and cellular infiltration by de Oliveira, Daniel May, de Oliveira, Enedina Maria Lobato, Ferrari, Merari de Fátima Ramires, Semedo, Patrícia, Hiyane, Meire Ioshie, Cenedeze, Marcos Antônio, Pacheco-Silva, Alvaro, Câmara, Niels Olsen Saraiva, Peron, Jean Pierre Schatzmann

“…Aim Experimental autoimmune encephalomyelitis (EAE) is a CD4 + -mediated autoimmune pathology of the central nervous system (CNS) that is used as a model for…”
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Adenosine receptor type 2a is differently modulated by nicotine in dorsal brainstem cells of Wistar Kyoto and spontaneously hypertensive rats by de Matsumoto, Joao Paulo Pontes, de Ferrari, Merari Fatima Ramires, Fior-Chadi, Debora Rejane

“…Hypertension can result from neuronal network imbalance in areas of central nervous system that control blood pressure, such as the nucleus tractus solitarius…”
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Vitamin D3 supplementation may attenuate morphological and molecular abnormalities of the olfactory bulb in a mouse model of Down syndrome by Gomes, Fabiana de Campos, Santos, Isabella Boechat Faria, Stephani, Carolinne Makino, Ferrari, Merari de Fátima Ramires, Galvis-Alonso, Orfa Yineth, Goloni-Bertollo, Eny Maria, Melo-Neto, João Simão de, Pavarino, Érika Cristina

“…Individuals with Down syndrome (DS) exhibit impaired olfactory function and are at a higher risk of developing Alzheimer's disease (AD). Olfactory dysfunction…”
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BAG2 expression dictates a functional intracellular switch between the p38-dependent effects of nicotine on tau phosphorylation levels via the α7 nicotinic receptor by de Oliveira, Adriele Silva Alves, Santiago, Fernando Enrique, Balioni, Laiz Furlan, Ferrari, Merari de Fatima Ramires, Almeida, Maria Camila, Carrettiero, Daniel Carneiro

“…The histopathological hallmarks present in Alzheimer's disease (AD) brain are plaques of Aβ peptide, neurofibrillary tangles of hyperphosphorylated tau…”
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Vitamin D3 increases the Caspase-3 p12, MTHFR, and P-glycoprotein reducing amyloid-β42 in the kidney of a mouse model for Down syndrome by Gomes, Fabiana de Campos, de Melo-Neto, João Simão, Ferrari, Merari de Fátima Ramires, Carlos, Carla Patrícia, Goloni-Bertollo, Eny Maria, Pavarino, Érika Cristina

“…Renal dysfunction has been reported in individuals with Down syndrome (DS); however, the causes and mechanisms involved remain unknown. Here, we present a…”
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Vitamin D 3 increases the Caspase-3 p12, MTHFR, and P-glycoprotein reducing amyloid-β 42 in the kidney of a mouse model for Down syndrome by Gomes, Fabiana de Campos, de Melo-Neto, João Simão, Ferrari, Merari de Fátima Ramires, Carlos, Carla Patrícia, Goloni-Bertollo, Eny Maria, Pavarino, Érika Cristina

“…Renal dysfunction has been reported in individuals with Down syndrome (DS); however, the causes and mechanisms involved remain unknown. Here, we present a…”
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