Search Results - "Ferlinz, R"

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    Oncogene overexpression in non-small-cell lung cancer tissue: prevalence and clinicopathological significance by Lorenz, J, Friedberg, T, Paulus, R, Oesch, F, Ferlinz, R

    “…In contrast to small-cell lung cancer, few data are available on the role of oncogene overexpression in non-small-cell lung cancers (NSCLC). To determine the…”
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    Modulation of TNF-alpha secretions by alveolar macrophages and blood monocytes after soot-particle or asbestos fibre exposure by Kienast, K, Bay, M, Drumm, K, Smolarski, R, Ferlinz, R

    Published in European journal of medical research (25-06-1996)
    “…Activated monocytes secrete tumor necrosis factor-alpha (TNF-alpha), whose inflammatory and fibroblast activating characteristics may play a role in the…”
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    In vitro study of human alveolar macrophage and peripheral blood mononuclear cell reactive oxygen-intermediates release induced by sulfur dioxide at different concentrations by Kienast, K, Müller-Quernheim, J, Knorst, M, Lubjuhn, S, Ferlinz, R

    Published in Lung (01-11-1994)
    “…Sulfur dioxide (SO2) is a major air pollutant in urban areas. Alveolar macrophages (AM) located on the alveolar surface are in direct contact with this inhaled…”
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    Fibrosing alveolitis responsive to corticosteroids following Legionnaires' disease pneumonia by Hürter, T, Rumpelt, H J, Ferlinz, R

    Published in Chest (01-01-1992)
    “…Two male patients ages 54 and 58 years had persisting pneumonia with dry cough, dyspnea, weight loss, and fever up to 39 degrees C that did not respond to…”
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    Combined exposures of human ciliated cells to different concentrations of sulfur dioxide and nitrogen dioxide by Kienast, K, Riechelmann, H, Knorst, M, Haffner, B, Müller-Quernheim, J, Schellenberg, J, Ferlinz, R

    Published in European journal of medical research (20-09-1996)
    “…In the present study we investigated the influence of two common air pollutants, sulfur dioxide (SO2) and nitrogen dioxide (NO2) on ciliary beat frequency…”
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    Replacement therapy for alpha-1-protease inhibitor deficiency in PiZ subjects with chronic obstructive lung disease by Schmidt, E W, Rasche, B, Ulmer, W T, Konietzko, N, Becker, M, Fallise, J P, Lorenz, J, Ferlinz, R

    Published in The American journal of medicine (24-06-1988)
    “…In a six-month multicenter feasibility and safety study, 20 patients, who all had a congenital deficiency of alpha-1-protease inhibitor (A1PI) of the PiZ…”
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    An experimental model for the exposure of human ciliated cells to sulfur dioxide at different concentrations by Kienast, K, Riechelmann, H, Knorst, M, Schlegel, J, Müller-Quernheim, J, Schellenberg, J, Ferlinz, R

    Published in The Clinical Investigator (01-02-1994)
    “…Mucociliary transport is an important nonimmunological defense mechanism of the respiratory tract. The aim of this study was to investigate the effect of…”
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    Oxygen radical production by alveolar inflammatory cells in idiopathic pulmonary fibrosis by Strausz, J, Müller-Quernheim, J, Steppling, H, Ferlinz, R

    Published in The American review of respiratory disease (01-01-1990)
    “…Idiopathic pulmonary fibrosis (IPF) is a chronic inflammatory interstitial lung disease characterized by the accumulation of alveolar macrophages (AMs) and…”
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    Lung-restricted activation of the alveolar macrophage/monocyte system in pulmonary sarcoidosis by Müller-Quernheim, J, Pfeifer, S, Männel, D, Strausz, J, Ferlinz, R

    Published in The American review of respiratory disease (01-01-1992)
    “…An activation of T-cells that is restricted to the lung has been demonstrated in pulmonary sarcoidosis. The role of blood monocytes (MO) and alveolar…”
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    Assessment of local cellular immunity in lung cancer by bronchoalveolar lavage by Lorenz, J, Müller-Quernheim, J, Castillo-Höfer, C, Doboszỳnska, A, Ferlinz, R

    Published in Klinische Wochenschrift (17-07-1990)
    “…Small cell lung cancer (SCLC) is the most malignant of the pulmonary neoplasms and is associated with a poor local cellular immune response. 16 patients with…”
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    Guidelines for environmental contact tracing in tuberculosis. German Central Committee for control of tuberculosis by Ferlinz, R

    “…These guidelines were initiated by and worked out in cooperation with physicians of the public health service. Numerous publications and relevant legal…”
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    A macrophage-suppressing 40-kD protein in a case of pulmonary alveolar proteinosis by Müller-Quernheim, J, Schopf, R E, Benes, P, Schulz, V, Ferlinz, R

    Published in Klinische Wochenschrift (01-10-1987)
    “…Pulmonary alveolar proteinosis (PAP) is a rare disease of unknown etiology. Macrophage dysfunctions are claimed to be involved in the pathogenesis. We…”
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    Diabetes insipidus in sarcoidosis by Fischer, B, Müller-Quernheim, J, Ferlinz, R

    Published in Pneumologie (Stuttgart, Germany) (01-03-1991)
    “…Sarcoidosis is a generalized disorder which involves the central nervous system in 5 per cent of patients presenting with the disease. We describe the case of…”
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    Correlation of clinical and immunologic parameters of the inflammatory activity of pulmonary sarcoidosis by Müller-Quernheim, J, Pfeifer, S, Strausz, J, Ferlinz, R

    Published in The American review of respiratory disease (01-12-1991)
    “…The evaluation of activation markers such as T4/T8 ratio and HLA-DR expression of lymphocytes of bronchoalveolar lavage (L-BAL) is an important clinical…”
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    Effect of sulfur dioxide on cytokine production of human alveolar macrophages in vitro by Knorst, M M, Kienast, K, Müller-Quernheim, J, Ferlinz, R

    Published in Archives of environmental health (01-03-1996)
    “…Tumor necrosis factor-alpha, interleukin-1beta, interleukin-6, and transforming growth factor-beta are cytokines synthesized by alveolar macrophages. We…”
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    Interleukin-2 receptor gene expression by bronchoalveolar lavage lymphocytes in pulmonary sarcoidosis by Müller-Quernheim, J, Krönke, M, Strausz, J, Schykowski, M, Ferlinz, R

    Published in The American review of respiratory disease (01-07-1989)
    “…Current concepts of the immunopathogenesis of sarcoidosis favor a central role of activated, interleukin-2 (IL-2) producing helper T-cells at sites of…”
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