Search Results - "Faé, K C"

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  1. 1

    Rheumatic Fever: How S. pyogenes-Primed Peripheral T Cells Trigger Heart Valve Lesions by GUILHERME, L., FAÉ, K. C., OSHIRO, S. E., TANAKA, A. C., POMERANTZEFF, P M A, KALIL, J.

    Published in Annals of the New York Academy of Sciences (01-06-2005)
    “…: The pathogenesis of rheumatic fever (RF) is related to autoimmune humoral and cellular responses against human tissues triggered by Streptococcus pyogenes…”
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  2. 2

    Towards a vaccine against rheumatic fever by Guilherme, L, Faé, K C, Higa, F, Chaves, L, Oshiro, S E, Freschi de Barros, S, Puschel, C, Juliano, M A, Tanaka, A C, Spina, G, Kalil, J

    Published in Clinical & developmental immunology (01-06-2006)
    “…Rheumatic fever (RF) is an autoimmune disease which affects more than 20 million children in developing countries. It is triggered by Streptococcus pyogenes…”
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  3. 3

    How an autoimmune reaction triggered by molecular mimicry between streptococcal M protein and cardiac tissue proteins leads to heart lesions in rheumatic heart disease by Faé, K.C., Oshiro, S.E., Toubert, A., Charron, D., Kalil, J., Guilherme, L.

    Published in Journal of autoimmunity (01-03-2005)
    “…Molecular mimicry between microbial antigens and host tissue is suggested as a mechanism for post-infectious autoimmune disease. In the present work we…”
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  4. 4

    CD4⁺CD25⁺Foxp3⁺ Indirect Alloreactive T cells from Renal Transplant Patients Suppress Both the Direct and Indirect Pathways of Allorecognition by Spadafora-Ferreira, M, Caldas, C, Faé, K.C, Marrero, I, Monteiro, S.M, Lin-Wang, H.T, Socorro-Silva, A, Fonseca, S.G, Fonseca, J.A, Kalil, J, Coelho, V

    Published in Scandinavian journal of immunology (01-08-2007)
    “…Alloreactive T cells recognize donor antigens by two routes: direct and indirect pathways of allorecognition. Although the direct pathway is reported to be…”
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  5. 5

    HLA and β-myosin heavy chain do not influence susceptibility to Chagas' disease cardiomyopathy by Faé, Kellen C., Drigo, Sandra A., Cunha-Neto, Edécio, Ianni, Bárbara, Mady, Charles, Kalil, Jorge, Goldberg, Anna C.

    Published in Microbes and infection (01-06-2000)
    “…An inflammatory dilated cardiomyopathy occurs in 30% of Chagas' disease patients, chronically infected by Trypanosoma cruzi, while the remaining infected…”
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  6. 6

    BAT1, a Putative Anti-Inflammatory Gene, Is Associated with Chronic Chagas Cardiomyopathy by Ramasawmy, Rajendranath, Cunha-Neto, Edecio, Faé, Kellen C., Müller, Natalie G., Cavalcanti, Vanessa L., Drigo, Sandra A., Ianni, Barbara, Mady, Charles, Kalil, Jorge, Goldberg, Anna Carla

    Published in The Journal of infectious diseases (15-05-2006)
    “…BackgroundIt is not understood why only a subset of individuals infected with Trypanosoma cruzi develop chronic Chagas cardiomyopathy (CCC). Patients with CCC…”
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    Identification of group A beta hemolytic Streptococcus strains in Brazil by Freschi de Barros, S., Alencar, R., Higa, F., Alcântara, F., Rossi, F., Muller, R., Faé, K.C., Kalil, J., Guilherme, L.

    Published in International Congress series (2006)
    “…Despite the high incidence of RF and the prevalence of RHD in Brazil, the Streptococcus strains more prevalent in our country were not identified. Aiming to…”
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  9. 9

    The Monocyte Chemoattractant Protein–1 Gene Polymorphism Is Associated with Cardiomyopathy in Human Chagas Disease by Ramasawmy, Rajendranath, Cunha-Neto, Edecio, Faé, Kellen C., Martello, Fernanda G., Müller, Natalie G., Cavalcanti, Vanessa L., Ianni, Barbara, Mady, Charles, Kalil, Jorge, Goldberg, Anna C.

    Published in Clinical infectious diseases (01-08-2006)
    “…Background. Only a subset of individuals infected with Trypanosoma cruzi develop chronic Chagas cardiomyopathy (CCC). Familial aggregation of CCC in areas of…”
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  10. 10
  11. 11

    Polymorphisms in the Gene for Lymphotoxin-αPredispose to Chronic Chagas Cardiomyopathy by Ramasawmy, Rajendranath, Fae, Kellen C., Neto, Edecio Cunha, Müller, Natalie G., Cavalcanti, Vanessa L., Ferreira, Renata C., Drigo, Sandra A., Ianni, Barbara, Mady, Charles, Goldberg, Anna C., Kalil, Jorge

    Published in The Journal of infectious diseases (15-12-2007)
    “…Background. Chagas disease, caused by Trypanosoma cruzi infection, displays clinical heterogeneity and may be attributable to differential genetic…”
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  12. 12
  13. 13

    Lack of association of tumor necrosis factor-α polymorphisms with Chagas disease in Brazilian patients by Drigo, Sandra A, Cunha-Neto, Edecio, Ianni, Bárbara, Mady, Charles, Faé, Kellen C, Buck, Paula, Kalil, Jorge, Goldberg, Anna Carla

    Published in Immunology Letters (15-01-2007)
    “…Abstract One third of Trypanosoma cruzi -infected individuals develop chronic Chagas disease cardiomyopathy (CCC) while the majority remains asymptomatic…”
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  14. 14

    The role of allergic rhinitis in nasal responses to sudden temperature changes by Graudenz, Gustavo Silveira, Landgraf, Richardt G., Jancar, Sonia, Tribess, Arlindo, Fonseca, Simone G., Faé, Kellen Cristhina, Kalil, Jorge

    Published in Journal of allergy and clinical immunology (01-11-2006)
    “…Air conditioning–induced rhinitis in allergic individuals is a common epidemiologic finding, but its physiopathology is still controversial. The aim of this…”
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  15. 15

    CD4 super(+)CD25 super(+)Foxp3 super(+) Indirect Alloreactive T cells from Renal Transplant Patients Suppress Both the Direct and Indirect Pathways of Allorecognition by Spadafora-Ferreira, M, Caldas, C, Fae, K C, Marrero, I, Monteiro, S M, Lin-Wang, H T, Socorro-Silva, A, Fonseca, S G, Fonseca, JA, Kalil, J, Coelho, V

    Published in Scandinavian journal of immunology (01-08-2007)
    “…Alloreactive T cells recognize donor antigens by two routes: direct and indirect pathways of allorecognition. Although the direct pathway is reported to be…”
    Get full text
    Journal Article
  16. 16