Search Results - "FIDANZA, V"

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    Overexpression of the HMGA2 gene in transgenic mice leads to the onset of pituitary adenomas by Fedele, M, Battista, S, Kenyon, L, Baldassarre, G, Fidanza, V, Klein-Szanto, AJP, Parlow, A F, Visone, R, Pierantoni, G M, Outwater, E, Santoro, M, Croce, C M, Fusco, A

    Published in Oncogene (09-05-2002)
    “…Overexpression of the HMGA2 gene is a common feature of neoplastic cells both in experimental and human models. Intragenic and extragenic HMGA2 rearrangements…”
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    Activation of clones producing self-reactive antibodies by foreign antigen and antiidiotype antibody carrying the internal image of the antigen by BAILEY, N. C, FIDANZA, V, MAYER, R, MAZZA, G, FOUGEREAU, M, BONA, C

    Published in The Journal of clinical investigation (01-09-1989)
    “…Because we found in previous work that a high fraction of antibodies exhibiting various specificities bound to glutamic acid 50-tyrosine50 homopolymer (GT) and…”
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    The expression of a truncated HMGI-C gene induces gigantism associated with lipomatosis by Battista, S, Fidanza, V, Fedele, M, Klein-Szanto, A J, Outwater, E, Brunner, H, Santoro, M, Croce, C M, Fusco, A

    Published in Cancer research (Chicago, Ill.) (01-10-1999)
    “…Rearrangements of the HMGI-C gene have frequently been detected in human benign tumors of mesenchymal origin, including lipomas. The HMGI-C protein has three…”
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    The Tumor Spectrum in FHIT-Deficient Mice by Zanesi, Nicola, Fidanza, Vincenzo, Fong, Louise Y., Mancini, Rita, Druck, Teresa, Valtieri, Mauro, Rüdiger, Thomas, McCue, Peter A., Croce, Carlo M., Huebner, Kay

    “…Mice carrying one inactivated Fhit allele (Fhit +/- mice) are highly susceptible to tumor induction by N-nitrosomethylbenzylamine, with 100% of Fhit +/- mice…”
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    FHIT Gene Therapy Prevents Tumor Development in Fhit-Deficient Mice by Dumon, Kristoffel R., Ishii, Hideshi, Louise Y. Y. Fong, Zanesi, Nicola, Fidanza, Vincenzo, Mancini, Rita, Vecchione, Andrea, Baffa, Raffaele, Trapasso, Francesco, During, Matthew J., Huebner, Kay, Croce, Carlo M.

    “…The tumor suppressor gene FHIT spans a common fragile site and is highly susceptible to environmental carcinogens. FHIT inactivation and loss of expression is…”
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    High level expression of the HMGI (Y) gene during embryonic development by Chiappetta, G, Avantaggiato, V, Visconti, R, Fedele, M, Battista, S, Trapasso, F, Merciai, B M, Fidanza, V, Giancotti, V, Santoro, M, Simeone, A, Fusco, A

    Published in Oncogene (05-12-1996)
    “…The HMGI protein family includes three proteins, named HMG-I, HMG-Y and HMGI-C. The first two proteins are coded for by the same gene, HMGI (Y), through an…”
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    Muir-Torre-Like Syndrome in Fhit-Deficient Mice by Louise Y. Y. Fong, Fidanza, Vincenzo, Zanesi, Nicola, Lock, Leslie F., Siracusa, Linda D., Mancini, Rita, Siprashvili, Zurab, Ottey, Michelle, Martin, S. Eric, Druck, Teresa, McCue, Peter A., Croce, Carlo M., Huebner, Kay

    “…To investigate the role of the Fhit gene in carcinogen induction of neoplasia, we have inactivated one Fhit allele in mouse embryonic stem cells and produced…”
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    The significance of idiotype-anti-idiotype interactions in the activation of self-reactive clones by Zaghouani, H, Fidanza, V, Bona, C A

    Published in Clinical and experimental rheumatology (01-09-1989)
    “…Autoantibodies against a wide range of self antigens have been found in the serum of normal mice and healthy humans. It is now accepted that the production of…”
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    The FEZ1 Gene at Chromosome 8p22 Encodes a Leucine-Zipper Protein, and Its Expression Is Altered in Multiple Human Tumors by Ishii, Hideshi, Baffa, Raffaele, Numata, Shin-Ichiro, Murakumo, Yoshiki, Rattan, Shashi, Inoue, Hiroshi, Mori, Masaki, Fidanza, Vincenzo, Alder, Hansjuerg, Croce, Carlo M.

    “…Alterations of human chromosome 8p occur frequently in many tumors. We identified a 1.5-Mb common region of allelic loss on 8p22 by allelotype analysis. cDNA…”
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    An agarose gel resolving a wide range of DNA fragment lengths by Boncinelli, E, Simeone, A, de Falco, A, Fidanza, V, La Volpe, A

    Published in Analytical biochemistry (01-10-1983)
    “…To resolve DNA fragments ranging from several kilobases to some tens of base pairs in length, an agarose slab gel of steadily increasing thickness has been…”
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    Identification and Characterization of the ARP1 Gene, a Target for the Human Acute Leukemia ALL1 Gene by Arakawa, Hirofumi, Nakamura, Tatsuya, Zhadanov, Alexander B., Fidanza, Vincenzo, Yano, Takahiro, Bullrich, Florencia, Shimizu, Masayoshi, Blechman, Janna, Mazo, Alexander, Canaani, Eli, Croce, Carlo M.

    “…ALL1, the human homologue of Drosophila trithorax, is directly involved in human acute leukemias associated with abnormalities at 11q23. Using the differential…”
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    Altered gene expression in immunogenic poorly differentiated thyroid carcinomas from RET/PTC3p53-/- mice by POWELL, Daniel J, RUSSELL, John P, GUOQI LI, KUO, Bruce A, FIDANZA, Vincenzo, HUEBNER, Kay, ROTHSTEIN, Jay L

    Published in Oncogene (31-05-2001)
    “…Cancers develop and progress via activation of oncogenes and loss of tumor suppressor genes, a progression that can be recapitulated through cross breeding…”
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    DLX genes as targets of ALL-1: DLX 2,3,4 down-regulation in t(4;11) acute lymphoblastic leukemias by Ferrari, Nicoletta, Palmisano, Giulio L., Paleari, Laura, Basso, Giuseppe, Mangioni, Manuela, Fidanza, Vincenzo, Albini, Adriana, Croce, Carlo M., Levi, Giovanni, Brigati, Claudio

    Published in Journal of leukocyte biology (01-08-2003)
    “…Dlx genes constitute a gene family thought to be essential in morphogenesis and development. We show here that in vertebrate cells, Dlx genes appear to be part…”
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    Double knockout of the ALL-1 gene blocks hematopoietic differentiation in vitro by FIDANZA, V, MELOTTI, P, YANO, T, NAKAMURA, T, BRADLEY, A, CANAANI, E, CALABRETTA, B, CROCE, C. M

    Published in Cancer research (Chicago, Ill.) (15-03-1996)
    “…The ALL-1 gene is involved in translocations with many partner genes in different types of the acute leukemias, but it is not clear whether it acts as an…”
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    Altered gene expression in immunogenic poorly differentiated thyroid carcinomas from RET/PTC3 super(p53-/-) mice by Powell, DJ Jr, Russell, J P, Li, Guoqi, Kuo, BA, Fidanza, V, Huebner, K, Rothstein, J L

    Published in Oncogene (31-05-2001)
    “…Cancers develop and progress via activation of oncogenes and loss of tumor suppressor genes, a progression that can be recapitulated through cross breeding…”
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