Search Results - "Evrard, Céline"
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Epidermal Hyaluronan in Barrier Alteration-Related Disease
Published in Cells (Basel, Switzerland) (09-11-2021)“…In skin, although the extracellular matrix (ECM) is highly developed in dermis and hypodermis, discrete intercellular spaces between cells of the living…”
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Responses of Reconstructed Human Epidermis to Trichophyton rubrum Infection and Impairment of Infection by the Inhibitor PD169316
Published in Journal of investigative dermatology (01-10-2019)“…Despite the threatening incidence of dermatophytosis, information is still lacking about the consequences of infection on epidermal barrier functions and about…”
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Investigations into the FLG Null Phenotype: Showcasing the Methodology for CRISPR/Cas9 Editing of Human Keratinocytes
Published in Journal of investigative dermatology (01-08-2023)“…Ever since the association between FLG loss-of-function variants and ichthyosis vulgaris and atopic dermatitis disease onset was identified, FLGs function has…”
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Atopic Dermatitis Studies through In Vitro Models
Published in Frontiers in medicine (24-07-2017)“…Atopic dermatitis (AD) is a complex inflammatory skin condition that is not fully understood. Epidermal barrier defects and Th2 immune response dysregulations…”
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Hyaluronidase-1 Is Mainly Functional in the Upper Granular Layer, Close to the Epidermal Barrier
Published in Journal of investigative dermatology (01-12-2015)Get full text
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Deletion of TNFAIP6 Gene in Human Keratinocytes Demonstrates a Role for TSG-6 to Retain Hyaluronan Inside Epidermis
Published in JID innovations (01-12-2021)“…TSG-6 is a soluble protein secreted in the extracellular matrix by various cell types in response to inflammatory stimuli. TSG-6 interacts with extracellular…”
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Keratinocytes activated by IL‐4/IL‐13 express IL‐2Rγ with consequences on epidermal barrier function
Published in Experimental dermatology (01-05-2023)“…Atopic dermatitis (AD) is a Th2‐type inflammatory disease characterized by an alteration of epidermal barrier following the release of IL‐4 and IL‐13. These…”
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