Search Results - "Etemad, Behzad"

Non-invasive plasma glycomic and metabolic biomarkers of post-treatment control of HIV by Giron, Leila B., Palmer, Clovis S., Liu, Qin, Yin, Xiangfan, Papasavvas, Emmanouil, Sharaf, Radwa, Etemad, Behzad, Damra, Mohammad, Goldman, Aaron R., Tang, Hsin-Yao, Johnston, Rowena, Mounzer, Karam, Kostman, Jay R., Tebas, Pablo, Landay, Alan, Montaner, Luis J., Jacobson, Jeffrey M., Li, Jonathan Z., Abdel-Mohsen, Mohamed

“…Non-invasive biomarkers that predict HIV remission after antiretroviral therapy (ART) interruption are urgently needed. Such biomarkers can improve the safety…”
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Learning From the Exceptions: HIV Remission in Post-treatment Controllers by Etemad, Behzad, Esmaeilzadeh, Elmira, Li, Jonathan Z

“…Among the top priorities of the HIV field is the search for therapeutic interventions that can lead to sustained antiretroviral therapy (ART)-free HIV…”
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Detection and manipulation of live antigen-expressing cells using conditionally stable nanobodies by Tang, Jonathan Cy, Drokhlyansky, Eugene, Etemad, Behzad, Rudolph, Stephanie, Guo, Binggege, Wang, Sui, Ellis, Emily G, Li, Jonathan Z, Cepko, Constance L

“…The ability to detect and/or manipulate specific cell populations based upon the presence of intracellular protein epitopes would enable many types of studies…”
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Human Immunodeficiency Virus Type 1 V1-to-V5 Envelope Variants from the Chronic Phase of Infection Use CCR5 and Fuse More Efficiently than Those from Early after Infection by ETEMAD, Behzad, FELLOWS, Angela, KWAMBANA, Brenda, KAMAT, Anupa, YANG FENG, LEE, Sandra, SAGAR, Manish

“…Article Usage Stats Services JVI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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An Envelope Domain III-based Chimeric Antigen Produced in Pichia pastoris Elicits Neutralizing Antibodies Against All Four Dengue Virus Serotypes by Etemad, Behzad, Batra, Gaurav, Raut, Rajendra, Dahiya, Satinder, Khanam, Saima, Swaminathan, Sathyamangalam, Khanna, Navin

“…There is currently no vaccine to prevent dengue (DEN) virus infection, which is caused by any one of four closely related serotypes, DEN-1, DEN-2, DEN-3, or…”
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Humoral Immune Pressure Selects for HIV-1 CXC-chemokine Receptor 4-using Variants by Lin, Nina, Gonzalez, Oscar A., Registre, Ludy, Becerril, Carlos, Etemad, Behzad, Lu, Hong, Wu, Xueling, Lockman, Shahin, Essex, Myron, Moyo, Sikhulile, Kuritzkes, Daniel R., Sagar, Manish

“…Although both C-C chemokine receptor 5 (CCR5)- and CXC chemokine receptor 4 (CXCR4)-using HIV-1 strains cause AIDS, the emergence of CXCR4-utilizing variants…”
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Impact of HLA Class I Alleles on Timing of HIV Rebound After Antiretroviral Treatment Interruption by Park, You Jeong, Etemad, Behzad, Ahmed, Hayat, Naranbhai, Vivek, Aga, Evgenia, Bosch, Ronald J, Mellors, John W, Kuritzkes, Daniel R, Para, Michael, Gandhi, Rajesh T, Carrington, Mary, Li, Jonathan Z

“…Identifying host determinants associated with HIV reservoir size and timing of viral rebound after an analytic treatment interruption (ATI) is an important…”
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SARS-CoV-2 viral load is associated with increased disease severity and mortality by Fajnzylber, Jesse, Regan, James, Coxen, Kendyll, Corry, Heather, Wong, Colline, Rosenthal, Alexandra, Worrall, Daniel, Giguel, Francoise, Piechocka-Trocha, Alicja, Atyeo, Caroline, Fischinger, Stephanie, Chan, Andrew, Flaherty, Keith T., Hall, Kathryn, Dougan, Michael, Ryan, Edward T., Gillespie, Elizabeth, Chishti, Rida, Li, Yijia, Jilg, Nikolaus, Hanidziar, Dusan, Baron, Rebecca M., Baden, Lindsey, Tsibris, Athe M., Armstrong, Katrina A., Kuritzkes, Daniel R., Alter, Galit, Walker, Bruce D., Yu, Xu, Li, Jonathan Z.

“…The relationship between SARS-CoV-2 viral load and risk of disease progression remains largely undefined in coronavirus disease 2019 (COVID-19). Here, we…”
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The size of the expressed HIV reservoir predicts timing of viral rebound after treatment interruption by Li, Jonathan Z, Etemad, Behzad, Ahmed, Hayat, Aga, Evgenia, Bosch, Ronald J, Mellors, John W, Kuritzkes, Daniel R, Lederman, Michael M, Para, Michael, Gandhi, Rajesh T

“…OBJECTIVES:Therapies to achieve sustained antiretroviral therapy-free HIV remission will require validation in analytic treatment interruption (ATI) trials…”
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Drug resistance mutations in HIV provirus are associated with defective proviral genomes with hypermutation by Li, Yijia, Etemad, Behzad, Dele-Oni, Ruth, Sharaf, Radwa, Gao, Ce, Lichterfeld, Mathias, Li, Jonathan Z.

“…HIV proviral sequencing overcomes the limit of plasma viral load requirement by detecting all the 'archived mutations', but the clinical relevance remains to…”
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SARS-CoV-2 Placentitis Associated With B.1.617.2 (Delta) Variant and Fetal Distress or Demise by Shook, Lydia L, Brigida, Sara, Regan, James, Flynn, James P, Mohammadi, Abbas, Etemad, Behzad, Siegel, Molly R, Clapp, Mark A, Li, Jonathan Z, Roberts, Drucilla J, Edlow, Andrea G

“…Abstract There is limited information on the specific impact of maternal infection with the SARS-CoV-2 B.1.617.2 (delta) variant on pregnancy outcomes. We…”
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Drug resistance mutations in HIV provirus are associated with defective proviral genomes with hypermutation by Li, Yijia, Etemad, Behzad, Dele-Oni, Ruth, Sharaf, Radwa, Gao, Charlie C., Yu, Xu, Li, Jonathan Z.

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Rapid, label-free CD4 testing using a smartphone compatible device by Kanakasabapathy, Manoj Kumar, Pandya, Hardik J, Draz, Mohamed Shehata, Chug, Manjyot Kaur, Sadasivam, Magesh, Kumar, Shreya, Etemad, Behzad, Yogesh, Vinish, Safavieh, Mohammadali, Asghar, Waseem, Li, Jonathan Z, Tsibris, Athe M, Kuritzkes, Daniel R, Shafiee, Hadi

“…The most recent guidelines have called for a significant shift towards viral load testing for HIV/AIDS management in developing countries; however…”
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HIV-1 proviral landscapes distinguish posttreatment controllers from noncontrollers by Sharaf, Radwa, Lee, Guinevere Q, Sun, Xiaoming, Etemad, Behzad, Aboukhater, Layla M, Hu, Zixin, Brumme, Zabrina L, Aga, Evgenia, Bosch, Ronald J, Wen, Ying, Namazi, Golnaz, Gao, Ce, Acosta, Edward P, Gandhi, Rajesh T, Jacobson, Jeffrey M, Skiest, Daniel, Margolis, David M, Mitsuyasu, Ronald, Volberding, Paul, Connick, Elizabeth, Kuritzkes, Daniel R, Lederman, Michael M, Yu, Xu G, Lichterfeld, Mathias, Li, Jonathan Z

“…HIV posttreatment controllers (PTCs) represent a natural model of sustained HIV remission, but they are rare and little is known about their viral reservoir…”
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Autologous neutralizing antibodies increase with early antiretroviral therapy and shape HIV rebound after treatment interruption by Esmaeilzadeh, Elmira, Etemad, Behzad, Lavine, Christy L, Garneau, Lauren, Li, Yijia, Regan, James, Wong, Colline, Sharaf, Radwa, Connick, Elizabeth, Volberding, Paul, Sagar, Manish, Seaman, Michael S, Li, Jonathan Z

“…Early initiation of antiretroviral therapy (ART) alters viral rebound kinetics after analytic treatment interruption (ATI) and may play a role in promoting…”
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HIV post-treatment controllers have distinct immunological and virological features by Etemad, Behzad, Sun, Xiaoming, Li, Yijia, Melberg, Meghan, Moisi, Daniela, Gottlieb, Rachel, Ahmed, Hayat, Aga, Evgenia, Bosch, Ronald J, Acosta, Edward P, Yuki, Yuko, Martin, Maureen P, Carrington, Mary, Gandhi, Rajesh T, Jacobson, Jeffrey M, Volberding, Paul, Connick, Elizabeth, Mitsuyasu, Ronald, Frank, Ian, Saag, Michael, Eron, Joseph J, Skiest, Daniel, Margolis, David M, Havlir, Diane, Schooley, Robert T, Lederman, Michael M, Yu, Xu G, Li, Jonathan Z

“…HIV post-treatment controllers (PTCs) are rare individuals who maintain low levels of viremia after stopping antiretroviral therapy (ART). Understanding the…”
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Transcriptomic Signatures of Human Immunodeficiency Virus Post-Treatment Control by Wedrychowski, Adam, Martin, Holly Anne, Li, Yijia, Telwatte, Sushama, Kadiyala, Gayatri Nikhila, Melberg, Meghan, Etemad, Behzad, Connick, Elizabeth, Jacobson, Jeffrey M, Margolis, David M, Skiest, Daniel, Volberding, Paul, Hecht, Frederick, Deeks, Steven, Wong, Joseph K, Li, Jonathan Z, Yukl, Steven A

“…Posttreatment controllers (PTCs) are rare HIV-infected individuals who can limit viral rebound after antiretroviral therapy interruption (ATI), but the…”
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Viral and host mediators of non-suppressible HIV-1 viremia by Mohammadi, Abbas, Etemad, Behzad, Zhang, Xin, Li, Yijia, Bedwell, Gregory J., Sharaf, Radwa, Kittilson, Autumn, Melberg, Meghan, Crain, Charles R., Traunbauer, Anna K., Wong, Colline, Fajnzylber, Jesse, Worrall, Daniel P., Rosenthal, Alex, Jordan, Hannah, Jilg, Nikolaus, Kaseke, Clarety, Giguel, Francoise, Lian, Xiaodong, Deo, Rinki, Gillespie, Elisabeth, Chishti, Rida, Abrha, Sara, Adams, Taylor, Siagian, Abigail, Dorazio, Dominic, Anderson, Peter L., Deeks, Steven G., Lederman, Michael M., Yawetz, Sigal, Kuritzkes, Daniel R., Lichterfeld, Mathias D., Sieg, Scott, Tsibris, Athe, Carrington, Mary, Brumme, Zabrina L., Castillo-Mancilla, Jose R., Engelman, Alan N., Gaiha, Gaurav D., Li, Jonathan Z.

“…Non-suppressible HIV-1 viremia (NSV) is defined as persistent low-level viremia on antiretroviral therapy (ART) without evidence of ART non-adherence or…”
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Transmembrane Domain Membrane Proximal External Region but Not Surface Unit-Directed Broadly Neutralizing HIV-1 Antibodies Can Restrict Dendritic Cell-Mediated HIV-1 Trans-infection by Sagar, Manish, Akiyama, Hisashi, Etemad, Behzad, Ramirez, Nora, Freitas, Ines, Gummuluru, Suryaram

“…Background. Although broadly neutralizing antibodies (bNAbs) have been shown to block a diverse array of cell-free human immunideficiency type 1 (HIV-1)…”
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Genome‐wide association analysis of COVID‐19 mortality risk in SARS‐CoV‐2 genomes identifies mutation in the SARS‐CoV‐2 spike protein that colocalizes with P.1 of the Brazilian strain by Hahn, Georg, Wu, Chloe M., Lee, Sanghun, Lutz, Sharon M., Khurana, Surender, Baden, Lindsey R., Haneuse, Sebastien, Qiao, Dandi, Hecker, Julian, DeMeo, Dawn L., Tanzi, Rudolph E., Choudhary, Manish C., Etemad, Behzad, Mohammadi, Abbas, Esmaeilzadeh, Elmira, Cho, Michael H., Li, Jonathan Z., Randolph, Adrienne G., Laird, Nan M., Weiss, Scott T., Silverman, Edwin K., Ribbeck, Katharina, Lange, Christoph

“…SARS‐CoV‐2 mortality has been extensively studied in relation to host susceptibility. How sequence variations in the SARS‐CoV‐2 genome affect pathogenicity is…”
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