Search Results - "Escobar, Gladys P"
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Caveolin-1 deletion exacerbates cardiac interstitial fibrosis by promoting M2 macrophage activation in mice after myocardial infarction
Published in Journal of molecular and cellular cardiology (01-11-2014)“…Abstract Adverse remodeling following myocardial infarction (MI) leading to heart failure is driven by an imbalanced resolution of inflammation. The macrophage…”
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Proteomic analysis identifies in vivo candidate matrix metalloproteinase-9 substrates in the left ventricle post-myocardial infarction
Published in Proteomics (Weinheim) (01-06-2010)“…Matrix metalloproteinase-9 (MMP-9) deletion has been shown to improve remodeling of the left ventricle post-myocardial infarction (MI), but the mechanisms to…”
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In vivo Matrix Metalloproteinase-7 Substrates Identified in the Left Ventricle Post-Myocardial Infarction Using Proteomics
Published in Journal of proteome research (07-05-2010)“…Matrix metalloproteinase-7 (MMP-7) deletion has been shown to improve survival after myocardial infarction (MI). MMP-7 has a large array of in vitro…”
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Cardiorenal effects of adenosine subtype 1 (A 1) receptor inhibition in an experimental model of heart failure
Published in Journal of the American College of Surgeons (01-05-2002)“…BACKGROUND: Elaboration of a number of bioactive substances, including adenosine, occurs in heart failure (HF). Adenosine, through the adenosine subtype 1 (A…”
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Proteomic analysis identifies in vivo candidate matrix metalloproteinase-9 substrates in the left ventricle post-myocardial infarction
Published in Proteomics. Clinical applications (01-11-2010)“…This article was originally published in Proteomics 2010, 10, 2214–2223, DOI 10.1002/pmic.200900587…”
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Cardiorenal Effects of Adenosine Subtype 1 (A1) Receptor Inhibition in an Experimental Model of Heart Failure1
Published in Journal of the American College of Surgeons (01-05-2002)Get full text
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