Search Results - "Ennis, I. L."

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  1. 1

    Stretch-Induced Alkalinization of Feline Papillary Muscle: An Autocrine-Paracrine System by Cingolani, H.E, Alvarez, B.V, Ennis, I.L, Camilion de Hurtado, NA

    Published in Circulation research (19-10-1998)
    “…Myocardial stretch is a well-known stimulus that leads to hypertrophy. Little is known, however, about the intracellular pathways involved in the transmission…”
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  2. 2

    Cardioprotective role of IGF‐1 in the hypertrophied myocardium of the spontaneously hypertensive rats: A key effect on NHE‐1 activity by Yeves, A. M., Burgos, J. I., Medina, A. J., Villa‐Abrille, M. C., Ennis, I. L.

    Published in Acta Physiologica (01-10-2018)
    “…Aim Myocardial Na+/H+ exchanger‐1 (NHE‐1) hyperactivity and oxidative stress are interrelated phenomena playing pivotal roles in the development of…”
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  3. 3

    Na+/H+ exchanger and cardiac hypertrophy by Yeves, A.M., Ennis, I.L.

    Published in Hipertensión y riesgo vascular (01-01-2020)
    “…Reactive cardiac hypertrophy (CH) is an increase in heart mass in response to hemodynamic overload. Exercise-induced CH emerges as an adaptive response with…”
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  4. 4

    The RAAS and SARS-CoV-2: A riddle to solve by Choi, M, Aiello, E A, Ennis, I L, Villa-Abrille, M C

    Published in Hipertension y riesgo vascular (01-10-2020)
    “…The first case of COVID-19 was reported on 31 December 2019 in Wuhan, China. Ever since there has been unprecedented and growing interest in learning about all…”
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    Mechanisms Underlying the Increase in Force and Ca2+ Transient That Follow Stretch of Cardiac Muscle: A Possible Explanation of the Anrep Effect by Alvarez, Bernardo V, Pérez, Néstor G, Ennis, Irene L, de Hurtado, María C. Camilión, Cingolani, Horacio E

    Published in Circulation research (15-10-1999)
    “…Myocardial stretch produces an increase in developed force (DF) that occurs in two phasesthe first (rapidly occurring) is generally attributed to an increase…”
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    Mitochondrial reactive oxygen species (ROS) as signaling molecules of intracellular pathways triggered by the cardiac renin-angiotensin II-aldosterone system (RAAS) by De Giusti, V C, Caldiz, C I, Ennis, I L, Pérez, N G, Cingolani, H E, Aiello, E A

    Published in Frontiers in physiology (2013)
    “…Mitochondria represent major sources of basal reactive oxygen species (ROS) production of the cardiomyocyte. The role of ROS as signaling molecules that…”
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  8. 8

    The signaling pathway for aldosterone-induced mitochondrial production of superoxide anion in the myocardium by Nolly, M.B, Caldiz, C.I, Yeves, A.M, Villa-Abrille, M.C, Morgan, P.E, Amado Mondaca, Nicolas, Portiansky, Enrique L, Chiappe de Cingolani, G.E, Cingolani, H.E, Ennis, I.L

    “…Abstract Mineralocorticoid receptor (MR) antagonists decrease morbidity and mortality in heart failure patients for whom oxidative stress is usual; however,…”
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  9. 9

    Angiotensin II Activates Na+-Independent Cl--HCO3- Exchange in Ventricular Myocardium by de Hurtado, Maria C. Camilion, Alvarez, Bernardo V, Perez, Nestor G, Ennis, Irene L, Cingolani, Horacio E

    Published in Circulation research (09-03-1998)
    “…The effect of angiotensin II (Ang II) on the activity of the cardiac Na Cl3 exchanger (anionic exchanger [AE]) was explored in cat papillary muscles. pHi was…”
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  10. 10

    New method for the devitalization of imported horsetail (Equisetum hyemale) by Blanchon, DJ, Ennis, IL, Lewthwaite, JR, Large, MF, Bussell, WT

    “…Despite being rated as one of the world's worst weeds, Equisetum hyemale was until recently imported into New Zealand for use by the floristry industry,…”
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  11. 11

    Regression of Isoproterenol-Induced Cardiac Hypertrophy by Na+/H+ Exchanger Inhibition by Ennis, Irene L, Escudero, Eduardo M, Console, Gloria M, Camihort, Gisela, Dumm, César Gomez, Seidler, Randolph W, Camilión de Hurtado, María C, Cingolani, Horacio E

    Published in Hypertension (Dallas, Tex. 1979) (01-06-2003)
    “…ABSTRACT—Cardiac hypertrophy is often associated with an increased sympathetic drive, and both in vitro and in vivo studies have demonstrated the development…”
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  12. 12

    Novel structural determinants of mu-conotoxin (GIIIB) block in rat skeletal muscle (mu1) Na+ channels by Li, R A, Ennis, I L, Vélez, P, Tomaselli, G F, Marbán, E

    Published in The Journal of biological chemistry (08-09-2000)
    “…mu-Conotoxin (mu-CTX) specifically occludes the pore of voltage-dependent Na(+) channels. In the rat skeletal muscle Na(+) channel (mu1), we examined the…”
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  13. 13

    Stimulation of myocardial Na(+)-independent Cl(-)-HCO(3)(-) exchanger by angiotensin II is mediated by endogenous endothelin by de Hurtado, M C, Alvarez, B V, Ennis, I L, Cingolani, H E

    Published in Circulation research (31-03-2000)
    “…Experiments were performed in isolated cat papillary muscles loaded with the pH-sensitive dye 2', 7'-bis(2-carboxyethyl)-5(6)-carboxyfluorescein in the…”
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    Latent Specificity of Molecular Recognition in Sodium Channels Engineered To Discriminate between Two “Indistinguishable” μ-Conotoxins by Li, Ronald A, Ennis, Irene L, Tomaselli, Gordon F, French, Robert J, Marbán, Eduardo

    Published in Biochemistry (Easton) (22-05-2001)
    “…μ-Conotoxins (μ-CTX) are potent oligopeptide blockers of sodium channels. The best characterized forms of μ-CTX, GIIIA and GIIIB, have similar primary and…”
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  15. 15

    Influence of Na+-Independent Cl−-HCO3− Exchange on the Slow Force Response to Myocardial Stretch by Cingolani, Horacio E, Chiappe, Gladys E, Ennis, Irene L, Morgan, Patricio G, Alvarez, Bernardo V, Casey, Joseph R, Dulce, Raúl A, Pérez, Néstor G, Camilión de Hurtado, María C

    Published in Circulation research (28-11-2003)
    “…ABSTRACT—Previous work demonstrated that the slow force response (SFR) to stretch is due to the increase in calcium transients (CaT) produced by an…”
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  16. 16

    Enalapril induces regression of cardiac hypertrophy and normalization of pHi regulatory mechanisms by ENNIS, I. L, ALVAREZ, B. V, CAMILION DE HURTADO, M. C, CINGOLANI, H. E

    Published in Hypertension (Dallas, Tex. 1979) (01-04-1998)
    “…Intracellular pH is under strict control in myocardium; H+ are extruded from the cells by sodium-dependent mechanisms, mainly Na+/H+ exchanger and Na+/HCO3-…”
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    Enalapril Induces Regression of Cardiac Hypertrophy and Normalization of pH (i) Regulatory Mechanisms by Ennis, Irene L, Alvarez, Bernardo V, de Hurtado, Maria C. Camilion, Cingolani, Horacio E

    Published in Hypertension (Dallas, Tex. 1979) (01-04-1998)
    “…Intracellular pH is under strict control in myocardium; H are extruded from the cells by sodium-dependent mechanisms, mainly Na exchanger and Na3 symport,…”
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    Journal Article
  18. 18

    Mechanisms underlying the increase in force and Ca(2+) transient that follow stretch of cardiac muscle: a possible explanation of the Anrep effect by Alvarez, B V, Pérez, N G, Ennis, I L, Camilión de Hurtado, M C, Cingolani, H E

    Published in Circulation research (15-10-1999)
    “…Myocardial stretch produces an increase in developed force (DF) that occurs in two phases: the first (rapidly occurring) is generally attributed to an increase…”
    Get full text
    Journal Article
  19. 19

    Prevalence of hypertension in 3154 young students by Ennis, I L, Gende, O A, Cingolani, H E

    Published in Medicina (Buenos Aires) (1998)
    “…Blood pressure (BP) levels were evaluated in 3154 students (mean age 21 years old) of La Plata University, School of Medicine. BP was registered three times in…”
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