Search Results - "Eisenbach, L"

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    O‐glycosylated versus non‐glycosylated MUC1‐derived peptides as potential targets for cytotoxic immunotherapy of carcinoma by Stepensky, D., Tzehoval, E., Vadai, E., Eisenbach, L.

    Published in Clinical and experimental immunology (01-01-2006)
    “…Summary Due to the fact that many cellular proteins are extensively glycosylated, processing and presentation mechanisms are expected to produce a pool of…”
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    Overexpression of a set of genes, including WISP-1, common to pulmonary metastases of both mouse D122 Lewis lung carcinoma and B16-F10.9 melanoma cell lines by Margalit, O, Eisenbach, L, Amariglio, N, Kaminski, N, Harmelin, A, Pfeffer, R, Shohat, M, Rechavi, G, Berger, R

    Published in British journal of cancer (21-07-2003)
    “…Despite advances in the management of solid tumours, the development of metastases continues to be the most significant problem and cause of death for cancer…”
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    Antimetastatic vaccination of tumor-bearing mice with two types of IFN- gamma gene-inserted tumor cells by Porgador, A, Bannerji, R, Watanabe, Y, Feldman, M, Gilboa, E, Eisenbach, L

    Published in The Journal of immunology (1950) (15-02-1993)
    “…IFN-gamma genes were introduced through retroviral vectors into the high metastatic, low H-2Kb class I expressor, and poorly immunogenic 3LL-D122 clone. Two…”
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    MAGE-A8 overexpression in transitional cell carcinoma of the bladder: identification of two tumour-associated antigen peptides by BAR-HAIM, E, PAZ, A, LEMONNIER, F. A, TZEHOVAL, E, EISENBACH, L, MACHLENKIN, A, HAZZAN, D, TIROSH, B, CARMON, L, BRENNER, B, VADAI, E, MOR, O, STEIN, A

    Published in British journal of cancer (19-07-2004)
    “…Bladder carcinoma is the fourth most common cancer in men and the eighth most common cancer among women. Our study is aimed to characterise tumour-associated…”
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    Developmental expression of the alpha receptor for platelet-derived growth factor, which is deleted in the embryonic lethal Patch mutation by Orr-Urtreger, A, Bedford, M T, Do, M S, Eisenbach, L, Lonai, P

    Published in Development (Cambridge) (01-05-1992)
    “…The alpha receptor of PDGF (Pdgfra) is expressed in primitive endoderm and mesoderm derivatives throughout embryogenesis. In the early primitive streak stage…”
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    Identification of shared tumor-associated antigen peptides between two spontaneous lung carcinomas by Mandelboim, O, Bar-Haim, E, Vadai, E, Fridkin, M, Eisenbach, L

    Published in The Journal of immunology (1950) (15-12-1997)
    “…CTLs recognize antigenic peptides bound to MHC class I Ags on the cell surface of tumor cells. Tumor-associated Ag (TAA) peptides are 8 to 10 amino acids long…”
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    Unmasking by soluble IL-6 receptor of IL-6 effect on metastatic melanoma : growth inhibition and differentiation of B16-F10.9 tumor cells by OH, J.-W, KATZ, A, HARROCH, S, EISENBACH, L, REVEL, M, CHEBATH, J

    Published in Oncogene (31-07-1997)
    “…Interleukin-6 (IL-6) inhibits the growth of melanocytes and of early stage melanoma cells, but not that of advanced melanoma cells. The in vitro IL-6 response…”
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    c-fos and c-jun overexpression in malignant cells reduces their tumorigenic and metastatic potential, and affects their MHC class I gene expression by Yamit-Hezi, A, Plaksin, D, Eisenbach, L

    Published in Oncogene (01-04-1994)
    “…Reduced co-expression of the c-fos and c-jun protooncogenes has been correlated with the down regulation of H-2K class I major histocompatibility antigens in…”
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    Expression of two H-2K genes, syngeneic and allogeneic, as a strategy for potentiating immune recognition of tumor cells by Mandelboim, O, Vadai, E, Feldman, M, Eisenbach, L

    Published in Gene therapy (01-12-1995)
    “…Metastatic clones of some tumors manifest an impaired expression of class I major histocompatibility complex (MHC) antigens. High metastatic, low immunogenic…”
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    Reversal of the Metastatic Phenotype in Lewis Lung Carcinoma Cells after Transfection with Syngeneic H-2Kb Gene by Plaksin, Daniel, Gelber, Cochava, Feldman, Michael, Eisenbach, Lea

    “…High metastatic clones of the murine 3LL carcinoma express greatly reduced levels of H-2Kb major histocompatibility complex class I antigens, while low…”
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    Antigenicity and immunogenicity of an intracellular delivery system of major histocompatibility complex class I epitopes that bypasses proteasome processing by TIROSH, Boaz, FRIDKIN, Mati, TZEHOVAL, Eather, VADAI, Ezra, LEMONNIER, Francois A, EISENBACH, Lea

    Published in Journal of immunotherapy (1997) (01-11-2000)
    “…The development of a cell-free synthetic vaccine to induce an effective cytotoxic T lymphocyte response is an important challenge in T-cell--mediated immunity…”
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    A Novel Lytic Peptide Composed of dl-Amino Acids Selectively Kills Cancer Cells in Culture and in Mice by Papo, Niv, Shahar, Michal, Eisenbach, Lea, Shai, Yechiel

    Published in The Journal of biological chemistry (06-06-2003)
    “…The high toxicity of most chemotherapeutic drugs and their inactivation by multidrug resistance phenotypes motivated extensive search for drugs with new modes…”
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    Immunotherapy of tumor metastasis via gene therapy by Porgador, A, Feldman, M, Eisenbach, L

    Published in Natural immunity (01-03-1994)
    “…Active cellular immunotherapy of cancer, via gene-modified tumor cells, may develop to an effective treatment modality. Here we discuss some of the cytokines…”
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    c-fos transfection of 3LL tumor cells turns on MHC gene expression and consequently reduces their metastatic competence by Kushtai, G, Feldman, M, Eisenbach, L

    Published in International journal of cancer (15-06-1990)
    “…Transfection with c-fos genes of cells of a highly metastatic (H-2K- H-2D+) clone, DI22, of the 3LL carcinoma, causes activation of H-2K gene expression…”
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    The c-fos proto-oncogene in murine 3LL carcinoma clones controls the expression of MHC genes by Kushtai, G, Barzilay, J, Feldman, M, Eisenbach, L

    Published in Oncogene (01-02-1988)
    “…The c-fos proto-oncogene and H-2K class I major histocompatibility antigens are differentially expressed in low-metastatic Lewis lung carcinoma clones, but not…”
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