Search Results - "EMMANUELE, G."

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    On relative compactness in K(X,Y) by Emmanuele, G.

    “…We give a sufficient condition for the relative compactness of subsets of K(X,Y) that is similar to an 11-year-old result obtained by Mayoral, but actually…”
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    Autosomal recessive hypercholesterolemia in a Sicilian kindred harboring the 432insA mutation of the ARH gene by Barbagallo, C.M., Emmanuele, G., Cefalù, A.B., Fiore, B., Noto, D., Mazzarino, M.C., Pace, A., Brogna, A., Rizzo, M., Corsini, A., Notarbartolo, A., Travali, S., Averna, M.R.

    Published in Atherosclerosis (01-02-2003)
    “…We describe a Sicilian family presenting a recessive form of hypercholesterolemia harboring a mutation of the autosomal recessive hypercholesterolemia (ARH)…”
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    On complemented copies of c0 and ℓ by Emmanuele, G.

    Published in Acta mathematica Hungarica (01-04-2014)
    “…We furnish examples of pairs of Banach spaces X , Y so that none of c 0 and  l ∞ live inside X ∗ and  Y , but they embed complementably into the space DP ( X ,…”
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    New Model of Liver Regeneration Induced Through Use of Vascular Endothelial Growth Factor by Marino, G., Piazzese, E., Gruttadauria, S., Nicotra, G., Guarnaccia, M., Emmanuele, G., Bartoloni, G., Messina, A., Travali, S., Famulari, C., Gruttadauria, G.

    Published in Transplantation proceedings (01-05-2006)
    “…Vascular endothelial growth factor (VEGF) is an endothelial cell mitogen. The objective of this study was to verify the proregenerative effects of VEGF in an…”
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    The C(-260)>T gene polymorphism in the promoter of the CD14 monocyte receptor gene is not associated with acute myocardial infarction by LONGOBARDO, M. T, CEFALU, A. B, NOTARBARTOLO, A, TRAVALI, S, AVERNA, M. R, PEZZINO, F, NOTO, D, EMMANUELE, G, BARBAGALLO, C. M, FIORE, B, MONASTERO, R, CASTELLO, A, MOLINI, V

    Published in Clinical and experimental medicine (01-11-2003)
    “…CD surface molecules mediates cell activation and signaling. In particular, CD14 on blood monocytes mediate monocyte/macrophage activation by…”
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    Effectiveness of screening for known mutations in Sicilian patients with "probable" familial hypercholesterolemia by Cefalù, A B, Emmanuele, G, Marino, G, Fiore, B, Caldarella, R, Vivona, N, Noto, D, Barbagallo, C M, Costa, S, Gueli, M C, Bertolini, S, Notarbartolo, A, Travali, S, Averna, M R

    “…More than 750 mutations in the low-density lipoprotein (LDL) receptor gene are currently known to cause familial hypercholesterolemia (FH), but the array of…”
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    Rapid screening of the LDL receptor point mutation FH-Genoa/Palermo. Mutation in brief no. 238. Online by Marino, G, Travali, S, Reyes, T, Wallace, B R, Caldarella, R, Emmanuele, G, Stivala, F, Barbagallo, C M, Cantafora, A, Bertolini, S, Notarbartolo, A, Averna, M

    Published in Human mutation (1999)
    “…The LDL-receptor gene point mutation FH-Genoa/Palermo is the most frequent mutation responsible for Familial Hypercholesterolemia in Sicily. The mutation does…”
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    Phase diagram of the disordered Kitaev chain with long range pairing connected to external baths by Cinnirella, Emmanuele G, Nava, Andrea, Campagnano, Gabriele, Giuliano, Domenico

    Published 14-11-2024
    “…We study the interplay between topology and disorder in the disodered Kitaev model with long range pairing, connected to two metallic leads exchanging…”
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    The fate of high winding number topological phases in the disordered extended Su-Schrieffer-Heeger model by Cinnirella, Emmanuele G, Nava, Andrea, Campagnano, Gabriele, Giuliano, Domenico

    Published 30-01-2024
    “…Physical Review B 109, 035114 (2024) We use the Lindblad equation approach to investigate topological phases hosting more than one localized state at each side…”
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    IL‐1β, TNF‐α and IL‐6 release from monocytes in haemodialysis patients in relation to dialytic age by Malaponte, Grazia, Bevelacqua, Valentina, Fatuzzo, Pasquale, Rapisarda, Francesco, Emmanuele, Giovanni, Travali, Salvatore, Mazzarino, Maria Clorinda

    Published in Nephrology, dialysis, transplantation (01-11-2002)
    “…Background. It has been suggested that changes in immune response to infectious agents in patients on haemodialysis might be due to impaired monocyte function;…”
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