Search Results - "Dwenger, Marc M."
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Pyridine nucleotide redox potential in coronary smooth muscle couples myocardial blood flow to cardiac metabolism
Published in Nature communications (19-04-2022)“…Adequate oxygen delivery to the heart during stress is essential for sustaining cardiac function. Acute increases in myocardial oxygen demand evoke coronary…”
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Diversification of Potassium Currents in Excitable Cells via Kvβ Proteins
Published in Cells (Basel, Switzerland) (18-07-2022)“…Excitable cells of the nervous and cardiovascular systems depend on an assortment of plasmalemmal potassium channels to control diverse cellular functions…”
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Basal NAD(H) redox state permits hydrogen peroxide‐induced mesenteric artery dilatation
Published in The Journal of physiology (01-07-2023)“…Smooth muscle voltage‐gated K+ (Kv) channels in resistance arteries control vascular tone and contribute to the coupling of blood flow with local metabolic…”
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Integrated redox sensitivity of vascular Kv1 channels
Published in The FASEB journal (01-05-2022)“…Voltage‐gated potassium (Kv) channels in vascular smooth muscle are responsive to a range of molecular signals that link local metabolic demand to blood…”
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5
Coronary microvascular Kv1 channels as regulatory sensors of intracellular pyridine nucleotide redox potential
Published in Microcirculation (New York, N.Y. 1994) (01-01-2018)“…Smooth muscle voltage‐gated potassium (Kv) channels are important regulators of microvascular tone and tissue perfusion. Recent studies indicate that Kv1…”
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Myocardial Blood Flow Control by Oxygen Sensing Vascular Kvβ Proteins
Published in Circulation research (19-03-2021)“…[Figure: see text]…”
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Role for NADH‐sensitive Kv channels in the myocardial‐vascular signaling axis
Published in The FASEB journal (01-04-2020)“…Abstract only…”
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Abstract 16845: Functional Regulation of Native Coronary Kv1 Channels by Pyridine Nucleotides
Published in Circulation (New York, N.Y.) (06-11-2018)“…IntroductionRecent studies indicate that vasodilation and enhanced blood flow to the heart following increases in myocardial O2demand are mediated by…”
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