Search Results - "Dumans, Ana T."

Synonymous Genetic Polymorphisms within Brazilian Human Immunodeficiency Virus Type 1 Subtypes May Influence Mutational Routes to Drug Resistance by Dumans, Ana T., Soares, Marcelo A., Machado, Elizabeth S., Hué, Stéphane, Brindeiro, Rodrigo M., Pillay, Deenan, Tanuri, Amilcar

“…Background. Most published data on antiretroviral-drug resistance is generated from in vitro or in vivo studies of subtype B virus. However, this subtype is…”
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Prevalence of Protease and Reverse Transcriptase Drug Resistance Mutations over Time in Drug-Naïve Human Immunodeficiency Virus Type 1-Positive Individuals in Rio de Janeiro, Brazil by DUMANS, Ana T, SOARES, Marcelo A, PIENIAZEK, Danuta, KALISH, Marcia L, DE VROEY, Veronique, HERTOGS, Kurt, TANURI, Amilcar

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Different epidemic potentials of the HIV-1B and C subtypes by Salemi, Marco, de Oliveira, Tulio, Soares, Marcelo A, Pybus, Oliver, Dumans, Ana T, Vandamme, Anne-Mieke, Tanuri, Amilcar, Cassol, Sharon, Fitch, Walter M

“…HIV, the cause of AIDS in humans, is characterized by great genetic heterogeneity. In particular, HIV-1 group M subtypes are responsible for most of the…”
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Distinct resistance mutation and polymorphism acquisition in HIV-1 protease of subtypes B and F1 from children and adult patients under virological failure by Dumans, Ana T., Barreto, Cláudia C., Santos, André F., Arruda, Mônica, Sousa, Thatiana M., Machado, Elizabeth S., Sabino, Ester C., Brindeiro, Rodrigo M., Tanuri, Amílcar, Duarte, Alberto J., Soares, Marcelo A.

“…The goal of this work was to compare the differences between human immunodeficiency virus type 1 (HIV-1) of B and F1 subtypes in the acquisition of major and…”
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Prevalence of Protease and Reverse Transcriptase Drug Resistance Mutations over Time in Drug-Naïve Human Immunodeficiency Virus Type 1-Positive Individuals in Rio de Janeiro, Brazil by Dumans, Ana T, Soares, Marcelo A, Pieniazek, Danuta, Kalish, Marcia L, De Vroey, Veronique, Hertogs, Kurt, Tanuri, Amilcar

“…The prevalence of mutations that confer resistance to protease inhibitors and to nucleoside and nonnucleoside reverse transcriptase inhibitors in 49 blood…”
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