Search Results - "Draoui, Muriel"

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  1. 1

    Expression and retinoid modulation of N-arginine dibasic convertase and an aminopeptidase-B in human neuroblastoma cell lines by Draoui, M, Bellincampi, L, Hospital, V, Cadel, S, Foulon, T, Prat, A, Barré, N, Reichert, U, Melino, G, Cohen, P

    Published in Journal of neuro-oncology (01-01-1997)
    “…Under retinoic acid exposure, the three SK-N-BE(2)-derived human neuroblastoma cell lines, BE(2)-NA, BE(2)-SA and BE(2)-M17 undergo mainly differentiation,…”
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    Journal Article
  2. 2

    A Vasoactive Intestinal Peptide Antagonist Inhibits Non-Small Cell Lung Cancer Growth by Moody, Terry W., Zia, Farah, Draoui, Muriel, Brenneman, Douglas E., Fridkin, Mati, Davidson, Ariane, Gozes, Illana

    “…The most prevalent lung cancer, non-small cell lung cancer (NSCLC) has receptors for vasoactive intestinal peptide (VIP). Here the effects of a VIP antagonist…”
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  3. 3

    Retinoic Acid Receptors α and γ Mediate the Induction of “Tissue” Transglutaminase Activity and Apoptosis in Human Neuroblastoma Cells by Melino, Gerry, Draoui, Muriel, Bellincampi, Lorenza, Bernassola, Francesca, Bernardini, Sergio, Piacentini, Mauro, Reichert, Uwe, Cohen, Paul

    Published in Experimental cell research (25-08-1997)
    “…All-transretinoic acid (RA) reduces human neuroblastoma growth by inducing either differentiation or apoptosis. The apoptotic program in these cells is…”
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  4. 4
  5. 5

    TGF alpha-PE40 inhibits non-small cell lung cancer growth by Draoui, M, Siegall, C B, FitzGerald, D, Pastan, I, Moody, T W

    Published in Life sciences (1973) (1994)
    “…The ability of a chimeric toxin containing transforming growth factor alpha (TGF alpha) and truncated Pseudomonas exotoxin A to inhibit NSCLC growth was…”
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  6. 6

    Regulation by retinoic acid of insulin-degrading enzyme and of a related endoprotease in human neuroblastoma cell lines by MELINO, G, DRAOUI, M, BERNARDINI, S, BELLINCAMPI, L, REICHERT, U, COHEN, P

    Published in Cell growth & differentiation (01-06-1996)
    “…Physiologically, the action of insulin-like growth factors (IGFs) is controlled at different levels, from its transcription start by tissue-specific and…”
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