Search Results - "Douglas, Bonnie B."

PD-1 and CTLA-4 exert additive control of effector regulatory T cells at homeostasis by Pereira, Joseph A, Lanzar, Zachary, Clark, Joseph T, Hart, Andrew P, Douglas, Bonnie B, Shallberg, Lindsey, O'Dea, Keenan, Christian, David A, Hunter, Christopher A

“…At homeostasis, a substantial proportion of Foxp3 T regulatory cells (T ) have an activated phenotype associated with enhanced TCR signals and these effector T…”
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PD-L1–PD-1 interactions limit effector regulatory T cell populations at homeostasis and during infection by Perry, Joseph A., Shallberg, Lindsey, Clark, Joseph T., Gullicksrud, Jodi A., DeLong, Jonathan H., Douglas, Bonnie B., Hart, Andrew P., Lanzar, Zachary, O’Dea, Keenan, Konradt, Christoph, Park, Jeongho, Kuchroo, Juhi R., Grubaugh, Daniel, Zaretsky, Arielle Glatman, Brodsky, Igor E., Malefyt, Rene de Waal, Christian, David A., Sharpe, Arlene H., Hunter, Christopher A.

“…Phenotypic and transcriptional profiling of regulatory T (T reg ) cells at homeostasis reveals that T cell receptor activation promotes T reg cells with an…”
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Characterization of Protein Tyrosine Phosphatase 1B Inhibition by Chlorogenic Acid and Cichoric Acid by Lipchock, James M, Hendrickson, Heidi P, Douglas, Bonnie B, Bird, Kelly E, Ginther, Patrick S, Rivalta, Ivan, Ten, Nicholas S, Batista, Victor S, Loria, J. Patrick

“…Protein tyrosine phosphatase 1B (PTP1B) is a known regulator of the insulin and leptin signaling pathways and is an active target for the design of inhibitors…”
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Exploring protein structure and dynamics through a project‐oriented biochemistry laboratory module by Lipchock, James M., Ginther, Patrick S., Douglas, Bonnie B., Bird, Kelly E., Patrick Loria, J.

“…Here, we present a 10‐week project‐oriented laboratory module designed to provide a course‐based undergraduate research experience in biochemistry that…”
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Antigen Specific CD4+ T Cell Responses against a Gastrointestinal Nematode by Douglas, Bonnie B

“…Helminths are distinct from microbial pathogens in terms of size and complexity, and are likely the evolutionary driving force for type 2 immunity. CD4+ helper…”
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