Search Results - "Dote, H"

Aberrant promoter methylation in human DAB2 interactive protein ( ) gene in gastrointestinal tumour by Dote, H, Toyooka, S, Tsukuda, K, Yano, M, Ota, T, Murakami, M, Naito, M, Toyota, M, Gazdar, A F, Shimizu, N

“…The human DOC-2/DAB2 interactive protein (hDAB2IP) gene is a novel member of the Ras GTPase-activating family and has been demonstrated to be a…”
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1320 Potential of heat shock protein 90 expression in the nucleus as a useful parameter for neoadjuvant chemotherapy in gastric cancer by Dote, H, Nishimura, R, Yamamoto, T, Hato, S, Nozaki, I, Kurita, A

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385 POSTER Hsp90 inhibitor synergistically potentiates the growth inhibitory and pro-apoptotic effects of SN-38 in gastric carcinoma cells by Hato, S, Dote, H, Koshimune, R, Ino, H, Naito, M, Date, H

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374 POSTER Synergic antiproliferative effect of Hsp90 inhibitor in combination with cisplatin in gastric carcinoma cell lines by Dote, H, Hato, S, Koshimune, R, Ino, H, Naito, M, Date, H

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Aberrant promoter methylation of human DAB2 interactive protein (hDAB2IP) gene in lung cancers by Yano, Masaaki, Toyooka, Shinichi, Tsukuda, Kazunori, Dote, Hideaki, Ouchida, Mamoru, Hanabata, Tetsuro, Aoe, Motoi, Date, Hiroshi, Gazdar, Adi F., Shimizu, Nobuyoshi

“…The human DOC‐2/DAB2 interactive protein gene (hDAB2IP) is a novel member of the Ras GTPase‐activating gene family that is known to act as a tumor suppressor…”
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Enhancement of In vitro and In vivo Tumor Cell Radiosensitivity by the DNA Methylation Inhibitor Zebularine by DOTE, Hideaki, CERNA, David, BURGAN, William E, CARTER, Donna J, CERRA, Michael A, HOLLINGSHEAD, Melinda G, CAMPHAUSEN, Kevin, TOFILON, Philip J

“…Aberrant DNA hypermethylation is a frequent finding in tumor cells, which has suggested that inhibition of DNA methylation may be an effective cancer treatment…”
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Aberrant Promoter Methylation in Human DAB2 Interactive Protein (hDAB2IP) Gene in Breast Cancer by DOTE, Hideaki, TOYOOKA, Shinichi, SHIMIZU, Nobuyoshi, TSUKUDA, Kazunori, YANO, Masaaki, OUCHIDA, Mamoru, DOIHARA, Hiroyoshi, SUZUKI, Makoto, CHEN, Hong, HSIEH, Jer-Tsong, GAZDAR, Adi F

“…Purpose: Human DOC-2/DAB2 interactive protein ( hDAB2IP ) gene is a novel member of the Ras GTPase-activating family and has been demonstrated to be a tumor…”
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ErbB3 expression predicts tumor cell radiosensitization induced by Hsp90 inhibition by DOTE, Hideaki, CERNA, David, BURGAN, William E, CAMPHAUSEN, Kevin, TOFILON, Philip J

“…The ability to identify tumors that are susceptible to a given molecularly targeted radiosensitizer would be of clinical benefit. Towards this end, we have…”
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Aberrant methylation of p57KIP2 gene in lung and breast cancers and malignant mesotheliomas by Kobatake, Takaya, Yano, Masaaki, Toyooka, Shinichi, Tsukuda, Kazunori, Dote, Hideaki, Kikuchi, Takefumi, Toyota, Minoru, Ouchida, Mamoru, Aoe, Motoi, Date, Hiroshi, Pass, Harvey I, Doihara, Hiroyoshi, Shimizu, Nobuyoshi

“…The p57KIP2 gene belongs to the Cip/Kip family of CDK inhibitors and has been demonstrated to be a tumor suppressor gene, being inactivated in various types of…”
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Syntheses of 12-arm star polymers and star diblock copolymers by nitroxide-mediated radical polymerization using dendritic dodecafunctional macroinitiators by Miura, Yozo, Dote, Hiroyuki

“…Dendritic multifunctional macroinitiators having 12 TEMPO‐based alkoxyamines were prepared by the reaction of a benzyl alcohol having 4 TEMPO‐based…”
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Less frequent promoter hypermethylation of DLC-1 gene in primary breast cancers by Teramoto, Atsushi, Tsukuda, Kazunori, Yano, Masaaki, Toyooka, Shinichi, Dote, Hideaki, Doihara, Hiroyoshi, Shimizu, Nobuyoshi

“…Absence or low expression of DLC-1, a tumor suppressor gene, in breast cancers has been shown recently. LOH of 8p12-p22, on which DLC-1 is located, is frequent…”
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Mutation analysis of the BRAF codon 599 in malignant pleural mesothelioma by enriched PCR-RFLP by Dote, Hideaki, Tsukuda, Kazunori, Toyooka, Shinichi, Yano, Masaaki, Pass, Harvey I, Shimizu, Nobuyoshi

“…BRAF encodes a RAS-regulated serine/threonine kinase that mediates the pathway for cell growth and malignant transformation. Point mutations of BRAF were…”
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