Search Results - "Dixit, Bhavna"

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  1. 1

    Codon optimization is an essential parameter for the efficient allotopic expression of mtDNA genes by Lewis, Caitlin J., Dixit, Bhavna, Batiuk, Elizabeth, Hall, Carter J., O'Connor, Matthew S., Boominathan, Amutha

    Published in Redox biology (01-02-2020)
    “…Mutations in mitochondrial DNA can be inherited or occur de novo leading to several debilitating myopathies with no curative option and few or no effective…”
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    Journal Article
  2. 2

    Rapid enrichment of mitochondria from mammalian cell cultures using digitonin by Dixit, Bhavna, Vanhoozer, Shon, Anti, Nana Abena, O'Connor, Matthew S., Boominathan, Amutha

    Published in MethodsX (01-01-2021)
    “…We describe here a simple method to enrich mitochondrial fractions from mammalian cells for downstream analyses in the lab. Mitochondria purification involves…”
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    Journal Article
  3. 3

    The Mitochondrial Genome in Aging and Disease and the Future of Mitochondrial Therapeutics by Saravanan, Sanjana, Lewis, Caitlin J, Dixit, Bhavna, O'Connor, Matthew S, Stolzing, Alexandra, Boominathan, Amutha

    Published in Biomedicines (18-02-2022)
    “…Mitochondria are intracellular organelles that utilize nutrients to generate energy in the form of ATP by oxidative phosphorylation. Mitochondrial DNA (mtDNA)…”
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    Successful Exogenous Expression of ATP8, a Mitochondrial Encoded Protein, From the Nucleus In Vivo by Begelman, David, Dixit, Bhavna, Lewis, Caitlin, Watson, Mark, Brand, Martin, Boominathan, Amutha

    Published in Innovation in aging (17-12-2021)
    “…Replicative errors, inefficient repair, and proximity to reactive oxygen species production sites make the mitochondrial DNA (mtDNA) susceptible to damage with…”
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    Journal Article
  7. 7

    Successful Exogenous Expression of ATP8, a Mitochondrial Encoded Protein, From the Nucleus In Vivo by Begelman, David, Brand, Martin, Boominathan, Amutha, Lewis, Caitlin, Dixit, Bhavna, Watson, Mark

    Published in Innovation in aging (17-12-2021)
    “…Replicative errors, inefficient repair, and proximity to reactive oxygen species production sites make the mitochondrial DNA (mtDNA) susceptible to damage with…”
    Get full text
    Journal Article