Search Results - "Darmon, O."

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  1. 1

    Long-lasting thrombocytopenia induced by glycoprotein IIb/IIIa inhibitor by Christen, J-R, Soubrier, C, Martinez, E, Roumieu, V, Darmon, O, Scandaliaris, S, Picou, L, Cuisset, T, Grados, A, Bernit, E, Ebbo, M, Harlé, J-R, Schleinitz, N

    Published in La revue de medecine interne (01-11-2017)
    “…Glycoprotein IIb/IIIa inhibitors (anti-GPIIbIIIa) prevent platelet binding to fibrinogen. Transient sometimes-severe thrombocytopenia is a well-known side…”
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    Journal Article
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    Thrombopénie sévère et prolongée induite par un inhibiteur de la glycoprotéine IIb/IIIa by Christen, J.-R., Soubrier, C., Martinez, E., Roumieu, V., Darmon, O., Scandaliaris, S., Picou, L., Cuisset, T., Grados, A., Bernit, E., Ebbo, M., Harlé, J.-R., Schleinitz, N.

    Published in La revue de medecine interne (01-11-2017)
    “…Les inhibiteurs de la glycoprotéine IIb/IIIa (anti-GPIIbIIIa) préviennent l’agrégation plaquettaire en empêchant leur liaison au fibrinogène. La thrombopénie…”
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    Journal Article
  5. 5

    Genetic polymorphisms of the renin-angiotensin system and angiographic extent and severity of coronary artery disease : the CORGENE study by JEUNEMAITRE, X, LEDRU, F, DUCIMETIERE, P, BATTAGLIA, S, GUILLANNEUF, M.-T, COURBON, D, DUMONT, C, DARMON, O, GUIZE, L, GUERMONPREZ, J.-L, DIEBOLD, B

    Published in Human genetics (1997)
    “…Genetic polymorphisms of the renin-angiotensin system (RAS) have been associated with coronary artery disease (CAD) but no relation between these polymorphisms…”
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    Journal Article
  6. 6

    Characterization of the specific response to serotonin of mouse tumour-feeding arterioles by LAEMMEL, E, STÜCKER, O, DARMON, P.-L, VICAUT, E

    “…Purpose: To investigate the role of tumour versus non-tumour factors in the specific response to serotonin (5-HT) of tumour-feeding arterioles (TFA). Materials…”
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  7. 7

    AS heterozygote hemoglobinopathy and coronary risk by Ould Amar, A K, Pi Gibert, A, Darmon, O, Besse, P, Cénac, A, Césaire, R

    “…There have been several reports of vaso-occlusive events and sudden death in subjects with sickle cell trait. However, the precise mechanism underlying these…”
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    Journal Article