Search Results - "Dai, Maozheng"

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    Cyclin D1 Regulates Cellular Migration through the Inhibition of Thrombospondin 1 and ROCK Signaling by Li, Zhiping, Wang, Chenguang, Jiao, Xuanmao, Lu, Yinan, Fu, Maofu, Quong, Andrew A., Dye, Chip, Yang, Jianguo, Dai, Maozheng, Ju, Xiaoming, Zhang, Xueping, Li, Anping, Burbelo, Peter, Stanley, E. Richard, Pestell, Richard G.

    Published in Molecular and Cellular Biology (01-06-2006)
    “…Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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    Dynamic Cytoskeleton-Integrin Associations Induced by Cell Binding to Immobilized Fibronectin by Mueller, Susette C., Kelly, Thomas, Dai, Maozheng, Dai, Haining, Chen, Wen-Tien

    Published in The Journal of cell biology (01-12-1989)
    “…We have examined the early events of cellular attachment and spreading (10-30 min) by allowing chick embryonic fibroblasts transformed by Rous sarcoma virus to…”
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    Actin filament organization during endothelial wound healing in the rabbit cornea: comparison between transcorneal freeze and mechanical scrape injuries by Ichijima, H, Petroll, WM, Barry, PA, Andrews, PM, Dai, M, Cavanagh, HD, Jester, JV

    “…To compare and contrast the in vivo mechanism of wound healing after mechanical scrape and transcorneal freeze (TCF) injury in a rabbit eye model by examining…”
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  5. 5

    In vivo confocal microscopic studies of endothelial wound healing in rabbit cornea by Ichijima, H, Petroll, W M, Jester, J V, Barry, P A, Andrews, P M, Dai, M, Cavanagh, H D

    Published in Cornea (01-09-1993)
    “…Corneal endothelial wound healing in living rabbit eyes after mechanical scrape (MS) and transcorneal freeze (TCF) injury was studied using tandem scanning…”
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  6. 6

    Membrane proteases as potential diagnostic and therapeutic targets for breast malignancy by Chen, W T, Lee, C C, Goldstein, L, Bernier, S, Liu, C H, Lin, C Y, Yeh, Y, Monsky, W L, Kelly, T, Dai, M

    “…Metastasizing cancer cells can invade the extracellular matrix using plasma membrane protrusions, termed invadopodia, that contact and dissolve the matrix…”
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