Search Results - "DANIELISOVA, Viera"
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Delayed post-conditioning reduces post-ischemic glutamate level and improves protein synthesis in brain
Published in Neurochemistry international (01-05-2013)“…► Ischemia elevates blood and brain glutamate and tissue protein synthesis. ► Post-conditioning activates mechanisms to eliminate glutamate from brain and…”
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The effect of R-(-)-deprenyl administration on antioxidant enzymes in rat testis
Published in European journal of pharmacology (05-10-2016)“…The aim of the study was to investigate the effect of R-(-)-deprenyl administration on the activity and localization of superoxide dismutases (SODs) and…”
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Ischemic Tolerance: The Mechanisms of Neuroprotective Strategy
Published in Anatomical record (Hoboken, N.J. : 2007) (01-12-2009)“…The phenomenon of ischemic tolerance perfectly describes this quote “What does not kill you makes you stronger.” Ischemic pre‐ or postconditioning is actually…”
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Delayed postconditionig initiates additive mechanism necessary for survival of selectively vulnerable neurons after transient ischemia in rat brain
Published in Cellular and molecular neurobiology (01-11-2006)“…1. The aim of this study was to validate the role of postconditioning, used 2 days after lethal ischemia, for protection of selectively vulnerable brain…”
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Bradykinin Postconditioning Protects Pyramidal CA1 Neurons Against Delayed Neuronal Death in Rat Hippocampus
Published in Cellular and molecular neurobiology (01-09-2009)“…Aims The present study was undertaken to evaluate possible neuroprotective effect of bradykinin against delayed neuronal death in hippocampal CA1 neurons if…”
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Bradykinin preconditioning affects the number of degenerated neurons and the level of antioxidant enzymes in spinal cord ischemia in rabbits
Published in Acta histochemica (01-01-2014)“…Bradykinin preconditioning has been used for acquisition of tolerance after spinal cord ischemia. Rabbits were preconditioned intraperitoneally with bradykinin…”
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Neuroprotection and antioxidants
Published in Neural regeneration research (01-06-2016)“…Ischemia as a serious neurodegenerative disorder causes together with reperfusion injury many changes in nervous tissue. Most of the neuronal damage is caused…”
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Postconditioning and Anticonditioning: Possibilities to Interfere to Evoked Apoptosis
Published in Cellular and molecular neurobiology (01-09-2009)“…The aim of this study was to validate the ability of postconditioning, used 2 days after kainate intoxication, to protect selectively vulnerable hippocampal…”
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Evidence for a role of second pathophysiological stress in prevention of delayed neuronal death in the hippocampal CA1 region
Published in Neurochemical research (01-11-2005)“…In ischemic tolerance experiment, when we applied 5-min ischemia 2 days before 30-min ischemia, we achieved a remarkable (95.8%) survival of CA1 neurons…”
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Role of protein synthesis in the ischemic tolerance acquisition induced by transient forebrain ischemia in the rat
Published in Neurochemical research (01-08-2003)“…Although ischemic preconditioning of the heart and brain is a well-documented neuroprotective phenomenon, the mechanism underlying the increased resistance to…”
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Iron deposition after transient forebrain ischemia in rat brain
Published in Neurochemical research (01-03-2002)“…In this study, we investigated the iron deposition in the cerebral cortex, hippocampus CA1 area and corpus striatum pars dorsolateralis in a rat model of…”
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Effect of L-carnitine on postischemic inhibition of protein synthesis in the rat brain
Published in General physiology and biophysics (01-09-2009)“…The purpose of this study was to investigate effects of carnitine administration on protein synthesis recovery after transient cerebral ischemia. Rats received…”
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Effect of Bradykinin Postconditioning on Ischemic and Toxic Brain Damage
Published in Neurochemical research (01-08-2015)“…Brain damage caused by ischemia or toxic agents leads in selectively vulnerable regions to apoptosis-like delayed neuronal death and can result in irreversible…”
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Effects of Bradykinin Postconditioning on Endogenous Antioxidant Enzyme Activity After Transient Forebrain Ischemia in Rat
Published in Neurochemical research (01-06-2008)“…Bradykinin is considered an important mediator of the inflammatory response in both the peripheral and the central nervous system and it has attracted recent…”
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The changes in endogenous antioxidant enzyme activity after postconditioning
Published in Cellular and molecular neurobiology (01-11-2006)“…1. The aim of this work was to study potential mechanisms participating in postischemic protection of selectively vulnerable CA1 neurons in the hippocampus…”
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THE EFFECT OF NORMOVOLEMIC HEMODILUTION ON C-FOS PROTEIN IMMUNOREACTIVITY IN THE POSTISCHEMIC RAT BRAIN CORTEX
Published in International journal of neuroscience (01-04-2005)“…The hemodilution effect was evaluated in the parietal, temporal, and basal regions of postischemic rat brain neocortex by the immunohistochemical detection of…”
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Bradykinin postconditioning ameliorates focal cerebral ischemia in the rat
Published in Neurochemistry international (01-06-2014)“…•We tested possible delay of postconditioning after MCAO.•Regional distribution of SOD changes after ischemia was followed.•We confirmed that bradykinin is…”
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Development of a pattern in biochemical parameters in the core and penumbra during infarct evolution after transient MCAO in rats
Published in Neurochemistry international (01-01-2013)“…► Core and penumbra differ in biochemical parameters. ► Parameters clearly reflect spreading of tissue damage after MCAO. ► Patterns seem to be clearly…”
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Changes of endogenous antioxidant enzymes during ischemic tolerance acquisition
Published in Neurochemical research (01-04-2005)“…The purpose of this study was to investigate the role of superoxide dismutase (SOD) and catalase (CAT) in brain ischemic tolerance induced by ischemic…”
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Ionizing radiation as preconditioning against transient cerebral ischemia in rats
Published in General physiology and biophysics (01-10-2014)“…Induction of ischemic tolerance (IT), the ability of an organism to survive an otherwise lethal ischemia, is the most effective known approach to preventing…”
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