Search Results - "Cui, Wilson W."

Stac3 is a component of the excitation–contraction coupling machinery and mutated in Native American myopathy by Horstick, Eric J., Linsley, Jeremy W., Dowling, James J., Hauser, Michael A., McDonald, Kristin K., Ashley-Koch, Allison, Saint-Amant, Louis, Satish, Akhila, Cui, Wilson W., Zhou, Weibin, Sprague, Shawn M., Stamm, Demetra S., Powell, Cynthia M., Speer, Marcy C., Franzini-Armstrong, Clara, Hirata, Hiromi, Kuwada, John Y.

“…Excitation–contraction coupling, the process that regulates contractions by skeletal muscles, transduces changes in membrane voltage by activating release of…”
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Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease by Hirata, Hiromi, Watanabe, Takaki, Hatakeyama, Jun, Sprague, Shawn M, Saint-Amant, Louis, Nagashima, Ayako, Cui, Wilson W, Zhou, Weibin, Kuwada, John Y

“…Wild-type zebrafish embryos swim away in response to tactile stimulation. By contrast, relatively relaxed mutants swim slowly due to weak contractions of trunk…”
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Pharmacologic approaches to weaning from cardiopulmonary bypass and extracorporeal membrane oxygenation by Cui, Wilson W., MD, PhD, Ramsay, James G., MD

“…Cardiopulmonary bypass (CBP) and extracorporeal membrane oxygenation (ECMO) are two modalities of mechanical circulatory support. They provide hemodynamic…”
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TRPM7 is required within zebrafish sensory neurons for the activation of touch-evoked escape behaviors by Low, Sean E, Amburgey, Kimberly, Horstick, Eric, Linsley, Jeremy, Sprague, Shawn M, Cui, Wilson W, Zhou, Weibin, Hirata, Hiromi, Saint-Amant, Louis, Hume, Richard I, Kuwada, John Y

“…Mutations in the gene encoding TRPM7 (trpm7), a member of the Transient Receptor Potential (TRP) superfamily of cation channels that possesses an enzymatically…”
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Zebrafish bandoneon Mutants Display Behavioral Defects Due to a Mutation in the Glycine Receptor β-Subunit by Hirata, Hiromi, Saint-Amant, Louis, Downes, Gerald B., Cui, Wilson W., Zhou, Weibin, Granato, Michael, Kuwada, John Y., Landmesser, Lynn T.

“…Bilateral alternation of muscle contractions requires reciprocal inhibition between the two sides of the hindbrain and spinal cord, and disruption of this…”
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Connexin 39.9 Protein Is Necessary for Coordinated Activation of Slow-twitch Muscle and Normal Behavior in Zebrafish by Hirata, Hiromi, Wen, Hua, Kawakami, Yu, Naganawa, Yuriko, Ogino, Kazutoyo, Yamada, Kenta, Saint-Amant, Louis, Low, Sean E., Cui, Wilson W., Zhou, Weibin, Sprague, Shawn M., Asakawa, Kazuhide, Muto, Akira, Kawakami, Koichi, Kuwada, John Y.

“…In many tissues and organs, connexin proteins assemble between neighboring cells to form gap junctions. These gap junctions facilitate direct intercellular…”
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NaV1.6a is required for normal activation of motor circuits normally excited by tactile stimulation by Low, Sean E., Zhou, Weibin, Choong, Ingxin, Saint‐Amant, Louis, Sprague, Shawn M., Hirata, Hiromi, Cui, Wilson W., Hume, Richard I., Kuwada, John Y.

“…A screen for zebrafish motor mutants identified two noncomplementing alleles of a recessive mutation that were named non‐active (navmi89 and navmi130). nav…”
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The zebrafish ennui behavioral mutation disrupts acetylcholine receptor localization and motor axon stability by Saint‐Amant, Louis, Sprague, Shawn M., Hirata, Hiromi, Li, Qin, Cui, Wilson W., Zhou, Weibin, Poudou, Olivier, Hume, Richard I., Kuwada, John Y.

“…The zebrafish ennui mutation was identified from a mutagenesis screen for defects in early behavior. Homozygous ennui embryos swam more slowly than wild‐type…”
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Aorta-Right Atrial Tunnel in an Elderly Patient by Salehi, Ali, Cui, Wilson W.

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The Zebrafish shocked Gene Encodes a Glycine Transporter and Is Essential for the Function of Early Neural Circuits in the CNS by Cui, Wilson W, Low, Sean E, Hirata, Hiromi, Saint-Amant, Louis, Geisler, Robert, Hume, Richard I, Kuwada, John Y

“…shocked (sho) is a zebrafish mutation that causes motor deficits attributable to CNS defects during the first2dof development. Mutant embryos display reduced…”
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touché Is required for touch-evoked generator potentials within vertebrate sensory neurons by Low, Sean E, Ryan, Joel, Sprague, Shawn M, Hirata, Hiromi, Cui, Wilson W, Zhou, Weibin, Hume, Richard I, Kuwada, John Y, Saint-Amant, Louis

“…The process by which light touch in vertebrates is transformed into an electrical response in cutaneous mechanosensitive neurons is a largely unresolved…”
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shocked Gene Is Required for the Function of a Premotor Network in the Zebrafish CNS by Cui, Wilson W, Saint-Amant, Louis, Kuwada, John Y

“…1 Department of Molecular, Cellular and Developmental Biology, and 2 Cell and Molecular Biology Graduate Program, University of Michigan, Ann Arbor, Michigan…”
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Na V 1.6a is required for normal activation of motor circuits normally excited by tactile stimulation by Low, Sean E., Zhou, Weibin, Choong, Ingxin, Saint‐Amant, Louis, Sprague, Shawn M., Hirata, Hiromi, Cui, Wilson W., Hume, Richard I., Kuwada, John Y.

“…Abstract A screen for zebrafish motor mutants identified two noncomplementing alleles of a recessive mutation that were named non‐active ( nav mi89 and nav…”
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Non-sense mutations in the dihydropyridine receptor beta 1 gene, CACNB1, paralyze zebrafish relaxed mutants by Zhou, Weibin, Saint-Amant, Louis, Hirata, Hiromi, Cui, Wilson W, Sprague, Shawn M, Kuwada, John Y

“…Contractions by skeletal muscle require proper excitation-contraction (EC) coupling, whereby depolarization of the muscle membrane leads to an increase in…”
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Non-sense mutations in the dihydropyridine receptor beta1 gene, CACNB1, paralyze zebrafish relaxed mutants by Zhou, Weibin, Saint-Amant, Louis, Hirata, Hiromi, Cui, Wilson W, Sprague, Shawn M, Kuwada, John Y

“…Contractions by skeletal muscle require proper excitation-contraction (EC) coupling, whereby depolarization of the muscle membrane leads to an increase in…”
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Non-sense mutations in the dihydropyridine receptor β1 gene, CACNB1, paralyze zebrafish relaxed mutants by Zhou, Weibin, Saint-Amant, Louis, Hirata, Hiromi, Cui, Wilson W., Sprague, Shawn M., Kuwada, John Y.

“…Contractions by skeletal muscle require proper excitation–contraction (EC) coupling, whereby depolarization of the muscle membrane leads to an increase in…”
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From behavior to circuitry: The role of glycine regulation in CNS motor output by Cui, Wilson W

“…One central goal in neurobiology is to establish the link between genes and behavior. The gap between the motor behaviors of whole organisms and our growing…”
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Na(v)1.6a is required for normal activation of motor circuits normally excited by tactile stimulation by Low, Sean E, Zhou, Weibin, Choong, Ingxin, Saint-Amant, Louis, Sprague, Shawn M, Hirata, Hiromi, Cui, Wilson W, Hume, Richard I, Kuwada, John Y

“…A screen for zebrafish motor mutants identified two noncomplementing alleles of a recessive mutation that were named non-active (nav(mi89) and nav(mi130)). nav…”
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From behavior to circuitry: The role of glycine regulation in CNS motor output by Cui, Wilson W

“…One central goal in neurobiology is to establish the link between genes and behavior. The gap between the motor behaviors of whole organisms and our growing…”
Get full text