Search Results - "Clatterbuck Soper, Sarah F"

  • Showing 1 - 5 results of 5
Refine Results
  1. 1

    In Vivo X-Ray Footprinting of Pre-30S Ribosomes Reveals Chaperone-Dependent Remodeling of Late Assembly Intermediates by Clatterbuck Soper, Sarah F, Dator, Romel P, Limbach, Patrick A, Woodson, Sarah A

    Published in Molecular cell (21-11-2013)
    “…Assembly of 30S ribosomal subunits from their protein and RNA components requires extensive refolding of the 16S rRNA and is assisted by 10–20 assembly factors…”
    Get full text
    Journal Article
  2. 2

    Rapid and reversible suppression of ALT by DAXX in osteosarcoma cells by Yost, Kathryn E., Clatterbuck Soper, Sarah F., Walker, Robert L., Pineda, Marbin A., Zhu, Yuelin J., Ester, Corbin D., Showman, Soyeon, Roschke, Anna V., Waterfall, Joshua J., Meltzer, Paul S.

    Published in Scientific reports (14-03-2019)
    “…Many tumors maintain chromosome-ends through a telomerase-independent, DNA-templated mechanism called alternative lengthening of telomeres (ALT). While ALT…”
    Get full text
    Journal Article
  3. 3

    FUS inclusions disrupt RNA localization by sequestering kinesin-1 and inhibiting microtubule detyrosination by Yasuda, Kyota, Clatterbuck-Soper, Sarah F, Jackrel, Meredith E, Shorter, James, Mili, Stavroula

    Published in The Journal of cell biology (03-04-2017)
    “…Cytoplasmic inclusions of the RNA-binding protein fused in sarcoma (FUS) represent one type of membraneless ribonucleoprotein compartment. Formation of FUS…”
    Get full text
    Journal Article
  4. 4

    ATRX/DAXX: Guarding the Genome against the Hazards of ALT by Clatterbuck Soper, Sarah F, Meltzer, Paul S

    Published in Genes (24-03-2023)
    “…Proliferating cells must enact a telomere maintenance mechanism to ensure genomic stability. In a subset of tumors, telomeres are maintained not by telomerase,…”
    Get full text
    Journal Article
  5. 5

    Abstract 3467: A DAXX-KIFC3 fusion potentiates alternative lengthening of telomeres in osteosarcoma by Soper, Sarah F. Clatterbuck, Showman, Soyeon A., Driest, Kathryn E., Waterfall, Joshua J., Walker, Robert L., Pineda, Marbin A., Zhu, Yuelin J., Wang, Yonghong, Ester, Corbin D., Bilke, Sven, Meltzer, Paul S.

    Published in Cancer research (Chicago, Ill.) (01-07-2017)
    “…Proliferating cells must enact a program of telomere lengthening to counteract the chromosome end replication problem. In most types of cancer cells, telomeres…”
    Get full text
    Journal Article