Search Results - "Chevion, S"

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  1. 1

    The antioxidant properties of thioctic acid: Characterization by cyclic voltammetry by Chevion, S., Hofmann, M., Ziegler, R., Chevion, M., Nawroth, P. P.

    “…Thioctic acid (TA) level and its antioxidant capacity were monitored by cyclic voltammetry (CV), in solution and in human plasma. A linear correlation between…”
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  2. 2

    Advanced glycation end product-induced activation of NF-kappaB is suppressed by alpha-lipoic acid in cultured endothelial cells by Bierhaus, A., Chevion, S., Chevion, M., Hofmann, M., Quehenberger, P., Illmer, T., Luther, T., Berentshtein, E., Tritschler, H., Muller, M., Wahl, P., Ziegler, R., Nawroth, P. P.

    Published in Diabetes (New York, N.Y.) (01-09-1997)
    “…Advanced glycation end product-induced activation of NF-kappaB is suppressed by alpha-lipoic acid in cultured endothelial cells. A Bierhaus , S Chevion , M…”
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  3. 3

    The use of cyclic voltammetry for the evaluation of antioxidant capacity by Chevion, Shlomit, Roberts, Matthew A., Chevion, Mordechai

    Published in Free radical biology & medicine (15-03-2000)
    “…Low–molecular weight antioxidants (LMWAs) play a major role in protecting biological systems against reactive oxygen–derived species and reflect the…”
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  4. 4

    Plasma Antioxidant Status and Cell Injury after Severe Physical Exercise by Chevion, Shlomit, Moran, Danny S., Heled, Yuval, Shani, Yoav, Regev, Gilad, Abbou, Benny, Berenshtein, Eduard, Stadtman, Earl R., Epstein, Yoram

    “…Strenuous exercise leads to an increase in metabolic rate, increased production of reactive oxygen species, and compromised antioxidant defense systems. To…”
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  5. 5

    Advanced glycation end product-induced activation of NF-κB is suppressed by α-lipoic acid in cultured endothelial cells by BIERHAUS, A, CHEVION, S, WAHL, P, ZIEGLER, R, NAWROTH, P. P, CHEVION, M, HOFMANN, M, QUEHENBERGER, P, ILLMER, T, LUTHER, T, BERENTSHTEIN, E, TRITSCHLER, H, MÜLLER, M

    Published in Diabetes (New York, N.Y.) (01-09-1997)
    “…Depletion of cellular antioxidant defense mechanisms and the generation of oxygen free radicals by advanced glycation end products (AGEs) have been proposed to…”
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  6. 6

    Antioxidant Status and Human Health: Use of Cyclic Voltammetry for the Evaluation of the Antioxidant Capacity of Plasma and of Edible Plants by CHEVION, SHLOMIT, CHEVION, MORDECHAI

    Published in Annals of the New York Academy of Sciences (01-01-2000)
    “…: The low molecular weight antioxidants (LMWA) play a major role in protecting biological systems against reactive oxygen‐derived species (ROS), and reflect…”
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  7. 7

    Evaluation of Plasma Low Molecular Weight Antioxidant Capacity by Cyclic Voltammetry by Chevion, Shlomit, Berry, Elliot M., Kitrossky, Nahum, Kohen, Ron

    Published in Free radical biology & medicine (1997)
    “…The low molecular weight antioxidants (LMWA) of biological samples include many compounds and contribute to the total antioxidant capacity of the system. They…”
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  8. 8

    Citrus fruit supplementation reduces lipoprotein oxidation in young men ingesting a diet high in saturated fat: presumptive evidence for an interaction between vitamins C and E in vivo by Harats, D, Chevion, S, Nahir, M, Norman, Y, Sagee, O, Berry, EM

    Published in The American journal of clinical nutrition (01-02-1998)
    “…To determine the effects of vitamin C on cardiovascular risk factors, we studied dietary vitamin C enrichment in 36 healthy male students consuming a diet high…”
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  9. 9

    antioxidant status of patients subjected to total body irradiation by Chevion, S, Or, R, Berry, E.M

    “…Total body irradiation (TBI) is a routine preconditioning procedure for the treatment of leukemia and aplastic anemia, prior to bone marrow transplantation…”
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  10. 10
  11. 11

    A New Concept in Antidiabetic Therapeutics: A Concerted Removal of Labile Iron and Intracellular Deposition of Zinc by Vladimir Vinokur, Eduard Berenshtein, Mordechai Chevion, Dror Chevion

    Published in Diabetes & metabolism journal (31-01-2024)
    “…Background: The inflammatory process is known to be an integral part of the pathophysiology of type 2 diabetes mellitus (T2DM). The “labile,” redox-active…”
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