Search Results - "Cheroni, C."

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    Accumulation of human SOD1 and ubiquitinated deposits in the spinal cord of SOD1G93A mice during motor neuron disease progression correlates with a decrease of proteasome by Cheroni, C., Peviani, M., Cascio, P., DeBiasi, S., Monti, C., Bendotti, C.

    Published in Neurobiology of disease (01-04-2005)
    “…Mutations in SOD1 cause selective motor neuron degeneration in familial amyotrophic lateral sclerosis patients and transgenic mice overexpressing the mutant…”
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    Lack of changes in the PI3K/AKT survival pathway in the spinal cord motor neurons of a mouse model of familial amyotrophic lateral sclerosis by Peviani, M., Cheroni, C., Troglio, F., Quarto, M., Pelicci, G., Bendotti, C.

    Published in Molecular and cellular neuroscience (01-04-2007)
    “…The vulnerability of motor neurons in transgenic SOD1G93A mice, a model of familial amyotrophic lateral sclerosis (ALS), may depend on the failure of these…”
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    Inter- and intracellular signaling in amyotrophic lateral sclerosis: role of p38 mitogen-activated protein kinase by Bendotti, C, Bao Cutrona, M, Cheroni, C, Grignaschi, G, Lo Coco, D, Peviani, M, Tortarolo, M, Veglianese, P, Zennaro, E

    Published in Neuro-degenerative diseases (01-01-2005)
    “…The pathogenetic processes underlying the selective motor neuron degeneration in amyotrophic lateral sclerosis (ALS) are complex and still not completely…”
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    Journal Article
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    Protein nitration in a mouse model of familial amyotrophic lateral sclerosis: possible multifunctional role in the pathogenesis by Casoni, Filippo, Basso, Manuela, Massignan, Tania, Gianazza, Elisabetta, Cheroni, Cristina, Salmona, Mario, Bendotti, Caterina, Bonetto, Valentina

    Published in The Journal of biological chemistry (22-04-2005)
    “…Multiple mechanisms have been proposed to contribute to amyotrophic lateral sclerosis (ALS) pathogenesis, including oxidative stress. Early evidence of a role…”
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    Journal Article