Search Results - "Chang, B. X."

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  1. 1

    Denitrification as the dominant nitrogen loss process in the Arabian Sea by Chang, B. X, Pratihary, Anil, Rich, J. J, Devol, A. H, Jayakumar, A, Bulow, S. E, Ward, B. B, Naik, Hema

    Published in Nature (London) (03-09-2009)
    “…Primary production in over half of the world’s oceans is limited by fixed nitrogen availability. The main loss term from the fixed nitrogen inventory is the…”
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    Journal Article
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    Nitrogen losses in anoxic marine sediments driven by Thioploca–anammox bacterial consortia by Prokopenko, M. G., Hirst, M. B., De Brabandere, L., Lawrence, D. J. P., Berelson, W. M., Granger, J., Chang, B. X., Dawson, S., Crane III, E. J., Chong, L., Thamdrup, B., Townsend-Small, A., Sigman, D. M.

    Published in Nature (London) (08-08-2013)
    “…A novel symbiotic consortium is described between two chemolithotrophic bacteria — anaerobic ammonium-oxidizing (anammox) bacteria and the nitrate-sequestering…”
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    Journal Article
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    Activity and abundance of denitrifying bacteria in the subsurface biosphere of diffuse hydrothermal vents of the Juan de Fuca Ridge by Bourbonnais, A, Juniper, S. K, Butterfield, D. A, Devol, A. H, Kuypers, M. M. M, Lavik, G, Hallam, S. J, Wenk, C. B, Chang, B. X, Murdock, S. A, Lehmann, M. F

    Published in Biogeosciences (22-11-2012)
    “…Little is known about fixed nitrogen (N) transformation and elimination at diffuse hydrothermal vents where anoxic fluids are mixed with oxygenated crustal…”
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    Journal Article
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    Phosphorylation of protein kinase C sites in NBD1 and the R domain control CFTR channel activation by PKA by Chappe, V, Hinkson, D A, Zhu, T, Chang, X-B, Riordan, J R, Hanrahan, J W

    Published in The Journal of physiology (01-04-2003)
    “…Activation of the cystic fibrosis transmembrane conductance regulator (CFTR) channel by protein kinase A (PKA) is enhanced by protein kinase C (PKC). However,…”
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    Journal Article
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    Multiple flavonoid-binding sites within multidrug resistance protein MRP1 by Trompier, D, Baubichon-Cortay, H, Chang, X-B, Maitrejean, M, Barron, D, Riordon, J R, Di Pietro, A

    “…Recombinant nucleotide-binding domains (NBDs) from human multidrug resistance protein MRP1 were overexpressed in bacteria and purified to measure their direct…”
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    Journal Article
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    Influence of phosphorylation by protein kinase A on CFTR at the cell surface and endoplasmic reticulum by Seibert, F.S., Chang, X.-B., Aleksandrov, A.A., Clarke, D.M., Hanrahan, J.W., Riordan, J.R.

    Published in BBA - Biomembranes (06-12-1999)
    “…CFTR possesses a large cluster of strict dibasic consensus sites for phosphorylation by protein kinase A (PKA) in the R-domain and an obligatory dependence on…”
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    Book Review Journal Article
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    Nitrogen losses in anoxicmarine sediments driven by Thioploca-anammox bacterial consortia by Prokopenko, M G, Hirst, M B, DeBrabandere, L, Lawrence, D J P, Berelson, W M, Granger, J, Chang, B X, Dawson, S, Crane, E J, Chong, L, Thamdrup, B, Townsend-Small, A, Sigman, D M

    Published in Nature (London) (08-08-2013)
    “…Ninety per cent of marine organic matter burial occurs in continental margin sediments, where a substantial fraction of organic carbon escapes oxidation and…”
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    Journal Article
  8. 8

    CFTR is a monomer: biochemical and functional evidence by Chen, J-H, Chang, X-B, Aleksandrov, A A, Riordan, J R

    Published in The Journal of membrane biology (01-07-2002)
    “…Although the CFTR protein alone is sufficient to generate a regulated chloride channel, it is unknown how many of the polypeptides form the channel. Using…”
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    Journal Article
  9. 9

    The delta F508 mutation decreases the stability of cystic fibrosis transmembrane conductance regulator in the plasma membrane. Determination of functional half-lives on transfected cells by LUKACS, G. L, XIU-BAO CHANG, BEAR, C, KARTNER, N, MOHAMED, A, RIORDAN, J. R, GRINSTEIN, S

    Published in The Journal of biological chemistry (15-10-1993)
    “…Deletion of the phenylalanine at position 508 of the cystic fibrosis transmembrane conductance regulator (CFTR) is the most prevalent mutation in cystic…”
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    Journal Article
  10. 10

    Protein kinase A (PKA) still activates CFTR chloride channel after mutagenesis of all 10 PKA consensus phosphorylation sites by Chang, X.B., Tabcharani, J.A., Hou, Y.X., Jensen, T.J., Kartner, N., Alon, N., Hanrahan, J.W., Riordan, J.R.

    Published in The Journal of biological chemistry (25-05-1993)
    “…The cystic fibrosis transmembrane conductance regulator (CFTR) plays a central role in transepithelial ion transport by acting as a tightly regulated apical…”
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    Journal Article
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    Phosphatase Inhibitors Activate Normal and Defective CFTR Chloride Channels by Becq, Frederic, Jensen, Timothy J., Chang, Xiu-Bao, Savoia, Anna, Rommens, Johanna M., Tsui, Lap-Chee, Buchwald, Manuel, Riordan, John R., Hanrahan, John W.

    “…The cystic fibrosis transmembrane conductance regulator (CFTR) chloride channel is regulated by phosphorylation and dephosphorylation at multiple sites…”
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    Journal Article
  13. 13

    Mapping of cystic fibrosis transmembrane conductance regulator membrane topology by glycosylation site insertion by XIU-BAO CHANG, YUE-XIAN HOU, JENSEN, T. J, RIORDAN, J. R

    Published in The Journal of biological chemistry (15-07-1994)
    “…Technical difficulties in obtaining three-dimensional structures of intrinsic membrane proteins continues to limit understanding of their function. However,…”
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    Journal Article
  14. 14

    Multidrug Resistance Protein MRP1 Reconstituted into Lipid Vesicles:  Secondary Structure and Nucleotide-Induced Tertiary Structure Changes by Manciu, Liliana, Chang, Xiu-Bao, Riordan, John R, Ruysschaert, Jean-Marie

    Published in Biochemistry (Easton) (24-10-2000)
    “…Multidrug resistance protein MRP1 is an ATP-dependent drug efflux pump that confers resistance in human cancer cells to various chemotherapeutic drugs. We have…”
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    Journal Article
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    Mutations of the Walker B Motif in the First Nucleotide Binding Domain of Multidrug Resistance Protein MRP1 Prevent Conformational Maturation by Cui, Liying, Hou, Yue-Xian, Riordan, John R., Chang, Xiu-bao

    Published in Archives of biochemistry and biophysics (01-08-2001)
    “…ATP-binding cassette (ABC) transporters couple the binding and hydrolysis of ATP to the translocation of solutes across biological membranes. The so-called…”
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    Journal Article
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    Comparison of filler DNA at immune, nonimmune, and oncogenic rearrangements suggests multiple mechanisms of formation by ROTH, D. B, XIU-BAO CHANG, WILSON, J. H

    Published in Molecular and Cellular Biology (01-07-1989)
    “…Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley…”
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    Journal Article
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    Establishment of cytochrome P450 3A4 and glutathione S-transferase A1-transfected human hepatoma cell line and functional analysis by Chang, B X, You, S L, Liu, H L, Mao, P Y, Xin, S J

    Published in Genetics and molecular research (14-04-2014)
    “…This study aimed to enhance the drug metabolism function of the human hepatoma cell line C3A and to explore the related significance for patients with severe…”
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    Journal Article
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    The cystic fibrosis transmembrane regulator is present and functional in endosomes. Role as a determinant of endosomal pH by Lukacs, G L, Chang, X B, Kartner, N, Rotstein, O D, Riordan, J R, Grinstein, S

    Published in The Journal of biological chemistry (25-07-1992)
    “…Cystic fibrosis is caused by mutations in the gene encoding the cystic fibrosis transmembrane conductance regulator (CFTR), which lead to defective Cl-…”
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    Journal Article
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    Perturbation of Hsp90 interaction with nascent CFTR prevents its maturation and accelerates its degradation by the proteasome by Loo, Melinda A., Jensen, Timothy J., Cui, Liying, Hou, Yue-xian, Chang, Xiu-Bao, Riordan, John R.

    Published in The EMBO journal (01-12-1998)
    “…Maturation of wild‐type CFTR nascent chains at the endoplasmic reticulum (ER) occurs inefficiently; many disease‐associated mutant forms do not mature but…”
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    Journal Article
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