Search Results - "Cassam, A.K"

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  1. 1

    Changes in immunoreactivity for growth associated protein-43 suggest reorganization of synapses on spinal sympathetic neurons after cord transection by Weaver, L.C, Cassam, A.K, Krassioukov, A.V, Llewellyn-Smith, I.J

    Published in Neuroscience (01-11-1997)
    “…Cervical or high thoracic spinal cord injury often results in autonomic dysreflexia, a condition characterized by exaggerated spinal reflexes and episodic…”
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  2. 2

    Catecholamine enzymes and neuropeptides are expressed in fibres and somata in the intermediate gray matter in chronic spinal rats by CASSAM, A. K, LLEWELLYN-SMITH, I. J, WEAVER, L. C

    Published in Neuroscience (01-06-1997)
    “…Spinal cord injury disrupts control of sympathetic preganglionic neurons because bulbospinal input has been lost and the remaining regulation is accomplished…”
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  3. 3

    Glutamate- and GABA-immunoreactive synapses on sympathetic preganglionic neurons caudal to a spinal cord transection in rats by Llewellyn-Smith, I.J, Cassam, A.K, Krenz, N.R, Krassioukov, A.V, Weaver, L.C

    Published in Neuroscience (01-10-1997)
    “…Spinal cord injury destroys bulbospinal amino acid-containing pathways to sympathetic preganglionic neurons and severely disrupts blood pressure control,…”
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    Journal Article
  4. 4

    Co-localization of substance P and dopamine β-hydroxylase with growth-associated protein-43 is lost caudal to a spinal cord transection by Cassam, A.K, Rogers, K.A, Weaver, L.C

    Published in Neuroscience (01-02-1999)
    “…After spinal cord injury, abnormal responses of spinal cord neurons to sensory input lead to conditions such as autonomic dysreflexia, urinary bladder…”
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  5. 5

    Analysis of a multi-mutant herpes simplex virus type 1 for gene transfer into sympathetic preganglionic neurons and a comparison to adenovirus vectors by LeVatte, M.A, Cassam, A.K, Dekaban, G.A, Weaver, L.C

    Published in Neuroscience (01-10-1998)
    “…A non-replicating triple-mutant herpes simplex virus (14H Δ3vhsZ) expressing the bacterial marker enzyme β-galactosidase, was assessed for neurotropism and…”
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    Journal Article